Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Marjan Reinders-Luinge"'
Autor:
Dan-Dan Wu, Andy T. Y. Lau, Yan-Ming Xu, Marjan Reinders-Luinge, Mihaly Koncz, Antal Kiss, Wim Timens, Marianne G. Rots, Machteld N. Hylkema
Publikováno v:
Epigenetics, Vol 18, Iss 1 (2023)
Ubiquitin carboxyl-terminal hydrolase L1 (UCHL1) is highly expressed in smokers, but little is known about the molecular mechanism of UCHL1 in airway epithelium and its possible role in affecting extracellular matrix (ECM) remodelling in the underlyi
Externí odkaz:
https://doaj.org/article/2a12fdb3945348458e0b03b9495d19c6
Autor:
Khosbayar Lkhagvadorj, Karolin F. Meyer, Laura P. Verweij, Wierd Kooistra, Marjan Reinders-Luinge, Henk W. Dijkhuizen, Inge A. M. de Graaf, Torsten Plösch, Machteld N. Hylkema
Publikováno v:
Epigenetics, Vol 15, Iss 12, Pp 1370-1385 (2020)
Prenatal smoke exposure (PSE) is a risk factor for nicotine dependence. One susceptibility gene for nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conversion of nicotine to cotinine and nicotine clearance in the liver
Externí odkaz:
https://doaj.org/article/9c83266db7144cbbba7fce50a2335ecf
Autor:
Khosbayar Lkhagvadorj, Zhijun Zeng, Karolin F. Meyer, Laura P. Verweij, Wierd Kooistra, Marjan Reinders-Luinge, Henk W. Dijkhuizen, Inge A. M. de Graaf, Torsten Plösch, Machteld N. Hylkema
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 1, p 164 (2020)
Prenatal smoke exposure (PreSE) is a risk factor for nicotine dependence, which is further enhanced by postnatal smoke exposure (PostSE). One susceptibility gene to nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conve
Externí odkaz:
https://doaj.org/article/7ce5be2cb7c74864942a998f51e3c67a
Autor:
Fatemeh Fattahi, Corry-Anke Brandsma, Monique Lodewijk, Marjan Reinders-Luinge, Dirkje S Postma, Wim Timens, Machteld N Hylkema, Nick H T Ten Hacken
Publikováno v:
PLoS ONE, Vol 11, Iss 8, p e0161433 (2016)
Interleukin (IL)-17 plays a critical role in numerous immune and inflammatory responses and was recently suggested to contribute to the pathogenesis of nonatopic (non-eosinophil/neutrophil-dominant) asthma. We aimed to compare expression of IL-17 in
Externí odkaz:
https://doaj.org/article/a3dcd029e5da4f0bbd058bd78be4ee54
Autor:
Henk Koning, Antoon J M van Oosterhout, Uilke Brouwer, Lisette E den Boef, Renée Gras, Marjan Reinders-Luinge, Corry-Anke Brandsma, Marco van der Toorn, Machteld N Hylkema, Brigitte W M Willemse, Ian Sayers, Gerard H Koppelman, Martijn C Nawijn
Publikováno v:
PLoS ONE, Vol 9, Iss 7, p e98197 (2014)
Protocadherin-1 (PCDH1) is a novel susceptibility gene for airway hyperresponsiveness, first identified in families exposed to cigarette smoke and is expressed in bronchial epithelial cells. Here, we asked how mouse Pcdh1 expression is regulated in l
Externí odkaz:
https://doaj.org/article/a4ca3577b6484344b6266fcf8f1cacbe
Autor:
Karolin F Meyer, Henk W Dijkhuizen, Torsten Plösch, Laura P Verweij, Wierd Kooistra, Marjan Reinders-Luinge, Khosbayar Lkhagvadorj, Machteld N. Hylkema, Inge A. M. de Graaf
Publikováno v:
Epigenetics, 15(12), 1370-1385. TAYLOR & FRANCIS INC
Prenatal smoke exposure (PSE) is a risk factor for nicotine dependence. One susceptibility gene for nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conversion of nicotine to cotinine and nicotine clearance in the liver
Autor:
Machteld N. Hylkema, Wierd Kooistra, Marjan Reinders-Luinge, Juan Song, Torsten Plösch, Xia Huo, Zhijun Zeng, Karolin F Meyer, Khosbayar Lkhagvadorj, Xijin Xu
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology, 318(3), L549-L561. AMER PHYSIOLOGICAL SOC
Prenatal smoke exposure (PSE) is associated with reduced birth weight, impaired fetal development, and increased risk for diseases later in life. Changes in DNA methylation may be involved, as multiple large-scale epigenome-wide association studies s
Autor:
Machteld N. Hylkema, Henk W Dijkhuizen, Torsten Plösch, Wierd Kooistra, Zhijun Zeng, Karolin F Meyer, Laura P Verweij, Khosbayar Lkhagvadorj, Inge A. M. de Graaf, Marjan Reinders-Luinge
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 164, p 164 (2021)
International Journal of Molecular Sciences
Volume 22
Issue 1
International Journal of Molecular Sciences, 22(1):164. MDPI AG
International Journal of Molecular Sciences
Volume 22
Issue 1
International Journal of Molecular Sciences, 22(1):164. MDPI AG
Prenatal smoke exposure (PreSE) is a risk factor for nicotine dependence, which is further enhanced by postnatal smoke exposure (PostSE). One susceptibility gene to nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conve
Autor:
Juan Song, Khosbayar Lkhagvadorj, Barbro N. Melgert, Susanne Krauss-Etschmann, Shanshan Song, Wierd Kooistra, Zhijun Zeng, Marjan Reinders-Luinge, Machteld N. Hylkema, Junjun Cao
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology, 319(4), L742-L751. AMER PHYSIOLOGICAL SOC
Prenatal smoke exposure is a risk factor for impaired lung development in children. Recent studies have indicated that amphiregulin (AREG), which is a ligand of the epidermal growth factor receptor (EGFR), has a regulatory role in airway epithelial c
Autor:
Zhijun, Zeng, Karolin F, Meyer, Khosbayar, Lkhagvadorj, Wierd, Kooistra, Marjan, Reinders-Luinge, Xijin, Xu, Xia, Huo, Juan, Song, Torsten, Plösch, Machteld N, Hylkema
Publikováno v:
American journal of physiology. Lung cellular and molecular physiology. 318(3)
Prenatal smoke exposure (PSE) is associated with reduced birth weight, impaired fetal development, and increased risk for diseases later in life. Changes in DNA methylation may be involved, as multiple large-scale epigenome-wide association studies s