Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Marivi N. Moen"'
Autor:
Roar Fjær, Marivi N. Moen, Ying Sheng, Dag E. Undlien, El Hassan Hamdani, Robin Johansen Menchini, Farrukh A. Chaudhry, Kaja Kristine Selmer, Mustafa A. Salih, Heba Y. El Khashab, Jon K. Laerdahl, Magnus Dehli Vigeland, Bjørnar Hassel
Publikováno v:
Brain. 139:3109-3120
Progressive myoclonus epilepsy is a heterogeneous group of disorders characterized by myoclonic and tonic-clonic seizures, ataxia and cognitive decline. We here present two affected brothers. At 9 months of age the elder brother developed ataxia and
Autor:
Marina Alexeeva, Peter Ruoff, Izaskun Muruzábal-Lecumberri, Kristin Grøsvik, Xiang Ming Xu, Almaz Nigatu Tesfahun, Arne Klungland, Anette Rasmussen, Marivi N. Moen, Finn Kirpekar, Kristine M. Olsen, Svein Bjelland
Publikováno v:
Alexeeva, M, Moen, M N, Grøsvik, K, Tesfahun, A N, Xu, X M, Muruzábal-Lecumberri, I, Olsen, K M, Rasmussen, A, Ruoff, P, Kirpekar, F, Klungland, A & Bjelland, S 2019, ' Excision of uracil from DNA by hSMUG1 includes strand incision and processing ', Nucleic Acids Research, vol. 47, no. 2, pp. 779-793 . https://doi.org/10.1093/nar/gky1184
Nucleic Acids Research
Nucleic Acids Research
Uracil arises in DNA by hydrolytic deamination of cytosine (C) and by erroneous incorporation of deoxyuridine monophosphate opposite adenine, where the former event is devastating by generation of C → thymine transitions. The base excision repair (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::75365a8bc8f013d98f4a9e88573fb64b
http://hdl.handle.net/10852/71387
http://hdl.handle.net/10852/71387
Autor:
Torbjørn Rognes, David Lando, Ida Jonson, John Arne Dahl, Marivi N. Moen, Jeannie T. Lee, Arne Klungland, Rune Ougland, Elisabeth Larsen, Tony Kouzarides, Line M. Nordstrand
Publikováno v:
Stem Cells (Dayton, Ohio)
AlkB homolog 1 (ALKBH1) is one of nine members of the family of mammalian AlkB homologs. Most Alkbh1−/− mice die during embryonic development, and survivors are characterized by defects in tissues originating from the ectodermal lineage. In this
Autor:
Marivi N. Moen, Pål Ø. Falnes, Paul E. Grini, Cathrine Broberg Vågbø, Arne Klungland, Trine J. Meza, Hans E. Krokan
Publikováno v:
Nucleic Acids Research
The Escherichia coli AlkB protein (EcAlkB) is a DNA repair enzyme which reverses methylation damage such as 1-methyladenine (1-meA) and 3-methylcytosine (3-meC). The mammalian AlkB homologues ALKBH2 and ALKBH3 display EcAlkB-like repair activity in v
Autor:
Kristin Grøsvik, Arne Klungland, Marivi N. Moen, Gyri Teien Haugland, Ingeborg Knævelsrud, Nils-Kåre Birkeland, Svein Bjelland
Publikováno v:
Journal of Bacteriology. 193:4495-4508
Hydrolytic deamination of cytosine to uracil in DNA is increased in organisms adapted to high temperatures. Hitherto, the uracil base excision repair (BER) pathway has only been described in two archaeons, the crenarchaeon Pyrobaculum aerophilum and
Autor:
Lisiane B. Meira, Pål Ø. Falnes, Arne Klungland, Jeanette Ringvoll, Marivi N. Moen, Bo Pang, Anders Bekkelund, Peter C. Dedon, Leona D. Samson, Svein Bjelland, Line M. Nordstrand
Publikováno v:
Cancer Research. 68:4142-4149
Endogenous formation of the mutagenic DNA adduct 1,N6-ethenoadenine (εA) originates from lipid peroxidation. Elevated levels of εA in cancer-prone tissues suggest a role for this adduct in the development of some cancers. The base excision repair p
Autor:
Ingar Leiros, Ingeborg Knævelsrud, Kristin Grøsvik, Gyri Teien Haugland, Nils-Kåre Birkeland, Arne Klungland, Marivi N. Moen, Svein Bjelland
Hydrolytic deamination of cytosine to uracil in cellular DNA is a major source of C-to-T transition mutations if uracil is not repaired by the DNA base excision repair (BER) pathway. Since deamination increases rapidly with temperature, hyperthermoph
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e94c575b55b1f953823d376a0b7c1992
https://europepmc.org/articles/PMC2953685/
https://europepmc.org/articles/PMC2953685/
Autor:
Marina V. Omelchenko, Marivi N. Moen, Pål Ø. Falnes, Arne Klungland, Erwin van den Born, Anders Bekkelund
Publikováno v:
Nucleic Acids Research
The iron(II)- and 2-oxoglutarate (2OG)-dependent dioxygenase AlkB from Escherichia coli (EcAlkB) repairs alkylation damage in DNA by direct reversal. EcAlkB substrates include methylated bases, such as 1-methyladenine (m(1)A) and 3-methylcytosine (m(