Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Marissa Simko"'
Autor:
Christopher F Bennett, Jane J Kwon, Christine Chen, Joshua Russell, Kathlyn Acosta, Nikolay Burnaevskiy, Matthew M Crane, Alessandro Bitto, Helen Vander Wende, Marissa Simko, Victor Pineda, Ryan Rossner, Brian M Wasko, Haeri Choi, Shiwen Chen, Shirley Park, Gholamali Jafari, Bryan Sands, Carissa Perez Olsen, Alexander R Mendenhall, Philip G Morgan, Matt Kaeberlein
Publikováno v:
PLoS Genetics, Vol 13, Iss 3, p e1006695 (2017)
Mitochondrial dysfunction can increase oxidative stress and extend lifespan in Caenorhabditis elegans. Homeostatic mechanisms exist to cope with disruptions to mitochondrial function that promote cellular health and organismal longevity. Previously,
Externí odkaz:
https://doaj.org/article/16244a054fa74a9d8488d864fa5e9844
Publikováno v:
PLoS ONE, Vol 6, Iss 11, p e28036 (2011)
Dietary restriction (DR), the limitation of calorie intake while maintaining proper nutrition, has been found to extend life span and delay the onset of age-associated disease in a wide range of species. Previous studies have suggested that DR can re
Externí odkaz:
https://doaj.org/article/59e2197ec979412595f3cdd99e074df0
Autor:
Alexander Mendenhall, Haeri Choi, Victor V. Pineda, Jane J. Kwon, Matthew M. Crane, Philip G. Morgan, Christopher F. Bennett, Joshua Russell, Shirley Park, Nikolay Burnaevskiy, Bryan Sands, Helen Vander Wende, Brian M. Wasko, Ryan Rossner, Carissa Perez Olsen, Matt Kaeberlein, Kathlyn Acosta, Alessandro Bitto, Gholamali Jafari, Marissa Simko, Shiwen Chen, Christine Chen
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 13, Iss 3, p e1006695 (2017)
PLoS Genetics, Vol 13, Iss 3, p e1006695 (2017)
Mitochondrial dysfunction can increase oxidative stress and extend lifespan in Caenorhabditis elegans. Homeostatic mechanisms exist to cope with disruptions to mitochondrial function that promote cellular health and organismal longevity. Previously,
Autor:
Victor V. Pineda, Matt Kaeberlein, Shannon Klum, Sarah Barfield, Christopher F. Bennett, Helen Vander Wende, Haeri Choi, Marissa Simko
Publikováno v:
Nature communications
Recent studies have propagated the model that the mitochondrial unfolded protein response (UPR(mt)) is causal for lifespan extension from inhibition of the electron transport chain (ETC) in Caenorhabditis elegans. Here we report a genome-wide RNAi sc
Autor:
Autumn Tocchi, Mollie Holmberg, Matt Kaeberlein, George L. Sutphin, Sean Higgins, Minnie Singh, Ki-Soo Jeong, Simon C. Johnson, Sarani Goswami, Helen Vander Wende, Marissa Fletcher, Richard M. Moller, Vanessa Ros, Christopher F. Bennett, Bo Xian, Brady Olsen, Anthony S. Castanza, Peter S. Rabinovitch, Marissa Simko, Michael J. MacCoss, Benjamin L. Spector, Monika Jelic, Jin R. Kim, Dillon Pruett, Elroy H. An, Tao Yu, Winston Lo, Zhao J. Peng, Jennifer Schleit, Jing-Dong J. Han, Edward J. Hsieh, Christopher J. Murakami, Daniel B. Carr, Brian M. Wasko, Hillary Miller, Tom Pollard, Brian K. Kennedy, Joe R. Delaney, Michael S. Lin, Dilreet Rai, Prarthana Pradeep, Natalie Trongtham, Shannon Klum, Weiyang Chen, Eric C. Liao
Summary Dietary restriction (DR) increases lifespan and attenuates age-related phenotypes in many organisms; however, the effect of DR on longevity of individuals in genetically heterogeneous populations is not well characterized. Here, we describe a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ee7118c182336261c52dcb043ea3183
https://europepmc.org/articles/PMC3838465/
https://europepmc.org/articles/PMC3838465/