Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Marisa Noemí Sepúlveda"'
Autor:
Luisa Fernanda González Arbeláez, Marisa Noemí Sepúlveda, Martin Vila Petroff, Alejandro Ciocci Pardo, Susana M. Mosca, Juan Ignacio Burgos
Publikováno v:
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Journal of Cellular and Molecular Medicine
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Journal of Cellular and Molecular Medicine
Sepsis is associated with cardiac dysfunction, which is at least in part due to cardiomyocyte apoptosis. However, the underlying mechanisms are far from being understood. Using the colon ascendens stent peritonitis mouse model of sepsis (CASP), we ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::06116dd611a50f8c0ca03414889db2e7
https://hdl.handle.net/11577/3474157
https://hdl.handle.net/11577/3474157
Autor:
Fabiano Ferreira, Adriana Bastos Carvalho, Micaela Lopez Alarcon, Isalira Peroba Ramos, Hugo Justino Branda, Martin Vila Petroff, Emiliano Medei, Claudia N. Paiva, Heitor A. Paula-Neto, Julieta Fernández Ruocco, Marisa Noemí Sepúlveda
Publikováno v:
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Universidad Nacional de La Plata
instacron:UNLP
Hemodynamic collapse and myocardial dysfunction are among the major causes of death in severe sepsis. The purpose of this study was to assess the role played by toll-like receptor 4 and by the NLRP3 inflammasome in the cardiac dysfunction that occurs
Autor:
Amaia Alquiza, Ygor Schleier, Emiliano Medei, Oscar Brito, Rosana A. Bassani, Oscar Casis, Julieta Fernandez-Ruocco, Julian Zayas-Arrabal, Victor Fernández-López, Leyre Echeazarra, Ainhoa Rodriguez-de-Yurre, Marisa Noemí Sepúlveda, Juan M Rodriguez-Robledo, Mónica Gallego, Martin Vila-Petroff, Natália F. Oshiyama
Publikováno v:
Addi. Archivo Digital para la Docencia y la Investigación
instname
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Thyroid
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
instname
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Thyroid
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Background: Hypothyroidism, the most common endocrine disease, induces cardiac electrical remodeling that creates a substrate for ventricular arrhythmias. Recent studies report that high thyrotropin (TSH) levels are related to cardiac electrical abno
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99404a60b3c08d8cf26161e6e275c080
http://europepmc.org/backend/ptpmcrender.fcgi?accid=PMC6648210&blobtype=pdf
http://europepmc.org/backend/ptpmcrender.fcgi?accid=PMC6648210&blobtype=pdf
Autor:
Tom G. Backs, Vila Petroff Martin, Tamara Tottef, Luis Alberto Gonano, Malena Morell, Marisa Noemí Sepúlveda, S. R. Wayne Chen
Publikováno v:
Journal of Molecular and Cellular Cardiology. 98:1-85
Fil: Gonano, Luis Alberto. Consejo Nacional de Investigaciones Cientificas y Tecnicas. Centro Cientifico Tecnologico Conicet - La Plata. Centro de Investigaciones Cardiovasculares "Dr. Horacio Eugenio Cingolani". Universidad Nacional de La Plata. Fac
Autor:
María Florencia Racioppi, Luis Alberto Gonano, Jorge A. Negroni, Malena Morell, Maria Julieta Fernandez Ruocco, Thomas G. Back, S. R. Wayne Chen, Marisa Noemí Sepúlveda, Emiliano Medei, Santiago Gabriel Miriuka, Tamara Toteff, Alicia Mattiazzi, Martin Vila Petroff, Gabriel Neiman, Elena C. Lascano
Publikováno v:
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Universidad Nacional de La Plata
instacron:UNLP
Background It has been shown that carvedilol and its non β-blocking analog, VK-II-86, inhibit spontaneous Ca2+ release from the sarcoplasmic reticulum (SR). The aim of this study is to determine whether carvedilol and VK-II-86 suppress ouabain-induc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bcee899066f2ddc9996005b9f8827af4
http://sedici.unlp.edu.ar/handle/10915/124179
http://sedici.unlp.edu.ar/handle/10915/124179
Autor:
Ignacio Aiello, Martin Vila Petroff, Marisa Noemí Sepúlveda, Verónica Celeste De Giusti, María Sofía Espejo, Ernesto A. Aiello
Publikováno v:
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Universidad Nacional de La Plata
instacron:UNLP
The force-frequency relationship (FFR) is an important intrinsic regulatory mechanism of cardiac contractility. However, a decrease (negative FFR) or no effect (flat FFR) on contractile force in response to an elevation of heart rate is present in th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::92e63dc5e4b09568761af4b5096de256
http://sedici.unlp.edu.ar/handle/10915/144580
http://sedici.unlp.edu.ar/handle/10915/144580
Autor:
Isalira Peroba Ramos, Marisa Noemí Sepúlveda, Manuel Viotti, Luis Alberto Gonano, Paula Graciela Blanco, Emiliano Medei, Malena Morell, Adriana Bastos Carvalho, Micaela Lopez Alarcon, Martin Vila Petroff
Objectives: Sepsis is associated with cardiac contractile dysfunction attributed to alterations in Ca2+ handling. We examined the subcellular mechanisms involved in sarcoplasmic reticulum Ca2+ loss that mediate altered Ca2+ handling and contractile d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e5a983b21286cfa5908603ef8617499f
https://insights.ovid.com/crossref?an=00003246-201704000-00038
https://insights.ovid.com/crossref?an=00003246-201704000-00038
Autor:
Adriana Bonomo, Adriana Bastos Carvalho, Juan Ignacio Burgos, Guilherme Visconde Brasil, Luiz Ricardo Vasconcellos, Gustavo Monnerat, Bernd K. Fleischmann, Antonio Carlos Carvalho, Oscar Casis, Leonardo H. Travassos, Fabiano F. Dutra, Daniela Malan, Marcelo T. Bozza, Martin Vila-Petroff, Rosana A. Bassani, Marisa Noemí Sepúlveda, Camila Hochman-Mendez, Emiliano Medei, Claudia N. Paiva, Micaela Lopez Alarcon
Publikováno v:
Addi. Archivo Digital para la Docencia y la Investigación
instname
Nature Communications, Vol 7, Iss 1, Pp 1-15 (2016)
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Nature Communications
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
instname
Nature Communications, Vol 7, Iss 1, Pp 1-15 (2016)
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Nature Communications
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Diabetes mellitus (DM) encompasses a multitude of secondary disorders, including heart disease. One of the most frequent and potentially life threatening disorders of DM-induced heart disease is ventricular tachycardia (VT). Here we show that toll-li
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4f85c86bebffec8e04bf872009cf7321
http://sedici.unlp.edu.ar/handle/10915/85910
http://sedici.unlp.edu.ar/handle/10915/85910
Autor:
Marisa Noemí Sepúlveda
Publikováno v:
SEDICI (UNLP)
Universidad Nacional de La Plata
instacron:UNLP
Universidad Nacional de La Plata
instacron:UNLP
El presente trabajo de tesis realizado en un modelo celular de taquicardia crónica inducido por marcapaseo rápido (MR) tuvo como finalidad la comprobación de la hipótesis de que la taquicardia crónica a través del aumento sostenido del calcio (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e30d36bbb7769eea0fe334fc8ee4419e
http://sedici.unlp.edu.ar/handle/10915/44294
http://sedici.unlp.edu.ar/handle/10915/44294
Autor:
Martin Vila Petroff, S. R. Wayne Chen, Marisa Noemí Sepúlveda, Thomas G. Back, Luis Alberto Gonano
Tachycardia promotes cell death and cardiac remodeling, leading to congestive heart failure. However, the underlying mechanism of tachycardia- or rapid pacing (RP)-induced cell death remains unknown. Myocyte loss by apoptosis is recognized as a criti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7b82c8b407fa02b18b450fea2422eea3
http://www.sciencedirect.com/science/article/pii/S0022282813002460
http://www.sciencedirect.com/science/article/pii/S0022282813002460