Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Marisa J Lang"'
Autor:
Jonathan J. Petrocelli, Naomi M.M.P. de Hart, Marisa J. Lang, Elena M. Yee, Patrick J. Ferrara, Dennis K. Fix, Amandine Chaix, Katsuhiko Funai, Micah J. Drummond
Publikováno v:
Aging. 15:1808-1832
Autor:
Patrick J Ferrara, Marisa J Lang, Jordan M Johnson, Shinya Watanabe, Kelsey L McLaughlin, John Alan Maschek, Anthony R P Verkerke, Piyarat Siripoksup, Amandine Chaix, James E Cox, Kelsey H Fisher-Wellman, Katsuhiko Funai
Publikováno v:
Life Metab
Weight loss from an overweight state is associated with a disproportionate decrease in whole-body energy expenditure that may contribute to the heightened risk for weight regain. Evidence suggests that this energetic mismatch originates from lean tis
Autor:
Jordan M. Johnson, Alek D. Peterlin, Enrique Balderas, Elahu G. Sustarsic, J. Alan Maschek, Marisa J. Lang, Alejandro Jara-Ramos, Vanja Panic, Jeffrey T. Morgan, Claudio J. Villanueva, Alejandro Sanchez, Jared Rutter, Irfan J. Lodhi, James E. Cox, Kelsey H. Fisher-Wellman, Dipayan Chaudhuri, Zachary Gerhart-Hines, Katsuhiko Funai
Publikováno v:
Johnson, J M, Peterlin, A D, Balderas, E, Sustarsic, E G, Maschek, J A, Lang, M J, Jara-Ramos, A, Panic, V, Morgan, J T, Villanueva, C J, Sanchez, A, Rutter, J, Lodhi, I J, Cox, J E, Fisher-Wellman, K H, Chaudhuri, D, Gerhart-Hines, Z & Funai, K 2023, ' Mitochondrial phosphatidylethanolamine modulates UCP1 to promote brown adipose thermogenesis ', Science Advances, vol. 9, no. 8, eade7864 . https://doi.org/10.1126/sciadv.ade7864
Thermogenesis by uncoupling protein 1 (UCP1) is one of the primary mechanisms by which brown adipose tissue (BAT) increases energy expenditure. UCP1 resides in the inner mitochondrial membrane (IMM), where it dissipates membrane potential independent
Autor:
Patrick J. Ferrara, Marisa J. Lang, Jordan M. Johnson, Shinya Watanabe, Kelsey L. McLaughlin, J. Alan Maschek, Anthony R.P. Verkerke, Piyarat Siripoksup, Amandine Chaix, James E. Cox, Kelsey H. Fisher-Wellman, Katsuhiko Funai
Weight loss is associated with a disproportionate decrease in whole-body energy expenditure that may contribute to the heightened risk for weight-regain. Evidence suggests that this energetic mismatch originates from lean tissue. Although this phenom
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::97aa369535ea4fb5a64c3fba983fc737
https://doi.org/10.1101/2022.12.21.521461
https://doi.org/10.1101/2022.12.21.521461
Autor:
Piyarat Siripoksup, Guoshen Cao, Ahmad A. Cluntun, J. Alan Maschek, Quentinn Pearce, Marisa J. Lang, Hiroaki Eshima, Precious C. Opurum, Ziad S. Mahmassani, Eric B. Taylor, James E. Cox, Micah J. Drummond, Jared Rutter, Katsuhiko Funai
Carbohydrates and lipids provide the majority of substrates to fuel mitochondrial oxidative phosphorylation (OXPHOS). Metabolic inflexibility, defined as an impaired ability to switch between these fuels, is implicated in a number of metabolic diseas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cafb537be8fbd37d9784a551b231f260
https://doi.org/10.1101/2022.11.01.514767
https://doi.org/10.1101/2022.11.01.514767
Autor:
Jordan M. Johnson, Alek D. Peterlin, Enrique Balderas, Elahu G. Sustarsic, J. Alan Maschek, Marisa J. Lang, Alejandro Jara-Ramos, Vanja Panic, Jeffrey T. Morgan, Claudio J. Villanueva, Alejandro Sanchez, Jared Rutter, Irfan J. Lodhi, James E. Cox, Kelsey H. Fisher-Wellman, Dipayan Chaudhuri, Zachary Gerhart-Hines, Katsuhiko Funai
SUMMARYThermogenesis by uncoupling protein 1 (UCP1) is one of the primary mechanisms by which brown adipose tissue (BAT) increases energy expenditure. UCP1 resides in the inner mitochondrial membrane (IMM), where it dissipates membrane potential inde
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3afd0b0194f91b3062b36437a37a605c
https://doi.org/10.1101/2022.08.26.505474
https://doi.org/10.1101/2022.08.26.505474
Autor:
Maureen Walsh, Dipayan Chaudhuri, Andrea C Corbin, Emma Rekate, Anthony Balynas, David R Eberhardt, Xue Yin, Katsuhiko Funai, Sandra H Lee, Jackie N Kraiss, Marisa J Lang, Molly E. Streiff, Frank B. Sachse
Altered levels of intracellular calcium (Ca2+) is a highly prevalent feature in different forms of cardiac injury, producing changes in contractility, arrhythmias, and mitochondrial dysfunction. In cardiac ischemia-reperfusion injury, mitochondrial C
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::70f465d7b0c93e92f3f7f2f513148570
https://doi.org/10.1101/2021.10.18.464831
https://doi.org/10.1101/2021.10.18.464831
Autor:
David R. Eberhardt, Sandra H. Lee, Xue Yin, Anthony M. Balynas, Emma C. Rekate, Jackie N. Kraiss, Marisa J. Lang, Maureen A. Walsh, Molly E. Streiff, Andrea C. Corbin, Ying Li, Katsuhiko Funai, Frank B. Sachse, Dipayan Chaudhuri
Publikováno v:
J Mol Cell Cardiol
Altered levels of intracellular calcium (Ca(2+)) are a highly prevalent feature in different forms of cardiac injury, producing changes in contractility, arrhythmias, and mitochondrial dysfunction. In cardiac ischemia-reperfusion injury, mitochondria