Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Marina L. H. Honing"'
Autor:
Laurent Petit, Peter J. van Horssen, Udo Kaisers, Harvey B. M. Fijn, François Sztark, Jean Louis Vincent, Marina L. H. Honing, Arthur R. H. van Zanten, Jean-Charles Preiser, Jean-François Timsit, Zandrie Hofman, Didier Keh, Siegfried Zielmann, Jean-Fabien Zazzo, T.W. Felbinger, Jan J. De Waele, Armin Sablotzki
Publikováno v:
JAMA. 312(5)
Importance Enteral administration of immune-modulating nutrients (eg, glutamine, omega-3 fatty acids, selenium, and antioxidants) has been suggested to reduce infections and improve recovery from critical illness. However, controversy exists on the u
Autor:
Marina L. H. Honing, Ton J. Rabelink, Paul J. Morrison, Yonghong P. Yang, Robert J. Padley, David E. Ballard, Michel L. Hijmering
Publikováno v:
Journal of the American Society of Nephrology. 11:1498-1504
Endothelin (ET-1) acts as a potent vasoconstrictor in the human kidney, and this vasoconstriction could contribute to the ischemia seen in acute renal failure. In animal studies, the vasoactive properties of ET-1 are known to be ET A receptor-and/or
Autor:
Ton J. Rabelink, Robert J. Padley, Amy Y. Grahn, Anton W. M. Van Weerdt, Marina L. H. Honing, Yonghong P. Yang, Marianne C. Verhaar, Paul J. Morrison
Publikováno v:
British Journal of Clinical Pharmacology. 49:562-573
Aims Endothelins (ETs) may play a role in the pathogenesis of a variety of cardiovascular diseases. The present study was designed to investigate the pharmacokinetic and pharmacodynamic effects of the orally active ETA selective receptor antagonist A
Autor:
Hendrik A. Koomans, Ton J. Rabelink, Marina L. H. Honing, T. Van Dam, J.J.P. Kastelein, M. Zwart, Marianne C. Verhaar
Publikováno v:
Cardiovascular research, 42(3), 752-760. Oxford University Press
Objective: Dihydropyridine calcium antagonists have been shown to retard atherogenesis in animal models and to prevent the development of early angiographic lesions in human coronary arteries. Endothelial dysfunction is an early event in the pathogen
Publikováno v:
Diabetes / Metabolism Reviews. 14:241-249
Diabetes mellitus is associated with early development of cardiovascular complications. Under physiological conditions the endothelium protects against the development of atherosclerosis. Endothelial cells produce, e.g., nitric oxide (NO), a substanc
Publikováno v:
Clinical Science, 106(4), 345-346. Portland Press Ltd.
Can mild-to-moderate hyperhomocysteinaemia impair endothelial function in the absence of other risk factors for cardiovascular disease?Honing ML, Stehouwer CD.Department of Internal Medicine, VU University Medical Centre, PO Box 7057, 1007 MB Amsterd
Autor:
Ton J. Rabelink, Ronald W. van Etten, Marina L. H. Honing, Erik S. G. Stroes, Eelco J.P. de Koning, Carlo A. J. M. Gaillard
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology, 22(5), 799-804. Lippincott Williams and Wilkins
Cardiovascular disease is the most important cause of morbidity and mortality in patients with type 2 diabetes. Endothelial dysfunction predicts cardiovascular outcome. Type 2 diabetes is characterized by endothelial dysfunction, which may be caused
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b6fb2e2c56eba09d5752f8d0560a278
https://pure.amc.nl/en/publications/intensive-lipid-lowering-by-statin-therapy-does-not-improve-vasoreactivity-in-patients-with-type-2-diabetes(03231ed2-950f-417d-aa3b-40b79b8bf0db).html
https://pure.amc.nl/en/publications/intensive-lipid-lowering-by-statin-therapy-does-not-improve-vasoreactivity-in-patients-with-type-2-diabetes(03231ed2-950f-417d-aa3b-40b79b8bf0db).html
Publikováno v:
Hypertension, 37, 1179-83
ResearcherID
Hypertension, 37, 4, pp. 1179-83
ResearcherID
Hypertension, 37, 4, pp. 1179-83
Abstract —Animal studies have demonstrated that CNP causes endothelium-independent vasodilation, which is limited by neutral endopeptidase (NEP) activity. However, the vasodilating mechanism of CNP in humans is still unknown. Therefore, we investig
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b0c14b9213d053dc92388a0947f663cd
http://hdl.handle.net/2066/185576
http://hdl.handle.net/2066/185576
Publikováno v:
Hypertension, 35, pp. 1314-1318
Hypertension, 35, 1314-1318
Hypertension, 35, 1314-1318
Abstract —Bradykinin (BK) stimulates endothelial cells to release a number of relaxing factors, such as NO, prostanoids (PGs), and an endothelium-derived hyperpolarizing factor (EDHF). However, the contributions of NO, PG, and EDHF in the vascular
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e87cc8b5fe9cd99393a68f652f1dbdd4
https://hdl.handle.net/2066/185877
https://hdl.handle.net/2066/185877
Autor:
van Zanten, Arthur R. H., Sztark, François, Kaisers, Udo X., Zielmann, Siegfried, Felbinger, Thomas W., Sablotzki, Armin R., De Waele, Jan J., Timsit, Jean-Frangois, Honing, Marina L. H., Keh, Didier, Vincent, Jean-Louis, Zazzo, Jean-Fabien, Fijn, Harvey B. M., Petit, Laurent, Preiser, Jean-Charles, van Horssen, Peter J., Hofman, Zandrie
Publikováno v:
JAMA: Journal of the American Medical Association; 8/6/2014, Vol. 312 Issue 5, p514-524, 11p