Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Marina Izvolskaia"'
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 21, p 15983 (2023)
Maternal immune activation results in altered levels of cytokines in the maternal–fetal system, which has a negative impact on fetal development, including the gonadotropin-releasing hormone (GnRH) system, which is crucial for the reproduction. Sup
Externí odkaz:
https://doaj.org/article/39f0657f87bd4e179fab005fbe1420d4
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 3, p 2726 (2023)
The development of the neuroendocrine system, including the hypothalamic–pituitary–gonadal (HPG) axis, is sensitive to environmental impacts during critical developmental periods. Maternal immune system activation by bacterial or viral infection
Externí odkaz:
https://doaj.org/article/da412ab75de34427909e15ad3bdea300
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 1, p 114 (2020)
Different aspects of the reciprocal regulatory influence on the development of gonadotropin-releasing hormone (GnRH)-producing- and immune systems in the perinatal ontogenesis and their functioning in adults in normal and pathological conditions are
Externí odkaz:
https://doaj.org/article/0082b3de8a584c18a37763b09d1da0ac
Publikováno v:
International Journal of Molecular Sciences, Vol 19, Iss 11, p 3695 (2018)
Various stress factors during critical periods of fetal development modulate the epigenetic mechanisms controlling specific genes, which can affect the structure and function of physiological systems. Maternal immune stress by bacterial infection sim
Externí odkaz:
https://doaj.org/article/beb99e2f500d42a9b15f5a5e0f8b748c
Publikováno v:
Reproduction. 161:669-679
Sexual performance in adult male rats is highly sensitive to prenatal stress which can affect the functionality of the reproductive system and various brain structures involved in modulating sexual behavior. The immunomodulatory effect of mouse IgG o
Publikováno v:
Inflammation. 43:1175-1183
Viral and bacterial infections causing systemic inflammation are significant risk factors for developing body. Inflammatory processes can alter physiological levels of regulatory factors and interfere with developmental mechanisms. The brain is the m
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 114, p 114 (2021)
International Journal of Molecular Sciences
International Journal of Molecular Sciences
Different aspects of the reciprocal regulatory influence on the development of gonadotropin-releasing hormone (GnRH)-producing- and immune systems in the perinatal ontogenesis and their functioning in adults in normal and pathological conditions are
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 19, Iss 11, p 3695 (2018)
International Journal of Molecular Sciences, Vol 19, Iss 11, p 3695 (2018)
Various stress factors during critical periods of fetal development modulate the epigenetic mechanisms controlling specific genes, which can affect the structure and function of physiological systems. Maternal immune stress by bacterial infection sim
Autor:
L. A. Zakharova, Marina Izvolskaia, S. N. Voronova, Victoria S. Sharova, Vasilina M. Ignatiuk
Publikováno v:
Andrologia. 51(3)
During prenatal and early postnatal periods of development, multiple environmental factors have profound and long-lasting effects on the immune and reproductive functions. The aim of this study was to investigate the effects of maternal lipopolysacch
Publikováno v:
Stress
Stress, Informa Healthcare, 2016, 19 (2), pp.198-205. ⟨10.3109/10253890.2016.1149695⟩
Stress, Informa Healthcare, 2016, 19 (2), pp.198-205. ⟨10.3109/10253890.2016.1149695⟩
Postnatal treatment with bacterial endotoxin lipopolysaccharide (LPS) changes the activity of the hypothalamic-pituitary-gonadal (HPG) axis and the gonadotropin-releasing hormone (GnRH) surge in rats. Exposure to an immune challenge in the critical p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bc8fb56739d94a81dc05ece04c04b9cc
https://hal.archives-ouvertes.fr/hal-01353263
https://hal.archives-ouvertes.fr/hal-01353263