Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Marina Fernandez-Alvarez"'
Publikováno v:
Translational Psychiatry, Vol 13, Iss 1, Pp 1-9 (2023)
Abstract Amyloid-beta (Aβ) aggregates and myelin breakdown are among the earliest detrimental effects of Alzheimer’s disease (AD), likely inducing abnormal patterns of neuronal communication within cortical networks. However, human in vivo evidenc
Externí odkaz:
https://doaj.org/article/b8c575864bb24fe79e460c3bbea5389a
Publikováno v:
Alzheimer’s Research & Therapy, Vol 14, Iss 1, Pp 1-13 (2022)
Abstract Background Non-modifiable risk factors of Alzheimer’s disease (AD) have lifelong effects on cortical integrity that could be mitigated if identified at early stages. However, it remains unknown whether cortical microstructure is affected i
Externí odkaz:
https://doaj.org/article/36531a0980754b82a269ca21bcc16c31
Autor:
Karel M. Lopez-Vilaret, Marina Fernandez-Alvarez, Ehsan Shokri-Kojori, Dardo Tomasi, Jose L. Cantero, Mercedes Atienza
Publikováno v:
Frontiers in Aging Neuroscience, Vol 14 (2022)
Insulin resistance and glucose dysregulation are associated with patterns of regional brain hypometabolism characteristic of Alzheimer’s disease (AD). As predicted by evidence linking brain glucose metabolism to brain functional connectivity, type
Externí odkaz:
https://doaj.org/article/e57bdd3ac3cc4bd7a6b22b4b93fed248
Publikováno v:
Alzheimer’s Research & Therapy, Vol 13, Iss 1, Pp 1-12 (2021)
Abstract Background Aging is associated with declining protective immunity and persistent low-grade inflammatory responses, which significantly contribute to Alzheimer’s disease (AD) pathogenesis. Detecting aging-related cerebral vulnerability asso
Externí odkaz:
https://doaj.org/article/c65a1a65c74547b8acc708e919a7031b
Autor:
Marina Fernandez-Alvarez, Mercedes Atienza, Fatima Zallo, Carlos Matute, Estibaliz Capetillo-Zarate, Jose L. Cantero
Publikováno v:
Frontiers in Aging Neuroscience, Vol 14 (2022)
Evidence suggests that lightly myelinated cortical regions are vulnerable to aging and Alzheimer’s disease (AD). However, it remains unknown whether plasma markers of amyloid and neurodegeneration are related to deficits in intracortical myelin con
Externí odkaz:
https://doaj.org/article/0a64722548744b8ba73e64098125a30d
Publikováno v:
Alzheimer’s Research & Therapy, Vol 13, Iss 1, Pp 1-12 (2021)
Alzheimer's Research & Therapy
Alzheimer's Research & Therapy
Background Aging is associated with declining protective immunity and persistent low-grade inflammatory responses, which significantly contribute to Alzheimer’s disease (AD) pathogenesis. Detecting aging-related cerebral vulnerability associated wi