Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Marilda L. A De Maria"'
Autor:
Tatiane Cristine Silva de Almeida, Maria Aparecida Ribeiro Vieira, Roberta da Silva Filha, Anderson J. Ferreira, Ana Cristina Simões e Silva, Thiago Ruiz Rodrigues Prestes, Lucas M. Kangussu, Marilda L. A De Maria
Publikováno v:
Protein and peptide letters. 26(7)
Background:Angiotensin Converting Enzyme (ACE) 2 is an important modulator of the Renin Angiotensin System (RAS) and the RAS plays a central role in renovascular hypertension. Very few studies investigated the role of components of the counterregulat
Autor:
Anderson José Ferreira, Elizabeth Pereira Mendes, Diego Basile Colugnati, Clayton Luiz Borges, Robson A.S. Santos, Célia Maria de Almeida Soares, Marilda L. A De Maria, Gustavo Rodrigues Pedrino, Álvaro P. S. Souza, Carlos H. Castro, Larissa Matuda Macedo
Publikováno v:
Life Sciences. 155:63-69
Aims Angiotensin-converting enzyme 2 (ACE2) is a key modulator of the renin-angiotensin system. Recent studies have shown that diminazene aceturate (DIZE) acts as an ACE2 activator. The aim of this study was to evaluate the cardiac effects of chronic
Autor:
Gustavo B. Menezes, Rodrigo A. Fraga-Silva, Letícia A.S. Pereira, Liliane Diniz de Araújo, Anderson J. Ferreira, Heder José Ribeiro, Vinayak Shenoy, Marilda L. A De Maria, Mohan K. Raizada
Publikováno v:
Protein & Peptide Letters. 23:9-16
Previous studies have shown that activation of endogenous angiotensin-converting enzyme 2 (ACE2) results in various beneficial effects in the cardiovascular system. Recently, a new ACE2 activator, named diminazene aceturate (DIZE), was described. Her
Autor:
Silvia Guatimosim, Danielle Carvalho Oliveira Coutinho, Tatiane M. Murça, Enéas Ricardo de Morais Gomes, Patrícia L. Moraes, Marilda L. A De Maria, Giselle Foureaux, Robson A.S. Santos, Anderson J. Ferreira, Rodrigo Lolli Almeida Salles, Keyla D L Rodrigues
Publikováno v:
Journal of the Renin-Angiotensin-Aldosterone System, Vol 15 (2014)
Introduction: Angiotensin (Ang) A was first identified in human plasma and it differs from Ang II in Ala1 instead of Asp1. Here, we hypothesized that the actions of this peptide might explain, at least partially, the limited effects of AT 1 R antago-