Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Marijke A.E. Mol"'
Autor:
A. Josien Smits, Liza Botros, Marijke A.E. Mol, Kirsten A. Ziesemer, Martin R. Wilkins, Anton Vonk Noordegraaf, Harm Jan Bogaard, Jurjan Aman
Publikováno v:
ERJ Open Research, Vol 8, Iss 2 (2022)
Rationale The blood is a rich source of potential biomarkers for the diagnosis of idiopathic and hereditary pulmonary arterial hypertension (iPAH and hPAH, referred to as “PAH”). While a lot of biomarkers have been identified for PAH, the clinica
Externí odkaz:
https://doaj.org/article/0c49ba69f8b34608a326b234b27d1090
Autor:
Ingrid Anne Lie, Merlin M. Weeda, Rozemarijn M. Mattiesing, Marijke A.E. Mol, Petra J.W. Pouwels, Frederik Barkhof, Øivind Torkildsen, Lars Bø, Kjell-Morten Myhr, Hugo Vrenken
Publikováno v:
Neurology. 98(15)
Background and ObjectivesThere is currently no consensus about the extent of gray matter (GM) atrophy that can be attributed to secondary changes after white matter (WM) lesions or the temporal and spatial relationships between the 2 phenomena. Eluci
Publikováno v:
Toxicology and Applied Pharmacology. 107:439-449
Blister formation due to sulfur mustard (HD) exposure was studied in an ex vivo human skin model. Pieces of fresh human skin were exposed to HD vapor and subsequently incubated in medium for 48 hr. During this culture period cellular NAD + levels and
Autor:
Wiete Westerhof, Jan-Paul van Eendenburg, Marijke A.E. Mol, P. Bart Nanninga, Jan R. Mekkes, Cornelis J.W. van Ginkel
Publikováno v:
Journal of the American Academy of Dermatology. 24:77-82
Skin equivalents that consisted of a noncontracted collagen gel populated with allogeneic fibroblasts and covered with autologous cultured keratinocytes were used for grafting venous leg ulcers. The results were compared in the same patient with thos
Ca2+ homeostasis and Ca2+ signalling in sulphur mustard-exposed normal human epidermal keratinocytes
Autor:
William J. Smith, Marijke A.E. Mol
Publikováno v:
Chemico-biological interactions. 100(1)
To investigate the role of increased cytoplasmic free calcium levels ([Ca] i ) in sulphur mustard-induced cellular toxicity, the [Ca] i of human epidermal keratinocytes exposed to sulphur mustard concentrations between 50 and 800 μM were measured. N
Publikováno v:
Toxicology and applied pharmacology. 98(1)
In cultured human epidermal cells exposure to the vesicant sulfur mustard (HD) causes a decrease of the NAD+ content, which depends on the dose and the time period between exposure to HD and NAD+ measurement. Presumably, this NAD+ loss is due to acti