Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Mariella Bockstahler"'
Autor:
Hugo A. Katus, Andrea Kübler, Christian Salbach, Oliver J. Müller, Anna-Maria Müller, Ziya Kaya, Norbert Frey, Mariella Bockstahler
Publikováno v:
Immunology. 165:158-170
Treatment of myocarditis is often limited to symptomatic treatment due to unknown pathomechanisms. In order to identify new therapeutic approaches, the contribution of locked nucleic acid antisense oligonucleotides (LNA ASOs) in autoimmune myocarditi
Autor:
Hugo A. Katus, Anna-Maria Müller, Andrea Fischer, Ziya Kaya, Mariella Bockstahler, Vera Stroikova
Publikováno v:
Cytokine. 116:139-149
Background Adiponectin is a hormone that together with its receptors modulates a number of metabolic processes including gluconeogenesis and lipid catabolism. It belongs to the C1QTNF (complement C1q tumor necrosis factor-related protein) family, whi
Autor:
Hannah Louise Neumaier, Christin Meckes, Christian Salbach, Meike Kespohl, Ulf Landmesser, Carl Christoph Goetzke, Benjamin Meder, Antje Beling, Lorenz H. Lehmann, January Weiner, Andrea Fischer, Adelheid Kratzer, Hugo A. Katus, Ziya Kaya, Anna-Maria Müller, Karin Klingel, Mirjam Schenk, Arnd Heuser, Mariella Bockstahler, Martina Sauter
Publikováno v:
Bockstahler, Mariella; Fischer, Andrea; Goetzke, Carl Christoph; Neumaier, Hannah Louise; Sauter, Martina; Kespohl, Meike; Müller, Anna-Maria; Meckes, Christin; Salbach, Christian; Schenk, Mirjam; Heuser, Arnd; Landmesser, Ulf; Weiner, January; Meder, Benjamin; Lehmann, Lorenz; Kratzer, Adelheid; Klingel, Karin; Katus, Hugo A.; Kaya, Ziya and Beling, Antje (2020). Heart-Specific Immune Responses in an Animal Model of Autoimmune-Related Myocarditis Mitigated by an Immunoproteasome Inhibitor and Genetic Ablation. Circulation, 141(23), pp. 1885-1902. American Heart Association 10.1161/circulationaha.119.043171
Background: Immune checkpoint inhibitor (ICI) therapy is often accompanied by immune-related pathology, with an increasing occurrence of high-risk ICI-related myocarditis. Understanding the mechanisms involved in this side effect could enable the dev
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a750717bbca372c1b2e77483b5a8a05
http://edoc.mdc-berlin.de/18806/7/18806suppl.zip
http://edoc.mdc-berlin.de/18806/7/18806suppl.zip
Autor:
Mariella Bockstahler, Vera Stroikova, Ziya Kaya, Christoph Leib, Andrea Fischer, Anna-Maria Müller, Gabriele Pfitzer, Hugo A. Katus
Publikováno v:
Journal of cardiac failure. 25(8)
Background The pathogenesis of inflammatory cardiomyopathy is affected by the activation of autoimmune-mediated cascades. To study these cascades, we developed an experimental model of troponin I (TnI)-induced autoimmune myocarditis (EAM). One factor