Výsledky vyhledávání - "Marie-Annick Buendia"

Akademický článek

Deficiency of the LIM-only protein FHL2 reduces intestinal tumorigenesis in Apc mutant mice.

Autor: Charlotte Labalette, Yann Nouët, Florence Levillayer, Sabine Colnot, Ju Chen, Valere Claude, Michel Huerre, Christine Perret, Marie-Annick Buendia, Yu Wei

Publikováno v: PLoS ONE, Vol 5, Iss 4, p e10371 (2010)

BACKGROUND: The four and a half LIM-only protein 2 (FHL2) is capable of shuttling between focal adhesion and nucleus where it signals through direct interaction with a number of proteins including beta-catenin. Although FHL2 activation has been found

Externí odkaz: https://doaj.org/article/f00a7229ed1f4d32bfd0bdd3c5b828b8

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Akademický článek

The LIM-only protein FHL2 mediates ras-induced transformation through cyclin D1 and p53 pathways.

Autor: Charlotte Labalette, Yann Nouët, Florence Levillayer, Carolina Armengol, Claire-Angélique Renard, Guillaume Soubigou, Tian Xia, Marie-Annick Buendia, Yu Wei

Publikováno v: PLoS ONE, Vol 3, Iss 11, p e3761 (2008)

BACKGROUND: Four and a half LIM-only protein 2 (FHL2) has been implicated in multiple signaling pathways that regulate cell growth and tissue homeostasis. We reported previously that FHL2 regulates cyclin D1 expression and that immortalized FHL2-null

Externí odkaz: https://doaj.org/article/21b31204e8624d13a8fa210a32dc192c

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Data from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Autor: Marie-Annick Buendia, Christine Perret, Anne Dejean, Aurélien de Reyniès, Christine Neuveut, Pascal Pineau, Stefano Cairo, Yu Wei, Delphine Cougot, Carolina Armengol, Charlotte Labalette, Claire-Angélique Renard

Tbx3 encodes a transcriptional repressor that is important for diverse patterning events during development, and Tbx3 mutation in humans causes the ulnar-mammary syndrome. Here, we describe the identification of Tbx3 in array-based search for genes d

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d711f96fab95c91b5a6059e548810545
https://doi.org/10.1158/0008-5472.c.6495053

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Supplementary Table 2 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Supplementary Table 2 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9e940d9c65a1befb4491fb7de9c4a50d
https://doi.org/10.1158/0008-5472.22366241

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Supplementary Table 3 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Supplementary Table 3 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e9baebe5ee617e4c33f73b8a60420f97
https://doi.org/10.1158/0008-5472.22366238

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Supplementary Figure Legends 1-4 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Supplementary Figure Legends 1-4 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::41d80783ab6a0aabe07d27cf486daa37
https://doi.org/10.1158/0008-5472.22366247

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Supplementary Figures 1-4 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Supplementary Figures 1-4 from Tbx3 Is a Downstream Target of the Wnt/β-Catenin Pathway and a Critical Mediator of β-Catenin Survival Functions in Liver Cancer

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::44fc9a9fccadad63efc956939327ba4d
https://doi.org/10.1158/0008-5472.22366244.v1

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A combined clinical and biological risk classification improves prediction of outcome in hepatoblastoma patients

Autor: Catherine Guettier, Roland Kappler, Beate Häberle, Marie-Annick Buendia, Sophie Branchereau, Kristina Becker, Rudolf Maibach, Carolina Armengol, Dietrich von Schweinitz, Irene Schmid, Christian Vokuhl, Stefano Cairo, Juan Carrillo-Reixach

Publikováno v: European Journal of Cancer
r-IGTP. Repositorio Institucional de Producción Científica del Instituto de Investigación Germans Trias i Pujol
instname

Aim: Stratification of hepatoblastoma (HB) patients is based on clinical and imaging characteristics obtained at the time of diagnosis. We aim to integrate biomarkers into a tool that accurately predicts survival of HB patients. Methods: We retrospec

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f9ba70be490f45e24bccf89c969fdcad
https://doi.org/10.1016/j.ejca.2020.09.026

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Abstract PO011: Proteomic profiling of childhood liver cancer: identification of novel diagnostic and prognostic biomarkers

Publikováno v: Clinical Cancer Research. 28:PO011-PO011

Childhood liver cancers hepatoblastoma (HB) and hepatocellular carcinoma (HCC) are rare diseases but with a rising incidence. HEMNOS (Hepatocellular malignant neoplasm, not otherwise specified) is a recent entity with histopathological features of HB

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::f5d385588a2ee5167df7e89732509eda
https://doi.org/10.1158/1557-3265.liverca22-po011

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Epigenetic footprint enables molecular risk stratification of hepatoblastoma with clinical implications

Autor: Cristina Beléndez, Viera Bajčiová, Nicholas K. Akers, Aroa Soriano, David Piñeyro, Manuel López Santamaría, Michael A. Grotzer, Carolina Armengol, José Antonio Salinas, Lara Nonell, Mar Mallo, Jordi Abril-Fornaguera, Bruce Morland, Roland Kappler, Monique Fabre, Josep M. Llovet, Ramon Planas, Helena Masnou, Piotr Czauderna, María Elena Mateos, Constantino Sábado, Genevieve Laureys, Catherine Guettier, Ricardo López-Almaraz, Claudia Paris, Maria Rosa Sarrias, Montserrat Domingo-Sàbat, Yasmina Mozo, Olga Kuchuk, Marta Garrido, José Javier Uriz, Laura Torrens, Stefano Cairo, Julià Blanco, Gabriela Guillén, Blanca López-Ibor, Sophie Branchereau, Francisco Andrés Pérez Hernández, Daniela Sia, Bojan Losic, Bárbara Torres, Magdalena Arnal, Laura Guerra, Margarita Sala, Laura Royo, Maria Vázquez-Vitali, Gema Ramírez, Núria Villalmanzo, Alberto Villanueva, Ariadna Clos, Mireia Jordà, Nagore García de Andoin, Marina Simon-Coma, Marie Annick Buendia, Juan Carrillo-Reixach, Lauro Sumoy, Sonia Ragull

Publikováno v: Dipòsit Digital de la UB
Universidad de Barcelona
Journal of Hepatology
r-IGTP. Repositorio Institucional de Producción Científica del Instituto de Investigación Germans Trias i Pujol
instname
Dipòsit Digital de Documents de la UAB
Universitat Autònoma de Barcelona
JOURNAL OF HEPATOLOGY
r-IIS La Fe. Repositorio Institucional de Producción Científica del Instituto de Investigación Sanitaria La Fe

Background & Aims: Hepatoblastoma (HB) is a rare disease. Nevertheless, it is the predominant pediatric liver cancer, with limited therapeutic options for patients with aggressive tumors. Herein, we aimed to uncover the mechanisms of HB pathobiology

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5eda32a593d433d3874ce90c0752929b
http://hdl.handle.net/2445/171912

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