Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Marie Sanson"'
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e74676 (2013)
Our lab has previously shown in a mouse model that normalization of a low HDL level achieves atherosclerotic plaque regression. This included the shift from a pro ("M1") to an anti-inflammatory ("M2") phenotypic state of plaque macrophages. Whether H
Externí odkaz:
https://doaj.org/article/f452b087034e4ba98a4bcd0e714b5cb0
Autor:
Lisa Grauer, Ira J. Goldberg, Marie Sanson, Saj Parathath, Edward A. Fisher, Emilie Distel, Li-Shin Huang
Publikováno v:
Diabetes
OBJECTIVE Patients with diabetes have increased cardiovascular risk. Atherosclerosis in these patients is often associated with increased plaque macrophages and dyslipidemia. We hypothesized that diabetic atherosclerosis involves processes that impai
Autor:
Chao Ling Kuo, Andrea E. Bochem, Laurent Yvan-Charvet, Nora Bijl, Marie Sanson, Sonia Tolani, Mi Wang, Sandra Abramowicz, Mani Akhtari, Andrew J. Murphy, Tamara A. Pagler, Carrie L. Welch, Alan R. Tall, Jan Albert Kuivenhoven
Publikováno v:
The journal of clinical investigation, 121(10), 4138-4149. The American Society for Clinical Investigation
Journal of clinical investigation, 121(10), 4138-4149. The American Society for Clinical Investigation
Journal of clinical investigation, 121(10), 4138-4149. The American Society for Clinical Investigation
Leukocytosis is associated with increased cardiovascular disease risk in humans and develops in hypercholesterolemic atherosclerotic animal models. Leukocytosis is associated with the proliferation of hematopoietic stem and multipotential progenitor
Autor:
Ines Pineda-Torra, Peter Tontonoz, Michelle N. Bradley, Daniel Rubinstein, Edward A. Fisher, Marie Sanson, Chaowei Wu, Dusan Bogunovic, Michael J. Garabedian, Yuliya Vengrenyuk, Jianhua Liu, Cynthia Hong, Nico van Rooijen, Emmanuel L. Gautier, Nina Bhardwaj, Jonathan E. Feig
Publikováno v:
Feig, J E, Pineda-Torra, I, Sanson, M, Bradley, M N, Vengrenyuk, Y, Bogunovic, D, Gautier, E L, Rubinstein, D, Hong, C, Liu, J H, Wu, C W, van Rooijen, N, Bhardwaj, N, Garabedian, M J, Tontonoz, P & Fisher, E A 2010, ' LXR promotes the maximal egress of monocyte-derived cells from mouse aortic plaques during atherosclerosis regression ', Journal of Clinical Investigation, vol. 120, no. 12, pp. 4415-4424 . https://doi.org/10.1172/JCI38911
Journal of Clinical Investigation, 120(12), 4415-4424. The American Society for Clinical Investigation
Journal of Clinical Investigation, 120(12), 4415-4424. The American Society for Clinical Investigation
We have previously shown that mouse atherosclerosis regression involves monocyte-derived (CD68+) cell emigration from plaques and is dependent on the chemokine receptor CCR7. Concurrent with regression, mRNA levels of the gene encoding LXRalpha are i
Autor:
Hideru Obinata, Björn Dahlbäck, Timothy Hla, Jonathan D. Smith, Victoria A. Blaho, Sylvain Galvani, Steven L. Swendeman, Marie Sanson, Mari Kono, Richard L. Proia, Heather Conger
The sphingosine 1-phosphate receptor 1 (S1P1) is abundant in endothelial cells, where it regulates vascular development and microvascular barrier function. In investigating the role of endothelial cell S1P1 in adult mice, we found that the endothelia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2a73c997c2150facaa03d8a888efb61
https://europepmc.org/articles/PMC4768813/
https://europepmc.org/articles/PMC4768813/
Autor:
Denis Pinsard, Françoise Brucker-Davis, Felix Beuschlein, Francoise Doullay, Bruno Ragazzon, Françoise Borson-Chazot, Stephanie Lopez, Lionel Groussin, Jérôme Bertherat, Marie Sanson, G. Barrande, Guillaume Assié, W. Luscap, Florence Torremocha, Rossella Libe, Xavier Bertagna, Stéphanie Espiard, Emmanuel Sonneta, Nathalie Chabert-Buffet, Laurence Guignat
Publikováno v:
Endocrine Abstracts.
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 8, p e74676 (2013)
PLoS ONE, Vol 8, Iss 8, p e74676 (2013)
Our lab has previously shown in a mouse model that normalization of a low HDL level achieves atherosclerotic plaque regression. This included the shift from a pro ("M1") to an anti-inflammatory ("M2") phenotypic state of plaque macrophages. Whether H
Autor:
Maya Lodish, Maria Nesterova, Amato Fratticci, Isaak Levy, Anelia Horvath, Delphine Vezzosi, Fabio R. Faucz, Monalisa F. Azevedo, Constantine A. Stratakis, Rossella Libé, Anya Rothenbuhler, Marie Sanson, Jérôme Bertherat, Madson Q. Almeida
Publikováno v:
Clinical endocrinology. 77(2)
SummaryBackground Genetic aberrations in various components of cAMP signalling pathway predispose to endocrine tumours. Mutations in the phosphodiesterases (PDEs) are involved in the predisposition to adrenocortical neoplastic conditions. Objective T
Autor:
Jonathan E. Feig, Michael J. Garabedian, Kathryn J. Moore, Raanan Shamir, Marie Sanson, Yuliya Vengrenyuk, Jianhua Liu, James X. Rong, Edward A. Fisher, Katey J. Rayner
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America. 108(17)
HDL cholesterol (HDL-C) plasma levels are inversely related to cardiovascular disease risk. Previous studies have shown in animals and humans that HDL promotes regression of atherosclerosis. We hypothesized that this was related to an ability to prom
Autor:
Cécile Vindis, Yoshio Bando, Anne Nègre-Salvayre, Ziad Mallat, Robert Salvayre, Yoshiki Sawa, Marie Sanson, Yves Glock, Jean-Claude Thiers, Hervé Rousseau, Cécile Ingueneau
Publikováno v:
Cell Death and Differentiation
Cell Death and Differentiation, 2008, 15 (8), pp.1255-65. ⟨10.1038/cdd.2008.36⟩
Cell Death and Differentiation, Nature Publishing Group, 2008, 15 (8), pp.1255-65. ⟨10.1038/cdd.2008.36⟩
Cell Death and Differentiation, 2008, 15 (8), pp.1255-65. ⟨10.1038/cdd.2008.36⟩
Cell Death and Differentiation, Nature Publishing Group, 2008, 15 (8), pp.1255-65. ⟨10.1038/cdd.2008.36⟩
International audience; Oxidized LDLs (oxLDLs) induce apoptosis, which contributes to the pathogenesis of atherosclerosis. The 150 kDa oxygen-regulated protein (ORP150), an endoplasmic reticulum (ER)-resident chaperone, is upregulated by hypoxia and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::11fc78e3807c508ea5fcf94fa9ea9e68
https://www.hal.inserm.fr/inserm-00420356
https://www.hal.inserm.fr/inserm-00420356