Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Marie L Harth-Hertle"'
Autor:
Laura V Glaser, Simone Rieger, Sybille Thumann, Sophie Beer, Cornelia Kuklik-Roos, Dietmar E Martin, Kerstin C Maier, Marie L Harth-Hertle, Björn Grüning, Rolf Backofen, Stefan Krebs, Helmut Blum, Ralf Zimmer, Florian Erhard, Bettina Kempkes
Publikováno v:
PLoS Pathogens, Vol 13, Iss 10, p e1006664 (2017)
Epstein-Barr virus (EBV) infection converts resting human B cells into permanently proliferating lymphoblastoid cell lines (LCLs). The Epstein-Barr virus nuclear antigen 2 (EBNA2) plays a key role in this process. It preferentially binds to B cell en
Externí odkaz:
https://doaj.org/article/9ead8bedc269442990447aa13ffbca21
Autor:
Michael J McClellan, C David Wood, Opeoluwa Ojeniyi, Tim J Cooper, Aditi Kanhere, Aaron Arvey, Helen M Webb, Richard D Palermo, Marie L Harth-Hertle, Bettina Kempkes, Richard G Jenner, Michelle J West
Publikováno v:
PLoS Pathogens, Vol 9, Iss 9, p e1003636 (2013)
Epstein-Barr virus (EBV) epigenetically reprogrammes B-lymphocytes to drive immortalization and facilitate viral persistence. Host-cell transcription is perturbed principally through the actions of EBV EBNA 2, 3A, 3B and 3C, with cellular genes dereg
Externí odkaz:
https://doaj.org/article/f1ec0ab7ce444d78be87a77e27947c93
Autor:
Marie L Harth-Hertle, Barbara A Scholz, Florian Erhard, Laura V Glaser, Lars Dölken, Ralf Zimmer, Bettina Kempkes
Publikováno v:
PLoS Pathogens, Vol 9, Iss 9, p e1003638 (2013)
Epstein-Barr virus (EBV) causes a persistent infection in human B cells by establishing specific transcription programs to control B cell activation and differentiation. Transcriptional reprogramming of EBV infected B cells is predominantly driven by
Externí odkaz:
https://doaj.org/article/a46e1d947c0c4503a73861081fc37713
Autor:
Barbara A Scholz, Marie L Harth-Hertle, Georg Malterer, Juergen Haas, Joachim Ellwart, Thomas F Schulz, Bettina Kempkes
Publikováno v:
PLoS Pathogens, Vol 9, Iss 5, p e1003336 (2013)
Since Kaposi's sarcoma associated herpesvirus (KSHV) establishes a persistent infection in human B cells, B cells are a critical compartment for viral pathogenesis. RTA, the replication and transcription activator of KSHV, can either directly bind to
Externí odkaz:
https://doaj.org/article/388f48ce89134888acccc5889dafdc51
Autor:
Dietmar E. Martin, Sybille Thumann, Cornelia Kuklik-Roos, Laura V. Glaser, Sophie Beer, Ralf Zimmer, Bettina Kempkes, Stefan Krebs, Helmut Blum, Kerstin C. Maier, Marie L. Harth-Hertle, Florian Erhard, Björn Grüning, Simone Rieger, Rolf Backofen
Publikováno v:
PLoS Pathogens
PLoS Pathogens, Vol 13, Iss 10, p e1006664 (2017)
PLoS Pathog. 13, e1006664:e1006664 (2017)
PLoS Pathogens, Vol 13, Iss 10, p e1006664 (2017)
PLoS Pathog. 13, e1006664:e1006664 (2017)
Epstein-Barr virus (EBV) infection converts resting human B cells into permanently proliferating lymphoblastoid cell lines (LCLs). The Epstein-Barr virus nuclear antigen 2 (EBNA2) plays a key role in this process. It preferentially binds to B cell en
Autor:
Vincent Lotteau, Bettina Kempkes, Henri Gruffat, Pascal Leblanc, Maha Siouda, Quentin Bazot, Lionel Tafforeau, Chantal Rabourdin-Combe, Evelyne Manet, Massimo Tommasino, Marie L. Harth-Hertle, Thibaut Deschamps
Publikováno v:
Nucleic acids research
Nucleic Acids Research
Nucleic Acids Research, 2014, 42 (15), pp.9700-9716. ⟨10.1093/nar/gku697⟩
Nucleic Acids Research, Oxford University Press, 2014, 42 (15), pp.9700-9716. ⟨10.1093/nar/gku697⟩
Nucleic Acids Res. 42, 9700-9716 (2014)
Nucleic Acids Research
Nucleic Acids Research, 2014, 42 (15), pp.9700-9716. ⟨10.1093/nar/gku697⟩
Nucleic Acids Research, Oxford University Press, 2014, 42 (15), pp.9700-9716. ⟨10.1093/nar/gku697⟩
Nucleic Acids Res. 42, 9700-9716 (2014)
The Epstein-Barr virus (EBV) nuclear antigen 3 family of protein is critical for the EBV-induced primary B-cell growth transformation process. Using a yeast two-hybrid screen we identified 22 novel cellular partners of the EBNA3s. Most importantly, a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15131f53a2c438461fe81f242eb746ac
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/174717
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/174717
Autor:
Barbara A. Scholz, Juergen Haas, Georg Malterer, Bettina Kempkes, Thomas F. Schulz, Joachim W. Ellwart, Marie L. Harth-Hertle
Publikováno v:
PLoS Pathog. 9:e1003336 (2013)
PLoS Pathogens
PLoS Pathogens, Vol 9, Iss 5, p e1003336 (2013)
Scholz, B A, Harth-Hertle, M L, Malterer, G, Haas, J, Ellwart, J, Schulz, T F & Kempkes, B 2013, ' Abortive Lytic Reactivation of KSHV in CBF1/CSL Deficient Human B Cell Lines ', PLoS Pathogens, vol. 9, no. 5, e1003336 . https://doi.org/10.1371/journal.ppat.1003336
PLoS Pathogens
PLoS Pathogens, Vol 9, Iss 5, p e1003336 (2013)
Scholz, B A, Harth-Hertle, M L, Malterer, G, Haas, J, Ellwart, J, Schulz, T F & Kempkes, B 2013, ' Abortive Lytic Reactivation of KSHV in CBF1/CSL Deficient Human B Cell Lines ', PLoS Pathogens, vol. 9, no. 5, e1003336 . https://doi.org/10.1371/journal.ppat.1003336
Since Kaposi's sarcoma associated herpesvirus (KSHV) establishes a persistent infection in human B cells, B cells are a critical compartment for viral pathogenesis. RTA, the replication and transcription activator of KSHV, can either directly bind to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::883bb27774b7d4858982d4b18809e260
https://push-zb.helmholtz-muenchen.de/frontdoor.php?source_opus=25762
https://push-zb.helmholtz-muenchen.de/frontdoor.php?source_opus=25762
Autor:
Lars Dölken, Florian Erhard, Ralf Zimmer, Bettina Kempkes, Barbara A. Scholz, Laura V. Glaser, Marie L. Harth-Hertle
Publikováno v:
PLoS Pathogens, Vol 9, Iss 9, p e1003638 (2013)
PLoS Pathogens
PLoS Pathog. 9:e1003638 (2013)
PLoS Pathogens
PLoS Pathog. 9:e1003638 (2013)
Epstein-Barr virus (EBV) causes a persistent infection in human B cells by establishing specific transcription programs to control B cell activation and differentiation. Transcriptional reprogramming of EBV infected B cells is predominantly driven by