Zobrazeno 1 - 2
of 2
pro vyhledávání: '"Marie A.F. Sears"'
Autor:
C. Ian Spencer, Shiro Baba, Kenta Nakamura, Ethan A. Hua, Marie A.F. Sears, Chi-cheng Fu, Jianhua Zhang, Sadguna Balijepalli, Kiichiro Tomoda, Yohei Hayashi, Paweena Lizarraga, Julianne Wojciak, Melvin M. Scheinman, Katriina Aalto-Setälä, Jonathan C. Makielski, Craig T. January, Kevin E. Healy, Timothy J. Kamp, Shinya Yamanaka, Bruce R. Conklin
Publikováno v:
Stem Cell Reports, Vol 3, Iss 2, Pp 269-281 (2014)
Long-QT syndrome mutations can cause syncope and sudden death by prolonging the cardiac action potential (AP). Ion channels affected by mutations are various, and the influences of cellular calcium cycling on LQTS cardiac events are unknown. To bette
Externí odkaz:
https://doaj.org/article/e99f4cbcfba74440b1ffe91f48d916d9
Autor:
Craig T. January, Kevin E. Healy, Jianhua Zhang, Ethan A. Hua, Marie A.F. Sears, Jonathan C. Makielski, Julianne Wojciak, Melvin M. Scheinman, Paweena Lizarraga, Chi-cheng Fu, Kenta Nakamura, Sadguna Y. Balijepalli, Yohei Hayashi, Shiro Baba, Shinya Yamanaka, C. Ian Spencer, Kiichiro Tomoda, Timothy J. Kamp, Bruce R. Conklin, Katriina Aalto-Setälä
Publikováno v:
Stem cell reports, vol 3, iss 2
Stem Cell Reports
Stem Cell Reports, Vol 3, Iss 2, Pp 269-281 (2014)
Spencer, CI; Baba, S; Nakamura, K; Hua, EA; Sears, MAF; Fu, CC; et al.(2014). Calcium transients closely reflect prolonged action potentials in iPSC models of inherited cardiac arrhythmia. Stem Cell Reports, 3(2), 269-281. doi: 10.1016/j.stemcr.2014.06.003. UCSF: Retrieved from: http://www.escholarship.org/uc/item/9720z7s2
Stem Cell Reports
Stem Cell Reports, Vol 3, Iss 2, Pp 269-281 (2014)
Spencer, CI; Baba, S; Nakamura, K; Hua, EA; Sears, MAF; Fu, CC; et al.(2014). Calcium transients closely reflect prolonged action potentials in iPSC models of inherited cardiac arrhythmia. Stem Cell Reports, 3(2), 269-281. doi: 10.1016/j.stemcr.2014.06.003. UCSF: Retrieved from: http://www.escholarship.org/uc/item/9720z7s2
Summary Long-QT syndrome mutations can cause syncope and sudden death by prolonging the cardiac action potential (AP). Ion channels affected by mutations are various, and the influences of cellular calcium cycling on LQTS cardiac events are unknown.