Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Mariana Paulson"'
Publikováno v:
Reproductive BioMedicine Online. 41:128-137
Research question Is endometrial expression of anti-Mullerian hormone (AMH) and its receptor II (AMH-R) altered in women with polycystic ovary syndrome (PCOS) and affected by lifestyle intervention? Design Endometrial immunostaining of AMH and AMH-R
Autor:
Marika H. Kangasniemi, Elina K. Komsi, Henna-Riikka Rossi, Annikki Liakka, Masuma Khatun, Joseph C. Chen, Mariana Paulson, Angelica L. Hirschberg, Riikka K. Arffman, Terhi T. Piltonen
Publikováno v:
FS science. 3(2)
To study whether artificial intelligence (AI) technology can be used to discern quantitative differences in endometrial immune cells between cycle phases and between samples from women with polycystic ovary syndrome (PCOS) and non-PCOS controls. Only
Publikováno v:
Gynecological Endocrinology. 36:226-232
Polycystic ovary syndrome (PCOS) is associated with increased risk of endometrial cancer. There is growing evidence that prolactin and its receptor (PRLR) are involved in the development of cancer. We assessed endometrial expression of PRLR mRNA, and
Autor:
Masuma Khatun, Riikka K Arffman, Terhi Piltonen, Leif C. Andersson, Darja Lavogina, Andres Salumets, Mariana Paulson, Johanna Laru, Siri Lehtonen, Marika Kangasniemi, Anne Ahtikoski, Angelica Lindén Hirschberg
Publikováno v:
Biology of Reproduction
Stanniocalcin-1 (STC-1) is a pro-survival factor that protects tissues against stressors, such as hypoxia and inflammation. STC-1 is co-expressed with the endometrial receptivity markers, and recently endometrial STC-1 was reported to be dysregulated
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::76c74869b366470a0393234387692bd0
http://hdl.handle.net/10138/325184
http://hdl.handle.net/10138/325184
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 102:1244-1253
Polycystic ovary syndrome (PCOS) is associated with increased risk of endometrial cancer. This is usually explained by chronic anovulation and deficient progesterone activity. However, the role of progesterone receptors (PRs) in endometrial prolifera