Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Mariana Colino-Oliveira"'
Autor:
Catarina Miranda-Lourenço, Sofia T. Duarte, Cátia Palminha, Cláudia Gaspar, Tiago M. Rodrigues, Teresa Magalhães-Cardoso, Nádia Rei, Mariana Colino-Oliveira, Rui Gomes, Sara Ferreira, Jéssica Rosa, Sara Xapelli, Judith Armstrong, Àngels García-Cazorla, Paulo Correia-de-Sá, Ana M. Sebastião, Maria José Diógenes
Publikováno v:
Neurobiology of Disease, Vol 145, Iss , Pp 105043- (2020)
Rett syndrome (RTT; OMIM#312750) is mainly caused by mutations in the X-linked MECP2 gene (methyl-CpG-binding protein 2 gene; OMIM*300005), which leads to impairments in the brain-derived neurotrophic factor (BDNF) signalling. The boost of BDNF media
Externí odkaz:
https://doaj.org/article/a7b74adeec4f45b6a70564d38ab6a0f5
Autor:
Catarina Miranda-Lourenço, Sofia Duarte, Cátia Palminha, Mariana Colino-Oliveira, Jéssica Rosa, Rui Gomes, Tiago Rodrigues, Sara Xapelli, Luísa Lopes, Ana Sebastião, Maria Diógenes
Publikováno v:
Frontiers in Cellular Neuroscience. 13
Autor:
Mariana Colino-Oliveira, Joaquim A. Ribeiro, Raquel B. Dias, Ana M. Sebastião, Diogo M. Rombo
Publikováno v:
Purinergic Signalling. 12:283-294
Brain-derived neurotrophic factor (BDNF) and adenosine are widely recognized as neuromodulators of glutamatergic transmission in the adult brain. Most BDNF actions upon excitatory plasticity phenomena are under control of adenosine A2A receptors (A2A
Autor:
Cátia Palminha, Maria José Diógenes, Ana M. Sebastião, Cláudia Gaspar, Nádia Rei, Sofia T. Duarte, Sara Ferreira, Judith Armstrong, Mariana Colino-Oliveira, Jéssica Rosa, Tiago Rodrigues, Sara Xapelli, Angels García-Cazorla, Rui Gomes, Catarina Miranda-Lourenço, Teresa Magalhães-Cardoso, Paulo Correia-de-Sá
Publikováno v:
Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
Agência para a Sociedade do Conhecimento (UMIC)-FCT-Sociedade da Informação
instacron:RCAAP
NEUROBIOLOGY OF DISEASE
r-FSJD: Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
Fundació Sant Joan de Déu
r-FSJD. Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
instname
Neurobiology of Disease
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
Neurobiology of Disease, Vol 145, Iss, Pp 105043-(2020)
Agência para a Sociedade do Conhecimento (UMIC)-FCT-Sociedade da Informação
instacron:RCAAP
NEUROBIOLOGY OF DISEASE
r-FSJD: Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
Fundació Sant Joan de Déu
r-FSJD. Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
instname
Neurobiology of Disease
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
Neurobiology of Disease, Vol 145, Iss, Pp 105043-(2020)
© 2020 The Authors. Published by Elsevier Inc. This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/BY-NC-ND/4.0/)
Rett syndrome (RTT; OMIM#312750) is mainly caused by mutations in the X-linked MECP2
Rett syndrome (RTT; OMIM#312750) is mainly caused by mutations in the X-linked MECP2
Autor:
Roberta Schmatz, Maria Rosa Chitolina Schetinger, Maribel Antonello Rubin, Andréia Machado Cardoso, Mariana Colino-Oliveira, Jessié Martins Gutierres, Jéssica Lopes Gomes, Victor Camera Pimentel, Ana M. Sebastião, Fabiano B. Carvalho, Margarete Dulce Bagatini, Maria Beatriz Moretto, Daniela Zanini, Vera Maria Morsch
Publikováno v:
Neurochemical Research
It is well known that the levels of adenosine in the brain increase dramatically during cerebral hypoxic-ischemic (HI) insults. Its levels are tightly regulated by physiological and pathophysiological changes that occur during the injury acute phase.
Autor:
Detlev Boison, Long Liu, Abbie Jones, Yuqing Huo, Shayla Q. Coffman, Bounmy Saleumvong, Catarina Miranda-Lourenço, Mariana Colino-Oliveira, Ana M. Sebastião, Maria José Diógenes, Cátia Palminha, Yiming Xu, Vânia L. Batalha, Ursula S. Sandau
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 36(48)
Adenosine kinase (ADK) deficiency in human patients (OMIM:614300) disrupts the methionine cycle and triggers hypermethioninemia, hepatic encephalopathy, cognitive impairment, and seizures. To identify whether this neurological phenotype is intrinsica
Autor:
Daniela Zanini, Maria Rosa Chitolina Schetinger, Victor Camera Pimentel, Mariana Colino Oliveira, Ana M. Sebastião, Maria Beatriz Moretto
Publikováno v:
Molecular and Cellular Biochemistry
Hypoxic-ischemic (HI) injury perinatal brain is a major contributor to morbidity and mortality to infants and children. Adenosine may play a role in the pathophysiology of HI, since it modulates the inflammatory process and the release of several neu
Differential role of the proteasome in the early and late phases of BDNF-induced facilitation of LTP
Autor:
Mariana Colino-Oliveira, Diogo M. Rombo, Maddalena Grimaldi, Blanka Kellermayer, Ana Rita Santos, Ana M. Sebastião, Diogo Comprido, Carlos B. Duarte, Miranda Mele, Sandra H. Vaz
Publikováno v:
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Journal of Neuroscience
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Journal of Neuroscience
Copyright © 2015 the authors
The neurotrophin brain-derived neurotrophic factor (BDNF) mediates activity-dependent long-term changes of synaptic strength in the CNS. The effects of BDNF are partly mediated by stimulation of local translation, w
The neurotrophin brain-derived neurotrophic factor (BDNF) mediates activity-dependent long-term changes of synaptic strength in the CNS. The effects of BDNF are partly mediated by stimulation of local translation, w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a9c9053693a3b060e45020ba78e44249
Publikováno v:
Journal of neurophysiology. 112(11)
Different forms of plasticity occur concomitantly in the nervous system. Whereas homeostatic plasticity monitors and maintains neuronal activity within a functional range, Hebbian changes such as long-term potentiation (LTP) modify the relative stren
Autor:
Joaquim A. Ribeiro, Natália Assaife-Lopes, Ana M. Sebastião, Mariana Colino-Oliveira, Raquel B. Dias
Publikováno v:
Neuropharmacology
The synapse is a crowded area. In the last years, the concept that proteins can be organized in different membrane domains according to their structure has emerged. Cholesterol-rich membrane domains, or lipid rafts, form an organized portion of the m