Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Maria van Betteraey-Nikoleit"'
Cdk2 activity is dispensable for triggering replicon initiation after transient hypoxia in T24 cells
Publikováno v:
FEBS Journal. 272:5623-5634
We examined whether the fast release of replicon initiation after sudden O2 recovery of hypoxically incubated mammalian cells depends on kinase activity of Cdk2. We used a system based on starved/refed T24 cells elaborated previously for such investi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f9c47f881e2328bdd915410c0aab84f
https://doi.org/10.1111/j.1742-4658.2005.04957.x
https://doi.org/10.1111/j.1742-4658.2005.04957.x
Publikováno v:
European Journal of Biochemistry. 270:3880-3890
It was shown previously [Riedinger, H. J., van Betteraey-Nikoleit, M & Probst, H. (2002) Eur. J. Biochem.269, 2383-2393] that initiation of in vivo SV40 DNA replication is reversibly suppressed by hypoxia in a state where viral minichromosomes exhibi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7bae57d8f446648cd25427d36981e144
https://doi.org/10.1046/j.1432-1033.2003.03769.x
https://doi.org/10.1046/j.1432-1033.2003.03769.x
Publikováno v:
European Journal of Biochemistry. 269:2383-2393
Hypoxia interrupts the initiation of simian virus 40 (SV40) replication in vivo at a stage situated before unwinding of the␣origin region. After re-oxygenation, unwinding followed by a synchronous round of viral replication takes place. To␣furthe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8bbbda07616fc7f2bf8759eec23d6a51
https://doi.org/10.1046/j.1432-1033.2002.02902.x
https://doi.org/10.1046/j.1432-1033.2002.02902.x
Autor:
Hans-Jörg Riedinger, Hans Probst, Maria van Betteraey-Nikoleit, Karl-Heinz Eisele, Uwe Hilfrich
Publikováno v:
Journal of Biological Chemistry. 276:47122-47130
Simian virus 40 (SV40)-infected CV1 cells exposed to hypoxia show an inhibition of viral replication. Reoxygenation after several hours of hypoxia results in new initiations followed by a nearly synchronous round of SV40 replication. In this communic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::916fb02415f62ed33b9ad22728ba5925
https://doi.org/10.1074/jbc.m106938200
https://doi.org/10.1074/jbc.m106938200
Publikováno v:
European journal of biochemistry. 270(19)
It was shown previously [Riedinger, H. J., van Betteraey-Nikoleit, MProbst, H. (2002) Eur. J. Biochem.269, 2383-2393] that initiation of in vivo SV40 DNA replication is reversibly suppressed by hypoxia in a state where viral minichromosomes exhibit a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::4262d4b373e7bc12d483562a216a5ab7
https://pubmed.ncbi.nlm.nih.gov/14511370
https://pubmed.ncbi.nlm.nih.gov/14511370
Publikováno v:
European journal of biochemistry. 269(9)
Hypoxia interrupts the initiation of simian virus 40 (SV40) replication in vivo at a stage situated before unwinding of the origin region. After re-oxygenation, unwinding followed by a synchronous round of viral replication takes place. To further ch
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7705be6d38be03bf9025496bf26c0901
https://pubmed.ncbi.nlm.nih.gov/11985622
https://pubmed.ncbi.nlm.nih.gov/11985622