Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Maria Tengvall-Linder"'
Autor:
Agne Liedén, Mårten C G Winge, Annika Sääf, Ingrid Kockum, Elisabeth Ekelund, Elke Rodriguez, Regina Fölster-Holst, Andre Franke, Thomas Illig, Maria Tengvall-Linder, Hansjörg Baurecht, Stephan Weidinger, Carl-Fredrik Wahlgren, Magnus Nordenskjöld, Maria Bradley
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e49694 (2012)
BackgroundAtopic dermatitis (AD) is a common chronic inflammatory skin disorder where epidermal barrier dysfunction is a major factor in the pathogenesis. The identification of AD susceptibility genes related to barrier dysfunction is therefore of im
Externí odkaz:
https://doaj.org/article/3cd28edbb3db455e91ff69e8e78d3e3c
Autor:
Annika M Sääf, Maria Tengvall-Linder, Howard Y Chang, Adam S Adler, Carl-Fredrik Wahlgren, Annika Scheynius, Magnus Nordenskjöld, Maria Bradley
Publikováno v:
PLoS ONE, Vol 3, Iss 12, p e4017 (2008)
Atopic eczema (AE) is a common chronic inflammatory skin disorder. In order to dissect the genetic background several linkage and genetic association studies have been performed. Yet very little is known about specific genes involved in this complex
Externí odkaz:
https://doaj.org/article/fb6ec7d3295a42a891fae203c51bbeef
Autor:
Enikö Sonkoly, Tianling Wei, Peter C J Janson, Annika Sääf, Lena Lundeberg, Maria Tengvall-Linder, Gunnar Norstedt, Harri Alenius, Bernhard Homey, Annika Scheynius, Mona Ståhle, Andor Pivarcsi
Publikováno v:
PLoS ONE, Vol 2, Iss 7, p e610 (2007)
MicroRNAs are a recently discovered class of posttranscriptional regulators of gene expression with critical functions in health and disease. Psoriasis is the most prevalent chronic inflammatory skin disease in adults, with a substantial negative imp
Externí odkaz:
https://doaj.org/article/8588f0c3025645b099020f60b7c61070
Autor:
Mårten C.G. Winge, Annika Sääf, Magnus Nordenskjöld, Maria Tengvall-Linder, Maria Bradley, Bernhard Homey, Andor Pivarcsi, Carl-Fredrik Wahlgren
Publikováno v:
Archives of Dermatological Research. 304:773-780
Atopic dermatitis (AD) is one of the most common chronic inflammatory skin diseases in industrialized countries. To identify candidate genes involved in the pathogenesis of AD, we previously undertook a genome-wide approach using DNA microarrays. A t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::617bc4f6179ece296c5de10a6db9e5f6
https://doi.org/10.1007/s00403-012-1242-4
https://doi.org/10.1007/s00403-012-1242-4
Autor:
Annika Scheynius, Lena Lundeberg, Markus Moll, Maria Tengvall Linder, Mikael C. I. Karlsson, Ola Winqvist, Carlotta Kuylenstierna, Catharina Johansson, Emilie Domange Jordö, Johan K. Sandberg, Sara M. Lind, Maria A Karlsson
Publikováno v:
European Journal of Immunology. 39:2293-2301
Atopic eczema (AE) is a chronic relapsing inflammatory skin disease where the commensal yeast Malassezia can act as a microbial trigger factor. Malassezia activates human DC to produce IL-18, an innate cytokine that is elevated in serum of AE patient
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::66f8ae9bffc7c6586f5387a360624b81
https://doi.org/10.1002/eji.200839195
https://doi.org/10.1002/eji.200839195
Autor:
Maria Tengvall Linder, Annika Scheynius, Niklas Ahlborg, Anna Andersson, Maria A Karlsson, Lena Lundeberg, Catharina Johansson
Publikováno v:
International Archives of Allergy and Immunology. 150:51-58
Background: Atopic eczema is a chronic inflammatory skin disease in which several subgroups of cases can be identified. Atopy patch testing (APT) reveals allergen sensitization also in atopic eczema patients devoid of detectable allergen-specific IgE
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2fe63ed722c3a83004f2c802b21be1da
https://doi.org/10.1159/000210380
https://doi.org/10.1159/000210380
Autor:
Sabine Flückiger, Catharina Johansson, Maria Tengvall Linder, Peter Schmid-Grendelmeier, Barbra Fischer Casagrande, Annika Scheynius, Reto Crameri
Publikováno v:
Journal of Investigative Dermatology. 126:2414-2421
The opportunistic yeast Malassezia sympodialis belongs to the normal cutaneous flora but can also cause IgE-mediated sensitization in patients suffering from atopic eczema (AE). We investigated 706 individuals by ImmunoCAPm70 and skin-prick tests wit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::677e309b2b1c88d7b0cfed5ef055267f
https://doi.org/10.1038/sj.jid.5700431
https://doi.org/10.1038/sj.jid.5700431
Autor:
Eva Buentke, Mauro D'Amato, Margit Schmidt, Susanne Gabrielsson, Annika Scheynius, Agne Liedén, Maria Tengvall-Linder
Publikováno v:
Acta Dermato-Venereologica. 84:339-345
It is known that 28-84% of patients with atopic dermatitis exhibit IgE and/or T-cell reactivity to the opportunistic yeast Malassezia sympodialis, which can be taken up by immature monocyte-derived dendritic cells (MDDCs), resulting in MDDC maturatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b643d77d188a8307341754f8c51d0881
https://doi.org/10.1080/00015550410026957
https://doi.org/10.1080/00015550410026957
Publikováno v:
International Archives of Allergy and Immunology. 135:93-100
Background: The opportunistic yeast Malassezia is considered to be one of the factors that can contribute to atopic eczema (AE). Elevated serum IgE levels, T-cell proliferation and positive skin prick test (SPT) and atopy patch test (APT) reactions t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f0263dace3c4db262081735d84a96dde
https://doi.org/10.1159/000080651
https://doi.org/10.1159/000080651
Publikováno v:
International Archives of Allergy and Immunology. 127:161-169
Atopic dermatitis is a chronic multifactorial inflammatory skin disease, which has had a marked increase in prevalence during the last decades. Recently, a new nomenclature was recommended where the term ‘atopic eczema/dermatitis syndrome’ (AEDS)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2196dcc2c9221a4fb5647ed726bd3ef7
https://doi.org/10.1159/000053860
https://doi.org/10.1159/000053860