Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Maria Rife"'
Publikováno v:
The Journal of Immunology. 206:20.33-20.33
House dust mites (HDM) are a predominant causative agent of airway hypersensitivity and asthma. The HDM group 1 allergens include Der p 1, a cysteine protease that contributes to sensitization and symptom exacerbation. Respiratory syncytial virus (RS
Autor:
Ben A. Oostra, Laura L. Kirkpatrick, Marianne Hoogeveen-Westerveld, Lies-Anne A. Severijnen, Ruben H. Willemsen, T. Zu, David L. Nelson, Ingeborg M. Nieuwenhuizen, Edwin Mientjes, Maria Rife
Publikováno v:
Neurobiology of Disease, Vol 21, Iss 3, Pp 549-555 (2006)
Neurobiology of Disease, 21(3), 549-555. Academic Press
Neurobiology of Disease, 21(3), 549-555. Academic Press
The FMR1 gene, mutated in Fragile X syndrome patients, has been modeled in mice with a neomycin cassette inserted in exon 5 of the mouse Fmr1 gene creating an Fmr1 knockout (Fmr1 KO) allele. This results in animals lacking Fmr1 protein (Fmrp) express
Autor:
Sandra van 't Padje, Ben A. Oostra, Mariëtte Schrier, Gert van Cappellen, Maria Rife, Lies-Anne Severijnen, Surya A. Reis, Rob Willemsen
Publikováno v:
Experimental Neurology, 189, 343-353. Academic Press
Lack of fragile X mental retardation protein (FMRP) causes the fragile X syndrome, a common form of inherited mental retardation. The syndrome usually results from the expansion of a CGG repeat in the FMR1 gene with consequent transcriptional silenci
Autor:
Rob Willemsen, Josien Levenga, Ronald A.M. Buijsen, Maria Rife, Ben A. Oostra, David L. Nelson, Femke M.S. de Vrij, Hervé Moine
Publikováno v:
Neurobiology of Disease
Neurobiology of Disease, Elsevier, 2009, 35 (2), pp.241-50. ⟨10.1016/j.nbd.2009.05.004⟩
Neurobiology of Disease, 35(2), 241-250. Academic Press
Neurobiology of Disease, Vol 35, Iss 2, Pp 241-250 (2009)
Neurobiology of Disease, 2009, 35 (2), pp.241-50. ⟨10.1016/j.nbd.2009.05.004⟩
Neurobiology of Disease, Elsevier, 2009, 35 (2), pp.241-50. ⟨10.1016/j.nbd.2009.05.004⟩
Neurobiology of Disease, 35(2), 241-250. Academic Press
Neurobiology of Disease, Vol 35, Iss 2, Pp 241-250 (2009)
Neurobiology of Disease, 2009, 35 (2), pp.241-50. ⟨10.1016/j.nbd.2009.05.004⟩
Fragile X syndrome is caused by lack of the protein FMRP. FMRP mediates mRNA binding, dendritic mRNA transport and translational control at spines. We examined the role of functional domains of FMRP in neuronal RNA-granule formation and dendritic tra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::649c484f53cde5b703cb3668263a57eb
https://www.hal.inserm.fr/inserm-00420367
https://www.hal.inserm.fr/inserm-00420367
Publikováno v:
Blood. 108:1922-1922
Immortal cell growth is considered the hallmark of tumor cells. In contrast, normal cells have a limited proliferative capacity of 40–60 cell divisions, also known as the Hayflick limit. The limited proliferative capacity of normal cells relates to
Autor:
Josien Levenga, Ronald A.M. Buijsen, Maria Rifé, Hervé Moine, David L. Nelson, Ben A. Oostra, Rob Willemsen, Femke M.S. de Vrij
Publikováno v:
Neurobiology of Disease, Vol 35, Iss 2, Pp 241-250 (2009)
Fragile X syndrome is caused by lack of the protein FMRP. FMRP mediates mRNA binding, dendritic mRNA transport and translational control at spines. We examined the role of functional domains of FMRP in neuronal RNA-granule formation and dendritic tra
Externí odkaz:
https://doaj.org/article/d58d87658b9d4e1fb6e0706e5be2a970