Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Maria Nowacka-Zawisza"'
Autor:
Maria Nowacka-Zawisza, Ewelina Wiśnik, Andrzej Wasilewski, Milena Skowrońska, Ewa Forma, Magdalena Bryś, Waldemar Różański, Wanda M. Krajewska
Publikováno v:
Analytical Cellular Pathology, Vol 2015 (2015)
Genetic polymorphisms in DNA repair genes may induce individual variations in DNA repair capacity, which may in turn contribute to the risk of cancer developing. Homologous recombination repair (HRR) plays a critical role in maintaining chromosomal i
Externí odkaz:
https://doaj.org/article/6a607255e66d4fa0b916ae7124d1b1ef
Autor:
Agata Raszkiewicz, Waldemar Różański, Maria Nowacka-Zawisza, Ewa Forma, Wanda M. Krajewska, Magdalena Bryś, Tomasz Kwasiborski
Publikováno v:
Journal of Oncology, Vol 2019 (2019)
Journal of Oncology
Journal of Oncology
Genetic polymorphisms in DNA repair genes may affect DNA repair efficiency and may contribute to the risk of developing cancer. The aim of our study was to investigate single nucleotide polymorphisms (SNPs) in RAD51 (rs2619679, rs2928140, and rs50307
Autor:
Piotr K. Zakrzewski, Tomasz Rechberger, Wanda M. Krajewska, Krzysztof Gałczyński, Andrzej Semczuk, Maria Nowacka-Zawisza
Publikováno v:
Oncology Reports. 35:932-938
Downregulation of betaglycan (β-glycan) [transforming growth factor β receptor type III (TGFβR3)], which belongs to co-receptors of the TGFβ pathway, occurs in a broad spectrum of primary human malignancies. However, in the case of endometrial ca
Autor:
Wioletta Pietruszewska, Katarzyna Starska, Iwona Lewy-Trenda, Magdalena Bryś, Piotr Ciesielski, Michał Skóra, Ewa Forma, Maria Nowacka-Zawisza
Publikováno v:
Tumor Biology. 37:4541-4557
Topoisomerase IIβ binding protein 1 (TopBP1), a multiple-BRCT-domain, protein plays crucial roles in chromosome replication, DNA damage repair, apoptosis, and cell cycle checkpoint signalling. The aim of this study was to identify five SNPs at loci
Autor:
Ewelina Wiśnik, Maria Nowacka-Zawisza
Publikováno v:
Oncology reports. 38(5)
Prostate cancer is the second most commonly diagnosed cancer in men in Poland after lung cancer and the third leading cause of cancer-related mortality after lung and colon cancer. The etiology of most cases of prostate cancer are not fully known, an
Autor:
Ewa Forma, Wanda M. Krajewska, Hanna Romanowicz-Makowska, Maria Nowacka-Zawisza, Anna Urbańska, Magdalena Bryś
Publikováno v:
Open Life Sciences, Vol 9, Iss 9, Pp 833-840 (2014)
Background Genetic variants in hormone receptor genes may be crucial predisposing factors for breast cancer, and microsatellites in the androgen receptor gene (AR) have been suggested to play a role. The aim of the study was to determine the associat
Publikováno v:
Postępy Higieny i Medycyny Doświadczalnej. 67:1090-1097
The term triple-negative breast cancer (TNBC) defines breast tumors that do not express estrogen receptors, progesterone receptor or epidermal growth factor receptor HER2 on immunohistochemical analysis. TNBC accounts for 12-17% of all types of breas
Autor:
Andrzej Kulig, Hanna Romanowicz-Makowska, Magdalena Brys, Wanda M. Krajewska, Maria Nowacka-Zawisza
Publikováno v:
Pathology International. 58:275-281
Recent studies suggest that genetic polymorphisms of the DNA repair genes have been implicated in breast cancer risk. BRCA1 and BRCA2, two breast cancer susceptibility genes, are essential to maintain chromosomal integrity. This is mediated via regul
Autor:
Hanna Romanowicz-Makowska, Maria Nowacka-Zawisza, Magdalena Bryś, Wanda M. Krajewska, Andrzej Kulig
Publikováno v:
Cancer Detection and Prevention. 32:144-148
Background : Loss of heterozygosity (LOH) in the 15q14-21 and 13q12-13 regions can contribute to the inactivation of RAD51 and BRCA2 genes implicated in the pathogenesis of breast cancer. We investigated allelic losses in microsatellites in the RAD51
Autor:
Wanda M. Krajewska, Ewa Forma, Waldemar Różański, Magdalena Bryś, Maciej Walczak, Maria Nowacka-Zawisza
Publikováno v:
Medical Oncology (Northwood, London, England)
Although prostate cancer is one of the most common cancers in men, the genetic defects underlying its pathogenesis remain poorly understood. DNA damage repair mechanisms have been implicated in human cancer. Accumulating evidence indicates that the f