Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Maria J. Carnicer"'
Autor:
Josep F. Nomdedeu, E Rubiol, Josep Ubeda, Olga Lopez, Camino Estivill, Maria J. Carnicer, L. Muñoz, Jordi Sierra, M. Bellido, R. Bordes
Publikováno v:
European Journal of Haematology. 66:100-106
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::62e63128730c9e548738a9d36a356447
https://doi.org/10.1111/j.1600-0609.2001.00313.x
https://doi.org/10.1111/j.1600-0609.2001.00313.x
Autor:
Vanesa Orantes, Adriana Lasa, Elena Serrano, Anna Aventin, Maria J. Carnicer, Salut Brunet, Jorge Sierra, Josep F. Nomdedeu, Jorge Pena
Publikováno v:
Leukemia Research. 32:944-953
Acute myeloid leukemia (AML) is a heterogeneous group of disorders characterized by an abnormal proliferation of the myeloid precursors and a maturation block. The most common chromosomal lesions in AML are the t(8;21) and inv(16). To better understa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f2dbb37b0149bcd23786b204b8e255f4
https://doi.org/10.1016/j.leukres.2007.11.038
https://doi.org/10.1016/j.leukres.2007.11.038
Autor:
Salut Brunet, Elena Serrano, Adriana Lasa, Isabel Badell, Anna Aventin, Maite Carricondo, Josep F. Nomdedeu, Jorge Sierra, Maria J. Carnicer
Publikováno v:
Annals of Hematology. 87:205-211
CEACAM family members are a set of widely expressed proteins involved in several biological functions, including cell adhesion, migration, signal transduction, and the regulation of gene expression. Abnormal overexpression and downregulation of some
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a948fb7a7965541c540719199e5d956
https://doi.org/10.1007/s00277-007-0388-1
https://doi.org/10.1007/s00277-007-0388-1
Autor:
Granada Perea, Anna Aventin, Salut Brunet, Adriana Lasa, Maria J. Carnicer, Jorge Sierra, Josep F. Nomdedeu, Camino Estivill
Publikováno v:
Annals of Hematology. 84:368-375
To investigate the biologic relevance of microsatellite instability (MSI) in de novo acute myeloid leukemia (AML), 102 consecutive adult patients were analyzed by using a panel of seven microsatellites (BAT25, BAT26, D13S1267, D13S174, D2S123, D5S346
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::475f6c47663b09db0043f2328ab6ab0f
https://doi.org/10.1007/s00277-005-1035-3
https://doi.org/10.1007/s00277-005-1035-3
Autor:
Josep F. Nomdedeu, Anna Aventin, Camino Estivill, Maria J. Carnicer, Jorge Sierra, Adriana Lasa, Salut Brunet
Publikováno v:
Leukemia Research. 28:19-23
The basic molecular defects underlying acute myeloid leukemias (AML) seem to be caused by inactivating mutations in transcription factors which control normal myeloid differentiation (Class II mutations) and genetic lesions in tyrosine kinases result
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e3281dad467bf91b597dc92fc7003580
https://doi.org/10.1016/s0145-2126(03)00125-5
https://doi.org/10.1016/s0145-2126(03)00125-5
Autor:
A. Aventin, Maria J. Carnicer, Adriana Lasa, Mar Bellido, Jordi Sierra, Josep F. Nomdedeu, Montserrat Baiget
Publikováno v:
Leukemia. 16:2329-2332
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0aae3c65d377e13761746a297d6e4ff5
https://doi.org/10.1038/sj.leu.2402656
https://doi.org/10.1038/sj.leu.2402656
Autor:
Roland, Jäger, Gabriele, Migliorini, Marc, Henrion, Radhika, Kandaswamy, Helen E, Speedy, Andreas, Heindl, Nicola, Whiffin, Maria J, Carnicer, Laura, Broome, Nicola, Dryden, Takashi, Nagano, Stefan, Schoenfelder, Martin, Enge, Yinyin, Yuan, Jussi, Taipale, Peter, Fraser, Olivia, Fletcher, Richard S, Houlston
Publikováno v:
Nature Communications
Multiple regulatory elements distant from their targets on the linear genome can influence the expression of a single gene through chromatin looping. Chromosome conformation capture implemented in Hi-C allows for genome-wide agnostic characterization
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::4e5259b595ae8f426a353185cb75ab7b
https://pubmed.ncbi.nlm.nih.gov/25695508
https://pubmed.ncbi.nlm.nih.gov/25695508
Autor:
Jorge Sierra, Elena Serrano, Adriana Lasa, Salut Brunet, Vanesa Orantes, Maria J. Carnicer, Camino Estivill, Josep F. Nomdedeu, Anna Aventin
Publikováno v:
Leukemialymphoma. 49(6)
The discovery of underlying genetic lesions helps to better understand the mechanisms of leukemogenesis and identify prognostic subgroups. Recent insights have allowed normal karyotype acute myeloid leukemia (AML) to be split into many molecular enti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c02f4673ef04f1ab3b90eeb80acb3d3
https://pubmed.ncbi.nlm.nih.gov/18452069
https://pubmed.ncbi.nlm.nih.gov/18452069
Autor:
Adriana, Lasa, Maria T, Carricondo, Maria J, Carnicer, Granada, Perea, Anna, Aventín, Josep F, Nomdedeu
Publikováno v:
Haematologica. 91(9)
One of the most common genetic events in acute myeloid leukemia (AML) is the t(8;21) (q22;q22) translocation, which contributes to leukemic transformation. However, different lines of evidence suggest that the AML1-ETO rearrangement is not sufficient
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e01242a01dcf3130f94606653eb7eb94
https://pubmed.ncbi.nlm.nih.gov/16956837
https://pubmed.ncbi.nlm.nih.gov/16956837
Autor:
Maria J. Carnicer, Jorge Sierra, Granada Perea, Adriana Lasa, Salut Brunet, Josep F. Nomdedeu, Elena Serrano, Anna Aventin
Publikováno v:
Acta haematologica. 116(2)
Acute myeloid leukemia (AML) is a heterogeneous group of disorders characterized by abnormal proliferation of myeloid precursors and a maturation block. Underlying genetic lesions determine an altered expression program (transcriptosome) that can be
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eca0e729ef6d17a66c490413aa39b724
https://pubmed.ncbi.nlm.nih.gov/16914901
https://pubmed.ncbi.nlm.nih.gov/16914901