Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Maria J Gravato-Nobre"'
Autor:
Delia O’Rourke, Maria J Gravato-Nobre, Dave Stroud, Emily Pritchett, Emily Barker, Rebecca L Price, Sarah A Robinson, Simon Spiro, Patricia Kuwabara, Jonathan Hodgkin
Publikováno v:
G3: Genes, Genomes, Genetics, Vol 13, Iss 5 (2023)
AbstractNumerous mutants of the nematode Caenorhabditis elegansMicrobacterium nematophilumYersinia pseudotuberculosissrfbussrf-2bus-5bus-22srf-2bus-5bus-6ptr-15/bus-13dhs-5/bus-21bus-24srf-5bus-10bus-28
Externí odkaz:
https://doaj.org/article/2fd1f36a41874f759e55412173788423
Publikováno v:
Biogerontology
An increasing amount of data implicate immunity-mostly innate immunity-in the ageing process; both during healthy ageing as well as in neurodegenerative diseases. Despite the aetiology however, the underlying mechanisms are poorly understood. Here we
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36c66373be8ab33e2d0403742bf4be45
https://ora.ox.ac.uk/objects/uuid:96bc57ee-6457-42c3-b4cd-ea7a236d3617
https://ora.ox.ac.uk/objects/uuid:96bc57ee-6457-42c3-b4cd-ea7a236d3617
Autor:
Juan Wang, Maria J. Gravato-Nobre, Cory Patrick, Jonathan Hodgkin, Alina Rashid, Dianaliz Santiago-Martinez, Coleen T. Murphy, April E. Williams, Rachel Kaletsky, Maureen M. Barr, David H. Hall, Jessica N. Landis, Malan Silva, Leonard A. Haas, Rebecca J. Androwski
Publikováno v:
Current Biology. 25:3232-3238
Cilia and extracellular vesicles (EVs) are signaling organelles [1]. Cilia act as cellular sensory antennae, with defects resulting in human ciliopathies. Cilia both release and bind to EVs [1]. EVs are sub-micron-sized particles released by cells an
Publikováno v:
Current Biology
Scopus-Elsevier
Scopus-Elsevier
Summary The nematode Caenorhabditis elegans has been much studied as a host for microbial infection. Some pathogens can infect its intestine [1, 2], while others attack via its external surface [1, 3–6]. Cultures of Caenorhabditis isolated from nat
C. elegans is developing in importance as a model for innate immunity. Several signaling pathways are known to be required for immune responses to a diverse range of pathogens, including the insulin signaling, p38 MAP kinase and transforming growth f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d62a0e3c765de298332e8cfbf763e703
https://doi.org/10.1002/dvdy.22232
https://doi.org/10.1002/dvdy.22232
Autor:
George Mosialos, Petros Ligoxygakis, Iro Chaitoglou, Eudoxia Hatzivassiliou, Paul Hadweh, Maria J. Gravato-Nobre
Publikováno v:
PLoS ONE, Vol 13, Iss 2, p e0191864 (2018)
PLoS ONE
PLoS ONE
The human cylindromatosis tumor suppressor (HsCyld) has attracted extensive attention due to its association with the development of multiple types of cancer. HsCyld encodes a deubiquitinating enzyme (HsCYLD) with a broad range of functions that incl
Publikováno v:
Developmental Biology. 317(2):695-704
We have identified Conserved Non-coding Elements (CNEs) in the regulatory region of Caenorhabditis elegans and Caenorhabditis briggsae mab-9, a T-box gene known to be important for cell fate specification in the developing C. elegans hindgut. Two adj
Autor:
Patricia M. Berninsone, Brian J. Libby, Maria J. Gravato-Nobre, Carlos B. Hirschberg, Christine Göbel, Jörg Höflich, Jonathan Hodgkin, Ralf Baumeister, Samuel M. Politz, Creg Darby
Publikováno v:
Journal of Biological Chemistry. 279:30440-30448
During the establishment of a bacterial infection, the surface molecules of the host organism are of particular importance, since they mediate the first contact with the pathogen. In Caenorhabditis elegans, mutations in the srf-3 locus confer resista
Publikováno v:
Worm
In a recent paper, we reported the isolation and surprising effects of two new bacterial pathogens for Caenorhabditis and related nematodes. These two pathogens belong to the genus Leucobacter and were discovered co-infecting a wild isolate of Caenor
The surface of the nematode Caenorhabditis elegans is poorly understood but critical for its interactions with the environment and with pathogens. We show here that six genes (bus-2, bus-4, and bus-12, together with the previously cloned srf-3, bus-8
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::248514495e8d863d36551d1b77c3c706
https://europepmc.org/articles/PMC3018313/
https://europepmc.org/articles/PMC3018313/