Zobrazeno 1 - 10
of 47
pro vyhledávání: '"Maria J Forteza"'
Autor:
Roland Baumgartner, Felipe B. Casagrande, Randi B. Mikkelsen, Martin Berg, Konstantinos A. Polyzos, Maria J. Forteza, Aastha Arora, Thue W. Schwartz, Siv A. Hjorth, Daniel F. J. Ketelhuth
Publikováno v:
Metabolites, Vol 11, Iss 7, p 411 (2021)
G-protein-coupled receptor-35 (GPR35) has been identified as a receptor for the tryptophan metabolite kynurenic acid (KynA) and suggested to modulate macrophage polarization in metabolic tissues. Whether GPR35 can influence vascular inflammation and
Externí odkaz:
https://doaj.org/article/1c8d0f2ac729443ca49cd942c48ac63b
Autor:
Maria J Forteza, Martin Berg, Andreas Edsfeldt, Jangming Sun, Roland Baumgartner, Ilona Kareinen, Felipe Beccaria Casagrande, Ulf Hedin, Song Zhang, Ivan Vuckovic, Petras P Dzeja, Konstantinos A Polyzos, Anton Gisterå, Mette Trauelsen, Thue W Schwartz, Lea Dib, Joerg Herrmann, Claudia Monaco, Ljubica Matic, Isabel Gonçalves, Daniel F J Ketelhuth
Publikováno v:
Cardiovascular Research.
AimsRecent studies have revealed a close connection between cellular metabolism and the chronic inflammatory process of atherosclerosis. While the link between systemic metabolism and atherosclerosis is well established, the implications of altered m
Autor:
Maria J. Forteza, Konstantinos A. Polyzos, Roland Baumgartner, Bianca E. Suur, Marion Mussbacher, Daniel K. Johansson, Andreas Hermansson, Göran K. Hansson, Daniel F. J. Ketelhuth
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
T-cell activation is characteristic during the development of atherosclerosis. While overall T-cell responses have been implicated in disease acceleration, regulatory T cells (Tregs) exhibit atheroprotective effects. The expression of the enzyme indo
Externí odkaz:
https://doaj.org/article/8ea87542973648e4934b564803e98872
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology
The outbreak of severe acute respiratory syndrome coronavirus 2 that first emerged in Wuhan in December 2019 has resulted in the devastating pandemic of coronavirus disease 2019, creating an emerging need for knowledge sharing. Meanwhile, myocardial
Autor:
Thue W. Schwartz, Ulf Hedin, Roland Baumgartner, Daniel F. J. Ketelhuth, Siv A. Hjorth, Göran K. Hansson, Ljubica Perisic Matic, Gabrielle Paulsson-Berne, K P Polyzos, Martin Berg, Maria J. Forteza
Publikováno v:
Baumgartner, R, Berg, M, Matic, L, Polyzos, K P, Forteza, M J, Hjorth, S A, Schwartz, T W, Paulson-Berne, G, Hansson, G K, Hedin, U & Ketelhuth, D F J 2021, ' Evidence that a deviation in the kynurenine pathway aggravates atherosclerotic disease in humans ', Journal of Internal Medicine, vol. 289, no. 1, pp. 53-68 . https://doi.org/10.1111/joim.13142
BACKGROUND: The metabolism of tryptophan (Trp) along the kynurenine pathway has been shown to carry strong immunoregulatory properties. Several experimental studies indicate that this pathway is a major regulator of vascular inflammation and influenc
Autor:
Christoph J. Binder, José Basílio, Bianca E. Suur, Florian Puhm, Maria J. Forteza, Viola Gleitsmann, Manuel Salzmann, Barbara Haigl, Daniel F. J. Ketelhuth, Mario Kuttke, Bernhard Hochreiter, Bastian Hoesel, Alice Assinger, Bernhard Moser, Caroline Ungerböck, Marion Mussbacher, Johannes A. Schmid
Publikováno v:
Atherosclerosis. 307:21-31
Background and aims Inflammatory activation of endothelial cells is considered to be the first step in the development of atherosclerosis. Here, we determined the consequences of chronic endothelial activation via the NF-κB activator Ikk2 (Inhibitor
Autor:
Maria J. Forteza, Martin Berg, Thue W. Schwartz, Siv A. Hjorth, Daniel F. J. Ketelhuth, Randi B. Mikkelsen, Konstantinos A. Polyzos, Felipe B. Casagrande, Roland Baumgartner, Aastha Arora
Publikováno v:
Metabolites
Volume 11
Issue 7
Baumgartner, R, Casagrande, F B, Mikkelsen, R B, Berg, M, Polyzos, K A, Forteza, M J, Arora, A, Schwartz, T W, Hjorth, S A & Ketelhuth, D F J 2021, ' Disruption of GPR35 Signaling in Bone Marrow-Derived Cells Does Not Influence Vascular Inflammation and Atherosclerosis in Hyperlipidemic Mice ', Metabolites, vol. 11, no. 7, 411 . https://doi.org/10.3390/metabo11070411
Metabolites, Vol 11, Iss 411, p 411 (2021)
Volume 11
Issue 7
Baumgartner, R, Casagrande, F B, Mikkelsen, R B, Berg, M, Polyzos, K A, Forteza, M J, Arora, A, Schwartz, T W, Hjorth, S A & Ketelhuth, D F J 2021, ' Disruption of GPR35 Signaling in Bone Marrow-Derived Cells Does Not Influence Vascular Inflammation and Atherosclerosis in Hyperlipidemic Mice ', Metabolites, vol. 11, no. 7, 411 . https://doi.org/10.3390/metabo11070411
Metabolites, Vol 11, Iss 411, p 411 (2021)
G-protein-coupled receptor-35 (GPR35) has been identified as a receptor for the tryptophan metabolite kynurenic acid (KynA) and suggested to modulate macrophage polarization in metabolic tissues. Whether GPR35 can influence vascular inflammation and
Autor:
Daniel F. J. Ketelhuth, Hutterer E, Otto Bergman, Andonovic Z, Joy Roy, Yumeng Zhang, Maria J. Forteza, Lars Holmgren, Per Eriksson, Hultin S
Arterial endothelial cells (ECs) have the ability to respond to mechanical forces exerted by fluid shear stress. This response is of importance, as it is protective against vascular diseases such as atherosclerosis and aortic aneurysms. Mechanical fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::335b105825e5129cc5f3045cc395c70a
https://doi.org/10.21203/rs.3.rs-600069/v1
https://doi.org/10.21203/rs.3.rs-600069/v1
Autor:
Yuanyuan Zhang, Evelyn Hutterer, Sara Hultin, Otto Bergman, Maria J. Forteza, Zorana Andonovic, Daniel F.J. Ketelhuth, Joy Roy, Per Eriksson, Lars Holmgren
Arterial endothelial cells (ECs) have the ability to respond to mechanical forces exerted by fluid shear stress. This response is of importance, as it is protective against vascular diseases such as atherosclerosis and aortic aneurysms. Mechanical fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::925d7db39dc9349e7353f68a9d6d055e
https://doi.org/10.1101/2021.05.07.443138
https://doi.org/10.1101/2021.05.07.443138
Autor:
Marcelo H. Petri, Craig E. Wheelock, Xintong Jiang, Andres Laguna-Fernandez, Miguel Carracedo, Hildur Arnardottir, Silke Thul, Magnus Bäck, Gabrielle Paulsson-Berne, Jesmond Dalli, Göran K. Hansson, Antonio Checa, Teodora Andonova, Gonzalo Artiach, Daniel F. J. Ketelhuth, Roland Baumgartner, Anton Gisterå, Maria J. Forteza, Mary Walker
Publikováno v:
Circulation
Supplemental Digital Content is available in the text.
Background: In addition to enhanced proinflammatory signaling, impaired resolution of vascular inflammation plays a key role in atherosclerosis. Proresolving lipid mediators formed through t
Background: In addition to enhanced proinflammatory signaling, impaired resolution of vascular inflammation plays a key role in atherosclerosis. Proresolving lipid mediators formed through t