Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Maria H.H. Balch"'
Autor:
Kelly A. Rich, Megan G. Pino, Mehmet E. Yalvac, Ashley Fox, Hallie Harris, Maria H.H. Balch, W. David Arnold, Stephen J. Kolb
Publikováno v:
Neurobiology of Disease, Vol 182, Iss , Pp 106148- (2023)
Kinesin family member 5A (KIF5A) is an essential, neuron-specific microtubule-associated motor protein responsible for the anterograde axonal transport of various cellular cargos. Loss of function variants in the N-terminal, microtubule-binding domai
Externí odkaz:
https://doaj.org/article/cda27b4962784d7c8318bfefea38a90e
Publikováno v:
Journal of Stroke, Vol 22, Iss 2, Pp 159-172 (2020)
Stroke research has traditionally focused on the cerebral processes following ischemic brain injury, where oxygen and glucose deprivation incite prolonged activation of excitatory neurotransmitter receptors, intracellular calcium accumulation, inflam
Externí odkaz:
https://doaj.org/article/6422035087884445a78cba3060a61771
Autor:
Dana M. McTigue, Chandan K. Sen, Aidan J. Maxwell, Daniel Gallego-Perez, Jordan T. Moore, Luke R. Lemmerman, Lilibeth Ortega-Pineda, Gina P. Guio-Vega, Hallie Harris, Lauren Wilch, Diego Alzate-Correa, Savita Khanna, Maria Angelica Rincon-Benavides, Ian B. Risser, Daniel J. Dodd, Maria H.H. Balch, W. David Arnold, Shahid M Nimjee, Ana Salazar-Puerta, Silvia Duarte-Sanmiguel, Surya Gnyawali, Cameron Rink, William Lawrence, Natalia Higuita-Castro
Publikováno v:
Science Advances
Fibroblasts nonvirally programmed to convert into induced endothelial cells show therapeutic potential for ischemic stroke.
Ischemic stroke causes vascular and neuronal tissue deficiencies that could lead to substantial functional impairment and
Ischemic stroke causes vascular and neuronal tissue deficiencies that could lead to substantial functional impairment and
Autor:
Hallie Harris, Surya Gnyawali, Maria H.H. Balch, Mohamed G Ewees, Yousef Hannawi, Cameron Rink, Jay L. Zweier, Shahid M Nimjee
Publikováno v:
Stroke. 51
Introduction: Endothelial dysfunction is an important mediator of post ischemic injury of the heart and brain following ischemia/reperfusion (I/R). We have reported that CD38 activation in heart I/R models leads to NADPH depletion with endothelial ni
Autor:
W. David Arnold, Maria H.H. Balch, Deepti Chugh, Shahid M Nimjee, Cameron Rink, Surya Gnyawali, Hallie Harris
Publikováno v:
Exp Neurol
Ischemic stroke is a leading cause of disability world-wide. Mounting evidence supports neuromuscular pathology following stroke, yet mechanisms of dysfunction and therapeutic action remain undefined. The objectives of our study were to investigate n
Autor:
Surya Gnyawali, Savita Khanna, Seth Teplitsky, Mallory Heigel, Cameron Rink, Richard Stewart, Maria H.H. Balch, Chandan K. Sen, Hallie Harris
Publikováno v:
The FASEB Journal. 31:927-936
The efficacy and optimization of poststroke physical therapy paradigms is challenged in part by a lack of objective tools available to researchers for systematic preclinical testing. This work represents a maiden effort to develop a robot-assisted me
Publikováno v:
Journal of Stroke, Vol 22, Iss 3, Pp 424-424 (2020)
Journal of Stroke
Journal of Stroke, Vol 22, Iss 2, Pp 159-172 (2020)
Journal of Stroke
Journal of Stroke, Vol 22, Iss 2, Pp 159-172 (2020)
Stroke research has traditionally focused on the cerebral processes following ischemic brain injury, where oxygen and glucose deprivation incite prolonged activation of excitatory neurotransmitter receptors, intracellular calcium accumulation, inflam
Autor:
Maria H.H. Balch, Allyson Huttinger, Spencer Talentino, Cole Anderson, Jenna Wilson, Cameron Rink, Debra G Wheeler, Shahid M Nimjee
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 38
Objective: To compare the functional effects of von Willebrand Factor (VWF) inhibition by RNA aptamer (T79) vs rTPA utilizing in vivo arterial thrombosis in mice. Approach and Results: We previously demonstrated that inhibition of VWF by a targeted R
Autor:
Richard Stewart, Chandan K. Sen, Savita Khanna, Hallie Harris, Maria H.H. Balch, Cameron Rink, Surya Gnyawali, James Spieldenner
In the pathophysiologic setting of cerebral ischemia, excitotoxic levels of glutamate contribute to neuronal cell death. Our previous work demonstrated the ability of glutamate oxaloacetate transaminase (GOT) to metabolize neurotoxic glutamate in the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::525c8839c523d738a21855b7dbf8ddfb
https://europepmc.org/articles/PMC5602902/
https://europepmc.org/articles/PMC5602902/
Publikováno v:
Stroke. 48
Introduction: A growing body of literature supports acute changes to skeletal muscle physiology in response to stroke-induced central nervous system injury. While stroke survivors depend on rehabilitation to facilitate functional recovery, study of p