Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Maria H Hansen"'
Autor:
Steffen Emmert, Holger A Haenssle, John R Zibert, Margarete Schön, Andreas Hald, Maria H Hansen, Thomas Litman, Michael P Schön
Publikováno v:
PLoS ONE, Vol 11, Iss 9, p e0160096 (2016)
UNLABELLED:The rapid and strong clinical efficacy of the first-in-class, ingenol mebutate, against actinic keratosis (AK) has resulted in its recent approval. We conducted the first comprehensive analysis of the cellular and molecular mode of action
Externí odkaz:
https://doaj.org/article/3a6fda4a8fdf405f9670bfd774d4c666
Autor:
Ida K. Lund, Shanya Supramaniyam, Boye L. Jensen, Camilla Enggaard, Maria H. Hansen, Henrik J. Andersen, Mette Staehr, Per Svenningsen, Ulla G. Friis, Pernille B. Lærkegaard Hansen, Kristian B Buhl
Publikováno v:
Andersen, H, Hansen, M H, Buhl, K B, Staehr, M, Friis, U G, Enggaard, C, Supramaniyam, S, Lund, I K, Svenningsen, P, Hansen, P B L & Jensen, B L 2020, ' Plasminogen Deficiency and Amiloride Mitigate Angiotensin II-Induced Hypertension in Type 1 Diabetic Mice Suggesting Effects Through the Epithelial Sodium Channel ', Journal of the American Heart Association, vol. 9, no. 23, 016387, pp. 1-36 . https://doi.org/10.1161/JAHA.120.016387
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease
Andersen, H, Hansen, M H, Buhl, K B, Stæhr, M, Friis, U G, Enggaard, C, Supramaniyam, S, Lund, I K, Svenningsen, P, Hansen, P B L & Jensen, B L 2020, ' Plasminogen Deficiency and Amiloride Mitigate Angiotensin II-Induced Hypertension in Type 1 Diabetic Mice Suggesting Effects Through the Epithelial Sodium Channel ', Journal of the American Heart Association, vol. 9, no. 23, e016387 . https://doi.org/10.1161/JAHA.120.016387
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease
Andersen, H, Hansen, M H, Buhl, K B, Stæhr, M, Friis, U G, Enggaard, C, Supramaniyam, S, Lund, I K, Svenningsen, P, Hansen, P B L & Jensen, B L 2020, ' Plasminogen Deficiency and Amiloride Mitigate Angiotensin II-Induced Hypertension in Type 1 Diabetic Mice Suggesting Effects Through the Epithelial Sodium Channel ', Journal of the American Heart Association, vol. 9, no. 23, e016387 . https://doi.org/10.1161/JAHA.120.016387
BackgroundDiabetic nephropathy is a common diabetes mellitus complication associated with hypertension, proteinuria, and excretion of urinary plasmin that activates the epithelial sodium channel, ENaC, in vitro. Here we hypothesized that the deletion
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8ec5e6af86e7d6210a98217a6c0a40d4
https://curis.ku.dk/portal/da/publications/plasminogen-deficiency-and-amiloride-mitigate-angiotensin-iiinduced-hypertension-in-type-1-diabetic-mice-suggesting-effects-through-the-epithelial-sodium-channel(19a2798d-5153-41b9-bfa6-5dc8a2fdb58d).html
https://curis.ku.dk/portal/da/publications/plasminogen-deficiency-and-amiloride-mitigate-angiotensin-iiinduced-hypertension-in-type-1-diabetic-mice-suggesting-effects-through-the-epithelial-sodium-channel(19a2798d-5153-41b9-bfa6-5dc8a2fdb58d).html
Autor:
Kim Ravnskjaer, Henrik Dimke, Maria H. Hansen, Sonja S Hohmann, Susanne Mandrup, Sofie M. Bendixen, Bjørk Ditlev Larsen, Mike Krogh Terkelsen, Sönke Detlefsen, Ann-Britt Marcher
Publikováno v:
Scientific Reports
Scientific Reports, Vol 9, Iss 1, Pp 1-13 (2019)
Marcher, A-B, Bendixen, S M, Terkelsen, M K, Hohmann, S S, Hansen, M H, Larsen, B D, Mandrup, S, Dimke, H, Detlefsen, S & Ravnskjaer, K 2019, ' Transcriptional regulation of Hepatic Stellate Cell activation in NASH ', Scientific Reports, vol. 9, no. 1, 2324 . https://doi.org/10.1038/s41598-019-39112-6
Scientific Reports, Vol 9, Iss 1, Pp 1-13 (2019)
Marcher, A-B, Bendixen, S M, Terkelsen, M K, Hohmann, S S, Hansen, M H, Larsen, B D, Mandrup, S, Dimke, H, Detlefsen, S & Ravnskjaer, K 2019, ' Transcriptional regulation of Hepatic Stellate Cell activation in NASH ', Scientific Reports, vol. 9, no. 1, 2324 . https://doi.org/10.1038/s41598-019-39112-6
Non-alcoholic steatohepatitis (NASH) signified by hepatic steatosis, inflammation, hepatocellular injury, and fibrosis is a growing cause of chronic liver disease, cirrhosis, and hepatocellular carcinoma. Hepatic fibrosis resulting from accumulation