Výsledky vyhledávání - "Maria Antonietta Valli"

Acute phase HBV-specific T cell responses associated with HBV persistence after HBV/HCV coinfection

Autor: S. Cerioni, Maria Antonietta Valli, Barbara Amadei, Carolina Boni, Paola Fisicaro, Carlo Ferrari, Simona Urbani, Gabriele Missale

Publikováno v: Hepatology. 41:826-831

To characterize acute-phase hepatitis B virus (HBV)-specific T cell responses associated with self-limited and persistent HBV infections, we compared a patient with acute HBV/HCV coinfection, who was able to control HCV but developed chronic hepatiti

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fbf3d20dfa10c4bf5e8cd568f72c1494
https://doi.org/10.1002/hep.20614

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Recombinant interferon α (rIFN-α) does not potentiate the effect of iodine excess on the development of thyroid abnormalities in patients with HCV chronic active hepatitis

Autor: Claudia Schianchi, Elio Roti, Maria Grazia Magotti, Mario Salvi, Lewis E. Braverman, Franco Fiaccadori, Mario Pedrazzoni, Roberta Minelli, Maria Antonietta Valli

Publikováno v: Clinical Endocrinology. 50:95-100

OBJECTIVE To determine whether the administration of pharmacological quantities of iodine during interferon-alpha (rIFN-α) treatment of chronic viral hepatitis B and C (HCV) would exacerbate the potential adverse effects of rIFNα on thyroid functio

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::4315734bc0277545e2823cddaecb7273
https://doi.org/10.1046/j.1365-2265.1999.00616.x

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Is steroid therapy needed in the treatment of destructive thyrotoxicosis induced by alpha-interferon in chronic hepatitis C?

Autor: Paolo Chiodera, Maria Antonietta Valli, Roberto Delsignore, Gloria Saccani Jotti, Angela Luciana Barilli, Carlo Ferrari, Clelia Di Seclì, Vittorio Coiro, Lorenzo Finardi, Roberta Minelli, Roberto Bertoni

Publikováno v: Hormone research. 63(4)

Objective: Treatment with interferon (IFN) of patients affected by chronic hepatitis C (CH-C) may produce alterations in thyroid function, such as hypothyroidism, Graves’-like hyperthyroidism and destructive thyrotoxicosis (DT). IFN-induced DT is c

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ffb41ce3dd629231baecc6fcd14d8bf7
https://pubmed.ncbi.nlm.nih.gov/15860921

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