Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Mari Usutani"'
Autor:
Shinobu Takayasu, Satoru Mizushiri, Yutaka Watanuki, Satoshi Yamagata, Mari Usutani, Yuki Nakada, Yuko Asari, Shingo Murasawa, Kazunori Kageyama, Makoto Daimon
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-9 (2022)
Abstract Immune checkpoint inhibitors (ICIs) treatment can result in endocrine immune-related adverse events (irAEs), including pituitary dysfunction. Quick diagnosis of secondary adrenal insufficiency (AI) is challenging because no universal definit
Externí odkaz:
https://doaj.org/article/c0fd8f00198140619fcc21afe2473ae2
Autor:
Shingo Murasawa, Kazunori Kageyama, Mari Usutani, Yuko Asari, Noriko Kinoshita, Yuki Nakada, Yutaka Watanuki, Shinobu Takayasu, Makoto Daimon
Publikováno v:
Journal of the Renin-Angiotensin-Aldosterone System. 2023:1-9
Primary aldosteronism (PA) is the most common cause of endocrine hypertension. Unilateral PA can be cured using unilateral adrenalectomy (Adx). PA surgery outcome (PASO) criteria, which include clinical and biochemical outcomes, have been proposed to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4498ce84fb2c23df5a3d03961dfd6085
https://doi.org/10.1155/2023/5732812
https://doi.org/10.1155/2023/5732812
Autor:
Ken Terui, Mari Usutani, Shinobu Takayasu, Makoto Daimon, Kohei Saito, Satoshi Yamagata, Kazunori Kageyama, Takeshi Usui
Publikováno v:
Endocrine Journal. 69:115-120
17α-Hydroxylase/17,20-lyase deficiency (17OHD) is caused by pathogenic mutations in CYP17A1. Impaired 17α-hydroxylase and 17,20-lyase activities typically induce hypertension, hypokalemia, sexual infantilism, and amenorrhea. Most patients with 17OH
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8d174a59dc8c0a13ebecb413a0b52dbd
https://doi.org/10.1507/endocrj.ej21-0266
https://doi.org/10.1507/endocrj.ej21-0266
Autor:
Satoshi, Yamagata, Kazunori, Kageyama, Takeshi, Usui, Kohei, Saito, Shinobu, Takayasu, Mari, Usutani, Ken, Terui, Makoto, Daimon
Publikováno v:
Endocrine journal. 69(2)
17α-Hydroxylase/17,20-lyase deficiency (17OHD) is caused by pathogenic mutations in CYP17A1. Impaired 17α-hydroxylase and 17,20-lyase activities typically induce hypertension, hypokalemia, sexual infantilism, and amenorrhea. Most patients with 17OH
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::88355a9f4ee49cc4b64d31934d415e99
https://pubmed.ncbi.nlm.nih.gov/34483146
https://pubmed.ncbi.nlm.nih.gov/34483146
Autor:
Satoshi Yamagata, Kazunori Kageyama, Takeshi Usui, Kohei Saito, Shinobu Takayasu, Mari Usutani, Ken Terui, Makoto Daimon
Publikováno v:
Endocrine Journal; 2022, Vol. 69 Issue 2, p115-120, 6p
Autor:
Koshi Makita, Mari Usutani, Makoto Daimon, Shinobu Takayasu, Yuki Nakada-Nakayama, Akira Sugawara, Kazunori Kageyama
Publikováno v:
Neuroscience Letters. 711:134407
Glucocorticoids and glucocorticoid receptors (GRs) suppress pituitary pro-opiomelanocortin (Pomc) gene expression. O-linked β-N-acetylglucosamine (O-GlcNAc) modification, mediated by O-GlcNAc transferase (OGT), plays an important role during gene tr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d6ea38a3938d37f5916e804147ed6c8
https://doi.org/10.1016/j.neulet.2019.134407
https://doi.org/10.1016/j.neulet.2019.134407