Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Marcie Jiménez"'
Autor:
Silvia Olivera-Bravo, Anabel Fernández, María Noel Sarlabós, Juan Carlos Rosillo, Gabriela Casanova, Marcie Jiménez, Luis Barbeito
Publikováno v:
PLoS ONE, Vol 6, Iss 6, p e20831 (2011)
BACKGROUND: We have investigated whether an acute metabolic damage to astrocytes during the neonatal period may critically disrupt subsequent brain development, leading to neurodevelopmental disorders. Astrocytes are vulnerable to glutaric acid (GA),
Externí odkaz:
https://doaj.org/article/4e44ed40e7744c5fa81b87c01795d858
Autor:
Gabriela Casanova, Juan Carlos Rosillo, Marcie Jiménez, Anabel Fernández, Magela Rodao, Gaby Martínez, Eugenia Isasi, Nadia Presa Gau, Gabriel Otero, Mauricio Cabrera, Pablo Díaz‐Amarilla, Hugo Cerecetto, Mercedes González, Silvia Olivera‐Bravo
Publikováno v:
International Journal of Developmental Neuroscience. 83:274-296
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d5e456ff400ca08dcdb7d0cc40fa9090
https://doi.org/10.1002/jdn.10256
https://doi.org/10.1002/jdn.10256
Autor:
Gabriela Casanova, Eugenia Isasi, Pablo Díaz-Amarilla, Marcie Jiménez-Riani, Silvia Olivera-Bravo, Luis Barbeito
Publikováno v:
Cell and tissue research. 370(3)
In the rat model of amyotrophic lateral sclerosis expressing the G93A superoxide dismutase-1 mutation, motor neuron death and rapid paralysis progression are associated with the emergence of a population of aberrant glial cells (AbAs) that proliferat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::784a84bd97c159a060a6ba2f31bf7a89
https://pubmed.ncbi.nlm.nih.gov/28864831
https://pubmed.ncbi.nlm.nih.gov/28864831
Autor:
Juan Carlos Rosillo, Eugenia Isasi, Gabriela Casanova, Marcie Jiménez, Luis Barbeito, María Noel Sarlabós, Anabel Fernández, Silvia Olivera-Bravo
Publikováno v:
Neurotoxicity Research. 25:381-391
Glutaric acid (GA) is a neurotoxic metabolite that accumulates in the CNS of patients with glutaric acidemia-I (GA-I), a neurometabolic disease caused by deficient activity of glutaryl-CoA dehydrogenase. Most GA-I patients display characteristic CNS
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::df0b77c37c4f893d41a4316c3cf35381
https://doi.org/10.1007/s12640-013-9445-9
https://doi.org/10.1007/s12640-013-9445-9