Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Marcel de Brito van Velze"'
Autor:
Marcel de Brito Van Velze, Dhanasak Dhanasobhon, Marie Martinez, Annunziato Morabito, Emmanuelle Berthaux, Cibele Martins Pinho, Yann Zerlaut, Nelson Rebola
Publikováno v:
Cell Reports, Vol 43, Iss 5, Pp 114197- (2024)
Summary: Interneurons (INs), specifically those in disinhibitory circuits like somatostatin (SST) and vasoactive intestinal peptide (VIP)-INs, are strongly modulated by the behavioral context. Yet, the mechanisms by which these INs are recruited duri
Externí odkaz:
https://doaj.org/article/a6f9936fefc4479f8dff58308c3b54f3
Autor:
Martina Proietti Onori, Linda M C Koene, Carmen B Schäfer, Mark Nellist, Marcel de Brito van Velze, Zhenyu Gao, Ype Elgersma, Geeske M van Woerden
Publikováno v:
PLoS Biology, Vol 19, Iss 5, p e3001279 (2021)
Hyperactivation of the mammalian target of rapamycin (mTOR) pathway can cause malformation of cortical development (MCD) with associated epilepsy and intellectual disability (ID) through a yet unknown mechanism. Here, we made use of the recently iden
Externí odkaz:
https://doaj.org/article/8d4f132af9724879a8c6978f1a598618
Autor:
Simon Bossi, Dhanasak Dhanasobhon, Graham C.R. Ellis-Davies, Jimena Frontera, Marcel de Brito Van Velze, Joana Lourenço, Alvaro Murillo, Rafael Luján, Mariano Casado, Isabel Perez-Otaño, Alberto Bacci, Daniela Popa, Pierre Paoletti, Nelson Rebola
Publikováno v:
Neuron
Neuron, 2022, 110 (15), pp.2438-2454.e8. ⟨10.1016/j.neuron.2022.05.016⟩
Neuron, 2022, 110 (15), pp.2438-2454.e8. ⟨10.1016/j.neuron.2022.05.016⟩
GluN3A is an atypical glycine-binding subunit of NMDA receptors (NMDARs) whose actions in the brain are mostly unknown. Here, we show that the expression of GluN3A subunits controls the excitability of mouse adult cortical and amygdalar circuits via
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f8cc478e11c5942840fd099d4be80d3
https://hal.science/hal-03782718
https://hal.science/hal-03782718
Autor:
Carmen B. Schäfer, Zhenyu Gao, Mark Nellist, Marcel de Brito van Velze, Martina Proietti Onori, Ype Elgersma, Geeske M. van Woerden, Linda M. C. Koene
Publikováno v:
PLoS Biology
PLoS Biology, 19(5):e3001279. Public Library of Science
PLoS Biology, Vol 19, Iss 5, p e3001279 (2021)
Plos Biology
PLoS Biology, 19(5):e3001279. Public Library of Science
PLoS Biology, Vol 19, Iss 5, p e3001279 (2021)
Plos Biology
Hyperactivation of the mammalian target of rapamycin (mTOR) pathway can cause malformation of cortical development (MCD) with associated epilepsy and intellectual disability (ID) through a yet unknown mechanism. Here, we made use of the recently iden
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::367aebb6d0451a001d88fd4d67aa8bc5
https://doi.org/10.1371/journal.pbio.3001279
https://doi.org/10.1371/journal.pbio.3001279