Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Marc Steinle"'
Autor:
Friederike A. Steudel, Corinna J. Mohr, Benjamin Stegen, Hoang Y. Nguyen, Andrea Barnert, Marc Steinle, Sandra Beer‐Hammer, Pierre Koch, Wing‐Yee Lo, Werner Schroth, Reiner Hoppe, Hiltrud Brauch, Peter Ruth, Stephan M. Huber, Robert Lukowski
Publikováno v:
Molecular Oncology, Vol 11, Iss 9, Pp 1172-1188 (2017)
Oncogenic signalling via Ca2+‐activated K+ channels of intermediate conductance (SK4, also known as KCa3.1 or IK) has been implicated in different cancer entities including breast cancer. Yet, the role of endogenous SK4 channels for tumorigenesis i
Externí odkaz:
https://doaj.org/article/d69d7c0b9d5d4580b6bcaccda5f3a017
Autor:
Sandra Beer-Hammer, Andrea Barnert, Corinna J. Mohr, Pierre Koch, Werner Schroth, Benjamin Stegen, Peter Ruth, Robert Lukowski, Wing-Yee Lo, Stephan M. Huber, Hiltrud Brauch, Reiner Hoppe, Friederike A. Steudel, Hoang Y. Nguyen, Marc Steinle
Publikováno v:
Molecular Oncology
Oncogenic signalling via Ca2+-activated K+ channels of intermediate conductance (SK4, also known as KCa3.1 or IK) has been implicated in different cancer entities including breast cancer. Yet, the role of endogenous SK4 channels for tumourigenesis is
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::44a08718b198cbe05b2e8db6798a3657
http://europepmc.org/articles/PMC5579333
http://europepmc.org/articles/PMC5579333
Autor:
Daniela Palme, Marc Steinle, Klaus Dittmann, Stephan M. Huber, Peter Ruth, Justine Rudner, Milan Misovic, Robert Lukowski
Publikováno v:
Radiotherapy and Oncology. 101:122-126
Background and purpose Glioblastoma cells express high levels of Ca 2+ -activated BK K + channels which have been proposed to be indispensable for glioblastoma proliferation and migration. Since migration of glioblastoma cells is reportedly stimulate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::162b1d5d10fa9932bb0cb533da5c5b24
https://doi.org/10.1016/j.radonc.2011.05.069
https://doi.org/10.1016/j.radonc.2011.05.069