Zobrazeno 1 - 5
of 5
pro vyhledávání: '"María G. Ledesma-Colunga"'
Autor:
Carmen Clapp, Georgina Ortiz, Jose F. García-Rodrigo, María G. Ledesma-Colunga, Oscar F. Martínez-Díaz, Norma Adán, Gonzalo Martínez de la Escalera
Publikováno v:
Frontiers in Endocrinology, Vol 13 (2022)
The term inflammatory arthritis defines a family of diseases, including rheumatoid arthritis (RA), caused by an overactive immune system, and influenced by host aspects including sex, reproductive state, and stress. Prolactin (PRL) is a sexually dimo
Externí odkaz:
https://doaj.org/article/3b8872ee9ec542ef8ef7aeb7feca8dd4
Autor:
Maria G. Ledesma-Colunga, Vanessa Passin, Maja Vujic Spasic, Lorenz C. Hofbauer, Ulrike Baschant, Martina Rauner
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-12 (2024)
Abstract Iron is an essential nutrient for all living organisms. Both iron deficiency and excess can be harmful. Bone, a highly metabolic active organ, is particularly sensitive to fluctuations in iron levels. In this study, we investigated the effec
Externí odkaz:
https://doaj.org/article/b2489d71ce5a4ab2901d9eeeb41f5e37
Autor:
Maria G. Ledesma-Colunga, Ulrike Baschant, Heike Weidner, Tiago C. Alves, Peter Mirtschink, Lorenz C. Hofbauer, Martina Rauner
Publikováno v:
Redox Biology, Vol 60, Iss , Pp 102616- (2023)
Objective: Rheumatoid arthritis is an inflammatory joint disease in which synovial iron deposition has been described. Transferrin receptor 2 (Tfr2) represents a critical regulator of systemic iron levels. Loss of Tfr2 function in humans and mice res
Externí odkaz:
https://doaj.org/article/b6dfec1e6fe644efb74ea14a924671ec
Publikováno v:
Bone Reports, Vol 14, Iss , Pp 100778- (2021)
Externí odkaz:
https://doaj.org/article/b0ae6baa6b5f4cd5a31e2786b17c65a2
Autor:
Maria G. Ledesma-Colunga, Norma Adán, Georgina Ortiz, Mariana Solís-Gutiérrez, Fernando López-Barrera, Gonzalo Martínez de la Escalera, Carmen Clapp
Publikováno v:
Arthritis Research & Therapy, Vol 19, Iss 1, Pp 1-16 (2017)
Abstract Background Prolactin (PRL) reduces joint inflammation, pannus formation, and bone destruction in rats with polyarticular adjuvant-induced arthritis (AIA). Here, we investigate the mechanism of PRL protection against bone loss in AIA and in m
Externí odkaz:
https://doaj.org/article/23930d7dcc264a84a87a18dbfefdbc8a