Zobrazeno 1 - 4
of 4
pro vyhledávání: '"María Duran-Moreno"'
Autor:
Laura Cubas-Nuñez, María Duran-Moreno, Jessica Castillo-Villalba, Jorge Fuentes-Maestre, Bonaventura Casanova, José M. García-Verdugo, Sara Gil-Perotín
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 11 (2017)
In situ RT-PCR detects and amplifies mRNA (cDNA) while obtaining spatial information of gene expression. When the intended use is an ultrastructural analysis of morphology, the procedure may be technically challenging and quality of tissue dramatical
Externí odkaz:
https://doaj.org/article/efebc4fa4e0a465fbc78f8769cbb3317
Autor:
Sara, Gil-Perotín, María, Duran-Moreno, Arantxa, Cebrián-Silla, Mónica, Ramírez, Paula, García-Belda, José Manuel, García-Verdugo
Publikováno v:
Anatomical record (Hoboken, N.J. : 2007). 296(9)
The possibility of obtaining large numbers of cells with potential to become functional neurons implies a great advance in regenerative medicine. A source of cells for therapy is the subventricular zone (SVZ) where adult neural stem cells (NSCs) reta
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::6a3592279b4552790d4e76651b4146d1
https://pubmed.ncbi.nlm.nih.gov/23904071
https://pubmed.ncbi.nlm.nih.gov/23904071
Autor:
Zaman Mirzadeh, Yael Kusne, Maria Duran-Moreno, Elaine Cabrales, Sara Gil-Perotin, Christian Ortiz, Bin Chen, Jose Manuel Garcia-Verdugo, Nader Sanai, Arturo Alvarez-Buylla
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-12 (2017)
Ependymal cells lining the adult brain ventricles are comprised of multiciliated cells and a rare subpopulation with two cilia (E2 cells) whose origin and function remain unknown. Here the authors find E2 cells in the 3rd ventricle of mice and humans
Externí odkaz:
https://doaj.org/article/0d4d22cbb44b43e885381003940754c8
Autor:
Ulises Gómez-Pinedo, Maria Salomé Sirerol-Piquer, María Durán-Moreno, José Manuel García-Verdugo, Jorge Matias-Guiu
Publikováno v:
Frontiers in Neurology, Vol 8 (2017)
BackgroundAlexander disease (AxD) is a rare disease caused by mutations in the gene encoding glial fibrillary acidic protein (GFAP). The disease is characterized by presence of GFAP aggregates in the cytoplasm of astrocytes and loss of myelin.Objecti
Externí odkaz:
https://doaj.org/article/52d1eaca91fd449989ff617ffce8b9a1