Zobrazeno 1 - 10
of 33
pro vyhledávání: '"Mar, Puigdellívol"'
Autor:
Stefan Milde, Francesca W. van Tartwijk, Anna Vilalta, Tamara C. Hornik, Jacob M. Dundee, Mar Puigdellívol, Guy C. Brown
Publikováno v:
Journal of Neuroinflammation, Vol 18, Iss 1, Pp 1-9 (2021)
Abstract Inflammation may contribute to multiple brain pathologies. One cause of inflammation is lipopolysaccharide/endotoxin (LPS), the levels of which are elevated in blood and/or brain during bacterial infections, gut dysfunction and neurodegenera
Externí odkaz:
https://doaj.org/article/890f380a8f9344229c6b9ea73e367087
Autor:
Iván Ballasch, Esther García-García, Cristina Vila, Anna Pérez-González, Anna Sancho-Balsells, Jéssica Fernández, David Soto, Mar Puigdellívol, Xavier Gasull, Jordi Alberch, Manuel J. Rodríguez, Josep M. Canals, Albert Giralt
Publikováno v:
Brain, Behavior, and Immunity. 109:144-161
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 14 (2020)
Microglia are brain macrophages that mediate neuroinflammation and contribute to and protect against neurodegeneration. The terminal sugar residue of all glycoproteins and glycolipids on the surface of mammalian cells is normally sialic acid, and add
Externí odkaz:
https://doaj.org/article/3a3e8e245d344b5e8b6ce6d85ebe57fe
Autor:
David Moreno-Delgado, Mar Puigdellívol, Estefanía Moreno, Mar Rodríguez-Ruiz, Joaquín Botta, Paola Gasperini, Anna Chiarlone, Lesley A Howell, Marco Scarselli, Vicent Casadó, Antoni Cortés, Sergi Ferré, Manuel Guzmán, Carmen Lluís, Jordi Alberch, Enric I Canela, Silvia Ginés, Peter J McCormick
Publikováno v:
eLife, Vol 9 (2020)
Early Huntington’s disease (HD) include over-activation of dopamine D1 receptors (D1R), producing an imbalance in dopaminergic neurotransmission and cell death. To reduce D1R over-activation, we present a strategy based on targeting complexes of D1
Externí odkaz:
https://doaj.org/article/e29f5e9398d54a2882b616c452b949ae
Publikováno v:
Frontiers in Immunology, Vol 10 (2019)
Opsonins are soluble, extracellular proteins, released by activated immune cells, and when bound to a target cell, can induce phagocytes to phagocytose the target cell. There are three known classes of opsonin: antibodies, complement factors and secr
Externí odkaz:
https://doaj.org/article/398a3259158947e295c43f137d74e7c3
Autor:
Alejandro Carrillo-Jimenez, Mar Puigdellívol, Anna Vilalta, Jose Luis Venero, Guy Charles Brown, Peter StGeorge-Hyslop, Miguel Angel Burguillos
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 12 (2018)
Microglia, the resident immune cells of the brain, have multiple functions in physiological and pathological conditions, including Alzheimer’s disease (AD). The use of primary microglial cell cultures has proved to be a valuable tool to study micro
Externí odkaz:
https://doaj.org/article/449fed1809fe4879bc27d4e932858aef
Publikováno v:
Aging Cell. 22
Publikováno v:
Aging cellREFERENCES.
Aging causes loss of brain synapses and memory, and microglial phagocytosis of synapses may contribute to this loss. Stressed neurons can release the nucleotide UTP, which is rapidly converted into UDP, that in turn activates the P2Y
Autor:
Mar Puigdellívol, Stefan Milde, Anna Vilalta, Tom O.J. Cockram, David H. Allendorf, Jeffrey Y. Lee, Jacob M. Dundee, Katryna Pampuščenko, Vilmante Borutaite, Hugh N. Nuthall, Jack H. Brelstaff, Maria Grazia Spillantini, Guy C. Brown
Publikováno v:
Cell Reports
Summary Microglia are implicated in neurodegeneration, potentially by phagocytosing neurons, but it is unclear how to block the detrimental effects of microglia while preserving their beneficial roles. The microglial P2Y6 receptor (P2Y6R) – activat
Autor:
Guy C. Brown, Jacob M Dundee, Francesca W. van Tartwijk, Anna Vilalta, Mar Puigdellívol, Stefan Milde, Tamara C. Hornik
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 18, Iss 1, Pp 1-9 (2021)
Dipòsit Digital de la UB
Universidad de Barcelona
Journal of Neuroinflammation, Vol 18, Iss 1, Pp 1-9 (2021)
Dipòsit Digital de la UB
Universidad de Barcelona
Inflammation may contribute to multiple brain pathologies. One cause of inflammation is lipopolysaccharide/endotoxin (LPS), the levels of which are elevated in blood and/or brain during bacterial infections, gut dysfunction and neurodegenerative dise
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::68aa90054ad6eb77dc45b70d83c39f22