Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Mao-Jun Liao"'
Autor:
Wei Yang, Yan Xiang, Mao-Jun Liao, Peng-Fei Wu, Lin Yang, Guo-Hao Huang, Bao-Zhong Shi, Liang Yi, Sheng-Qing Lv
Publikováno v:
Cell Communication and Signaling, Vol 19, Iss 1, Pp 1-16 (2021)
Abstract Background Alzheimer’s disease (AD) and glioblastoma are the most common and devastating diseases in the neurology and neurosurgery departments, respectively. Our previous research reports that the AD-related protein Presenilin1 represses
Externí odkaz:
https://doaj.org/article/a11393aa34b849108ff50c609a5f3e80
Publikováno v:
Scientific Reports, Vol 10, Iss 1, Pp 1-15 (2020)
Abstract The invasion ability of glioblastoma (GBM) causes tumor cells to infiltrate the surrounding brain parenchyma and leads to poor outcomes. Transient receptor potential vanilloid 4 (TRPV4) exhibits a remarkable role in cancer cell motility, but
Externí odkaz:
https://doaj.org/article/0504ec51c83a47389dc2d5bd205e65c0
Presenilin1 exerts antiproliferative effects by repressing the Wnt/β-catenin pathway in glioblastoma
Publikováno v:
Cell Communication and Signaling, Vol 18, Iss 1, Pp 1-13 (2020)
Abstract Background Glioblastoma and Alzheimer’s disease (AD) are the most common and devastating diseases in the central nervous system. The dysfunction of Presenilin1 is the main reason for AD pathogenesis. However, the molecular function of Pres
Externí odkaz:
https://doaj.org/article/0b2e9f5fc1d34ccbb31ce77d08e12872
Autor:
Guo-Hao Huang, Liang Yi, Yan Xiang, Mao-jun Liao, Lin Yang, Wei Yang, Bao-Zhong Shi, Sheng-Qing Lv, Pengfei Wu
Publikováno v:
Cell Communication and Signaling, Vol 19, Iss 1, Pp 1-16 (2021)
Cell Communication and Signaling : CCS
Cell Communication and Signaling : CCS
Background Alzheimer’s disease (AD) and glioblastoma are the most common and devastating diseases in the neurology and neurosurgery departments, respectively. Our previous research reports that the AD-related protein Presenilin1 represses cell prol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46a37f62455fc987a849463b96f5d6d6
https://doaj.org/article/a11393aa34b849108ff50c609a5f3e80
https://doaj.org/article/a11393aa34b849108ff50c609a5f3e80
Publikováno v:
Scientific Reports, Vol 10, Iss 1, Pp 1-15 (2020)
Scientific Reports
Scientific Reports
The invasion ability of glioblastoma (GBM) causes tumor cells to infiltrate the surrounding brain parenchyma and leads to poor outcomes. Transient receptor potential vanilloid 4 (TRPV4) exhibits a remarkable role in cancer cell motility, but the cont
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::595b5b88503038b8dc9cea53392f1b95
https://doaj.org/article/0504ec51c83a47389dc2d5bd205e65c0
https://doaj.org/article/0504ec51c83a47389dc2d5bd205e65c0
Autor:
Lunshan Xu, Peng-fei Wu, Liang Yi, Wei Yang, Mao-jun Liao, Jian-xing Ma, Jingyu Zhang, Minhui Xu
Publikováno v:
Oncology Letters
Intra-tumor heterogeneity (ITH) is one of the most important causes of therapy resistance, which eventually leads to the poor outcomes observed in patients with glioma. Mutant-allele tumor heterogeneity (MATH) values are based on whole-exon sequencin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::02484c786b5efd9bd5aa1a727cbdcf64
http://europepmc.org/articles/PMC6865645
http://europepmc.org/articles/PMC6865645
Autor:
Mao-jun Liao, Lunshan Xu, Peng-fei Wu, Minhui Xu, Wei Yang, Jian-xing Ma, Liang Yi, Xuhui Wang
Publikováno v:
Cell Death and Disease, Vol 10, Iss 3, Pp 1-15 (2019)
Cell Death & Disease
Cell Death & Disease
High aggressiveness is a hallmark of glioblastoma and predicts poor prognosis of patients with glioblastoma. The expression level of sortilin has been preliminarily reported to be elevated in high-grade glioma; however, the potential significance of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9b0f674597fe3dffe5bf2360558f280c
http://link.springer.com/article/10.1038/s41419-019-1449-9
http://link.springer.com/article/10.1038/s41419-019-1449-9
Presenilin1 exerts antiproliferative effects by repressing the Wnt/β-catenin pathway in glioblastoma
Publikováno v:
Cell Communication and Signaling : CCS
Cell Communication and Signaling, Vol 18, Iss 1, Pp 1-13 (2020)
Cell Communication and Signaling, Vol 18, Iss 1, Pp 1-13 (2020)
Background Glioblastoma and Alzheimer’s disease (AD) are the most common and devastating diseases in the central nervous system. The dysfunction of Presenilin1 is the main reason for AD pathogenesis. However, the molecular function of Presenilin1 a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c783a2c2abf56be7cf4f0e665b23dbea
http://europepmc.org/articles/PMC7014622
http://europepmc.org/articles/PMC7014622