Výsledky vyhledávání - "Manuela Moraru"

Akademický článek

Immunological features of patients affected by Barraquer-Simons syndrome

Autor: Fernando Corvillo, Giovanni Ceccarini, Pilar Nozal, Silvia Magno, Caterina Pelosini, Sofía Garrido, Alberto López-Lera, Manuela Moraru, Carlos Vilches, Silvia Fornaciari, Sabrina Gabbriellini, Ferruccio Santini, David Araújo-Vilar, Margarita López-Trascasa

Publikováno v: Orphanet Journal of Rare Diseases, Vol 15, Iss 1, Pp 1-11 (2020)

Abstract Background C3 hypocomplementemia and the presence of C3 nephritic factor (C3NeF), an autoantibody causing complement system over-activation, are common features among most patients affected by Barraquer-Simons syndrome (BSS), an acquired for

Externí odkaz: https://doaj.org/article/a0286d5c92324b9698ac45f6544d6096

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Akademický článek

FCGR Genetic Variation in Two Populations From Ecuador Highlands—Extensive Copy-Number Variation, Distinctive Distribution of Functional Polymorphisms, and a Novel, Locally Common, Chimeric FCGR3B/A (CD16B/A) Gene

Autor: Manuela Moraru, Adriana Perez-Portilla, Karima Al-Akioui Sanz, Alfonso Blazquez-Moreno, Antonio Arnaiz-Villena, Hugh T. Reyburn, Carlos Vilches

Publikováno v: Frontiers in Immunology, Vol 12 (2021)

Fcγ receptors (FcγR), cell-surface glycoproteins that bind antigen-IgG complexes, control both humoral and cellular immune responses. The FCGR locus on chromosome 1q23.3 comprises five homologous genes encoding low-affinity FcγRII and FcγRIII, an

Externí odkaz: https://doaj.org/article/e666edbf664d425997f1f13216521c3f

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Akademický článek

Haplotype-Based Analysis of KIR-Gene Profiles in a South European Population—Distribution of Standard and Variant Haplotypes, and Identification of Novel Recombinant Structures

Autor: Elisa Cisneros, Manuela Moraru, Natalia Gómez-Lozano, Aura Muntasell, Miguel López-Botet, Carlos Vilches

Publikováno v: Frontiers in Immunology, Vol 11 (2020)

Inhibitory Killer-cell Immunoglobulin-like Receptors (KIR) specific for HLA class I molecules enable human natural killer cells to monitor altered antigen presentation in pathogen-infected and tumor cells. KIR genes display extensive copy-number vari

Externí odkaz: https://doaj.org/article/e06a7268799144a7917e31e06f5d233c

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Akademický článek

Human Cytomegalovirus Antigen Presentation by HLA-DR+ NKG2C+ Adaptive NK Cells Specifically Activates Polyfunctional Effector Memory CD4+ T Lymphocytes

Autor: Marcel Costa-García, Michelle Ataya, Manuela Moraru, Carlos Vilches, Miguel López-Botet, Aura Muntasell

Publikováno v: Frontiers in Immunology, Vol 10 (2019)

Natural killer (NK) cells play a dual role in the defense against viral pathogens by directly lysing infected cells as well as by regulating anti-viral T cell immunity. Infection by human cytomegalovirus (HCMV) promotes a persistent expansion of NKG2

Externí odkaz: https://doaj.org/article/710d22039a3e42f7a9309d9c3b341d18

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Akademický článek

Correction to: Immunological features of patients affected by Barraquer-Simons syndrome

Publikováno v: Orphanet Journal of Rare Diseases, Vol 15, Iss 1, Pp 1-1 (2020)

Following the publication of the original article [1], the authors have requested to amend the Abstract and Discussion section as follows.

Externí odkaz: https://doaj.org/article/8c6561c987144b70910d7a50dfabeb72

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Akademický článek

Activated allogeneic NK cells preferentially kill poor prognosis B-cell chronic lymphocytic leukemia cells

Autor: Diego Sanchez-Martinez, Pilar Lanuza, Natalia Gomez, Aura Muntasell, Elisa Cisneros Leralta, Manuela Moraru, Gemma Azaceta, Alberto Anel, Luis Martínez-Lostao, Martin Villalba, Luis Palomera, Carlos Vilches, Jose A García Marco, Julian Pardo

Publikováno v: Frontiers in Immunology, Vol 7 (2016)

Mutational status of TP53 together with expression of wild type (wt) IGHV represents the most widely accepted biomarkers, establishing a very poor prognosis in B-cell chronic lymphocytic leukemia (B-CLL) patients. Adoptive cell therapy using allogene

Externí odkaz: https://doaj.org/article/39abf86fe4ec4145959151c4868b0b49

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Data from High Numbers of Circulating CD57+ NK Cells Associate with Resistance to HER2-Specific Therapeutic Antibodies in HER2+ Primary Breast Cancer

Natural killer (NK) cells can orchestrate effective antitumor immunity. The presence of tumor-infiltrating NK cells in diagnostic biopsies predicts pathologic complete response (pCR) to HER2-specific therapeutic antibodies in patients with primary br

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e5efee955b03a6fc572b6e8fe6efaed
https://doi.org/10.1158/2326-6066.c.6550095.v1

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Supplementary Figures and Tables from High Numbers of Circulating CD57+ NK Cells Associate with Resistance to HER2-Specific Therapeutic Antibodies in HER2+ Primary Breast Cancer

Supplementary Figures 1-6 and Tables 1-3

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7644965ef2ac1be46eb1c0ef4931129d
https://doi.org/10.1158/2326-6066.22542945.v1

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Immunological features of patients affected by Barraquer-Simons syndrome

Autor: Pilar Nozal, Fernando Corvillo, Sabrina Gabbriellini, David Araújo-Vilar, Margarita López-Trascasa, Sofía Garrido, Silvia Fornaciari, Silvia Magno, Giovanni Ceccarini, Caterina Pelosini, Carlos Vilches, Manuela Moraru, Ferruccio Santini, Alberto López-Lera

Publikováno v: Orphanet Journal of Rare Diseases, Vol 15, Iss 1, Pp 1-11 (2020)
Minerva. Repositorio Institucional de la Universidad de Santiago de Compostela
instname
Biblos-e Archivo: Repositorio Institucional de la UAM
Universidad Autónoma de Madrid
Biblos-e Archivo. Repositorio Institucional de la UAM
Orphanet Journal of Rare Diseases

Background: C3 hypocomplementemia and the presence of C3 nephritic factor (C3NeF), an autoantibody causing complement system over-activation, are common features among most patients affected by Barraquer-Simons syndrome (BSS), an acquired form of par

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb8bc2ae910c6141bad3f4a9c091b5b9
https://doaj.org/article/a0286d5c92324b9698ac45f6544d6096

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