Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Manfred Anim"'
Autor:
Manfred Anim, Georgios Sogkas, Gunnar Schmidt, Natalia Dubrowinskaja, Torsten Witte, Reinhold Ernst Schmidt, Faranaz Atschekzei
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
Externí odkaz:
https://doaj.org/article/c94f845c06d4499ba22944c154058974
Autor:
Torsten Witte, Georgios Sogkas, Natalia Dubrowinskaja, Manfred Anim, Faranaz Atschekzei, Thea Thiele
Publikováno v:
RMD Open, Vol 8, Iss 2 (2022)
Objectives Besides adaptive immunity genes, genetic risk factors for psoriatic arthritis (PsA) include innate immunity loci, which suggests an autoinflammatory disease mechanism, at least in a subset of patients. Here, we aimed at investigating the a
Externí odkaz:
https://doaj.org/article/63c09c2e144f47d190d3b241fcac884d
Autor:
Manfred Anim, Georgios Sogkas, Gunnar Schmidt, Natalia Dubrowinskaja, Torsten Witte, Reinhold Ernst Schmidt, Faranaz Atschekzei
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
NF-κB1 deficiency is suggested to be the most common cause of common variable immunodeficiency (CVID). NFKB1 encodes for the p105 precursor protein of NF-κB1, which is converted into the active transcriptional subunit p50 through proteasomal proces
Externí odkaz:
https://doaj.org/article/10d12765ce0d4b689e3d01538c11a056
Autor:
Emmanuel Amlabu, Philip Ilani, Grace Opoku, Prince B. Nyarko, Evelyn Quansah, Laty G. Thiam, Manfred Anim, Reuben Ayivor-Djanie, Ojo-ajogu Akuh, Henrietta Mensah-Brown, Julian C. Rayner, Gordon A. Awandare
Publikováno v:
Frontiers in Cellular and Infection Microbiology, Vol 10 (2020)
Nearly half of the genes in the Plasmodium falciparum genome have not yet been functionally investigated. We used homology-based structural modeling to identify multiple copies of Armadillo repeats within one uncharacterized gene expressed during the
Externí odkaz:
https://doaj.org/article/37e7c64db27a4e3f91e6e2e225d6a275
Autor:
Torsten Witte, Reinhold E. Schmidt, Georgios Sogkas, Ignatius Ryan Adriawan, Manfred Anim, Natalia Dubrowinskaja, Faranaz Atschekzei
Publikováno v:
Annals of the Rheumatic Diseases. 80:392-399
ObjectivesTreatment of rheumatic diseases requires immunomodulatory agents which can compromise antibody production. However, even in case of agents directly targeting B cells, a minority of patients develop hypogammaglobulinaemia, suggesting a genet