Zobrazeno 1 - 10
of 49
pro vyhledávání: '"Mandayam O. Nandan"'
Publikováno v:
Stem Cell Research, Vol 14, Iss 1, Pp 10-19 (2015)
Krüppel-like factor 5 (KLF5) is a pro-proliferative transcription factor that is expressed in dividing epithelial cells of the intestinal crypt. Leucine-rich repeat-containing G-protein coupled receptor 5 (Lgr5) has been identified as a stem cell ma
Externí odkaz:
https://doaj.org/article/0b322d42fbec404bb3c2549c8a789e92
The zinc finger-containing transcription factor, Krüppel-like factor 4 (KLF4), inhibits cell proliferation. An in vivo tumor-suppressive role for KLF4 is shown by the recent finding that Klf4 haploinsufficiency in ApcMin/+ mice promotes intestinal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::785c72160b3192fb712775f2f96514bd
https://doi.org/10.1158/1541-7786.c.6541801
https://doi.org/10.1158/1541-7786.c.6541801
Supplementary Figures S1-S8 from Notch Inhibits Expression of the Krüppel-Like Factor 4 Tumor Suppressor in the Intestinal Epithelium
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::77a1160712c45d2e437bce4b54709d36
https://doi.org/10.1158/1541-7786.22517692
https://doi.org/10.1158/1541-7786.22517692
Autor:
Vincent W. Yang, Klaus H. Kaestner, Jonathan P. Katz, Mandayam O. Nandan, Beth B. McConnell, Amr M. Ghaleb
Supplementary Figure 3 from Haploinsufficiency of Krüppel-Like Factor 4 Promotes Adenomatous Polyposis Coli–Dependent Intestinal Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6a8e4a30e2f27bf55accffcdfb80ff8a
https://doi.org/10.1158/0008-5472.22367760.v1
https://doi.org/10.1158/0008-5472.22367760.v1
Autor:
Vincent W. Yang, Frank J. Gordon, Amr M. Ghaleb, Mandayam O. Nandan, Agnieszka B. Bialkowska, Beth B. McConnell
Supplementary Methods and Materials from Haploinsufficiency of Krüppel-Like Factor 5 Rescues the Tumor-Initiating Effect of the ApcMin Mutation in the Intestine
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f1080ca5914c55cfb8b04b35d0c90d5
https://doi.org/10.1158/0008-5472.22378761
https://doi.org/10.1158/0008-5472.22378761
Autor:
Vincent W. Yang, Frank J. Gordon, Amr M. Ghaleb, Mandayam O. Nandan, Agnieszka B. Bialkowska, Beth B. McConnell
Supplementary Figure 3 from Haploinsufficiency of Krüppel-Like Factor 5 Rescues the Tumor-Initiating Effect of the ApcMin Mutation in the Intestine
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be670433cf890204102f80810b373134
https://doi.org/10.1158/0008-5472.22378767.v1
https://doi.org/10.1158/0008-5472.22378767.v1
Autor:
Vincent W. Yang, Klaus H. Kaestner, Jonathan P. Katz, Mandayam O. Nandan, Beth B. McConnell, Amr M. Ghaleb
Supplementary Figure 1 from Haploinsufficiency of Krüppel-Like Factor 4 Promotes Adenomatous Polyposis Coli–Dependent Intestinal Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::01224bcd65fe2add8dff73e0dc26b6ad
https://doi.org/10.1158/0008-5472.22367766
https://doi.org/10.1158/0008-5472.22367766
The mitotic checkpoint is a mechanism that arrests the progression to anaphase until all chromosomes have achieved proper attachment to mitotic spindles. In cancer cells, satisfaction of this checkpoint is frequently delayed or prevented by various d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::45805f7751715096c22a0b2617710dba
https://doi.org/10.1158/0008-5472.c.6496376
https://doi.org/10.1158/0008-5472.c.6496376
Autor:
Vincent W. Yang, Klaus H. Kaestner, Jonathan P. Katz, Mandayam O. Nandan, Beth B. McConnell, Amr M. Ghaleb
Supplementary Figure 2 from Haploinsufficiency of Krüppel-Like Factor 4 Promotes Adenomatous Polyposis Coli–Dependent Intestinal Tumorigenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9ac4ef0ad838b33b68156f869a2fd55d
https://doi.org/10.1158/0008-5472.22367763.v1
https://doi.org/10.1158/0008-5472.22367763.v1
Autor:
Vincent W. Yang, Frank J. Gordon, Amr M. Ghaleb, Mandayam O. Nandan, Agnieszka B. Bialkowska, Beth B. McConnell
Inactivation of the tumor suppressor adenomatous polyposis coli, with the resultant activation of β-catenin, is the initiating event in the development of a majority of colorectal cancers. Krüppel-like factor 5 (KLF5), a proproliferative transcrip
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::58ed8a756f6e8634332b69e52d41f583
https://doi.org/10.1158/0008-5472.c.6499014
https://doi.org/10.1158/0008-5472.c.6499014