Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Man Hau Ho"'
Autor:
Man-Hau Ho, Yih-Jeng Tsai, Chia-Yen Chen, Anastasia Yang, Thierry Burnouf, Yun Wang, Yung-Hsiao Chiang, Barry J. Hoffer, Szu-Yi Chou
Publikováno v:
Journal of Biomedical Science, Vol 31, Iss 1, Pp 1-22 (2024)
Abstract Background Traumatic brain injury (TBI) causes axon tearing and synapse degradation, resulting in multiple neurological dysfunctions and exacerbation of early neurodegeneration; the repair of axonal and synaptic structures is critical for re
Externí odkaz:
https://doaj.org/article/a99233fd0b8446cc9e60cc3921f12614
Autor:
Man-Hau Ho, Chia-Hung Yen, Tsung-Hsun Hsieh, Tzu-Jen Kao, Jing-Yuan Chiu, Yung-Hsiao Chiang, Barry J. Hoffer, Wen-Chang Chang, Szu-Yi Chou
Publikováno v:
Redox Biology, Vol 46, Iss , Pp 102067- (2021)
Traumatic brain injury (TBI) is a prevalent head injury worldwide which increases the risk of neurodegenerative diseases. Increased reactive oxygen species (ROS) and inflammatory chemokines after TBI induces secondary effects which damage neurons. Ta
Externí odkaz:
https://doaj.org/article/91593b3bc5904dee8b942b464cbec397
Autor:
Tzu Jen Kao, Szu Yi Chou, Jing Yuan Chiu, Barry J. Hoffer, Chia Hung Yen, Tsung Hsun Hsieh, Wen Chang Chang, Man Hau Ho, Yung Hsiao Chiang
Publikováno v:
Redox Biology
Redox Biology, Vol 46, Iss, Pp 102067-(2021)
Redox Biology, Vol 46, Iss, Pp 102067-(2021)
Traumatic brain injury (TBI) is a prevalent head injury worldwide which increases the risk of neurodegenerative diseases. Increased reactive oxygen species (ROS) and inflammatory chemokines after TBI induces secondary effects which damage neurons. Ta
Autor:
Hui Min Chen, Yuan Chin Hsiung, Yuan Hao Chen, Cheng Yang Lee, Wen Chang Chang, Man Hau Ho, Chiu Jing-Yuan, Yung Hsiao Chiang, Szu Yi Chou, Tzu Hao Chang, Yun Wang, Chun A. Changou, You Yin Chen, Barry J. Hoffer, Reni Ajoy, Yu Chun Lo
Publikováno v:
Molecular Psychiatry
Glucoregulatory efficiency and ATP production are key regulators for neuronal plasticity and memory formation. Besides its chemotactic and neuroinflammatory functions, the CC chemokine––CCL5 displays neurotrophic activity. We found impaired learn