Zobrazeno 1 - 10
of 96
pro vyhledávání: '"Mallick HN"'
Autor:
Mallick Hn, Nasreen Akhtar
Publikováno v:
The National medical journal of India. 32(1)
Publikováno v:
Sleep and Vigilance. 1:3-5
Publikováno v:
Sleep and Vigilance. 2:101-102
Publikováno v:
NeuroImage. 26:29-35
Changes in the activity of the basal forebrain sleep regulating areas were studied noninvasively in conscious rats by employing functional magnetic resonance imaging (fMRI). Sleep–wakefulness (S–W) stages were identified with the help of electrop
Publikováno v:
Magnetic Resonance in Medicine. 49:962-967
A stereotaxic restraining assembly was designed and developed for simultaneous electrophysiological recordings and functional MRI (fMRI) data acquisition from a conscious rat. The design of the nonmagnetic stereotaxic apparatus facilitated the restra
Publikováno v:
Behavioural Brain Research. 74:181-187
This study was aimed at investigating the role of the adrenergic mechanism in the medial preoptic area (mPOA) in sexual arousal and copulatory performance. Saline, norepinephrine (NE), phenoxybenzamine (PBZ) and propranolol (PROP) were injected into
Autor:
Mallick Hn, E. Jodo, Kamalesh K. Gulia, Yoshimasa Koyama, Tsuneharu Miki, Akihiro Kawauchi, Yukihiko Kayama
Publikováno v:
Neuroscience. 156(4)
The effects of electrical stimulation to the septum on penile erections in rats were examined to clarify the mechanisms for regulation of erectile responses during different states of vigilance. Penile responses were assessed by changes in pressure i
Publikováno v:
Physiologybehavior. 57(1)
Adrenergic alpha 2 agonist (clonidine) and its antagonist (yohimbine) were locally applied to the medial preoptic area (mPOA), to find out the role of alpha 2 receptors at this brain region in the regulation of sleep-wakefulness. Clonidine produced a
Publikováno v:
The Japanese journal of physiology. 44(3)
The role of the preoptic area (POA) neurons in the regulation of sleep-wakefulness (S-W) has been investigated in this study. The cell-specific neurotoxin, kainic acid (KA), was injected (0.8 microgram in 0.2 microliter) intracerebrally for lesioning
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