Zobrazeno 1 - 10
of 30
pro vyhledávání: '"Makoto Furuta"'
Autor:
Makoto Furuta, Takayuki Yamauchi, Yuta Miyagi, Kenta Ohashi, Hikaru Nakagawa, Yoshihiko Okabe, Masae Iwamoto Ishihara, Atsushi Hasegawa, Yasunori Kishimoto, Daisuke Hayashi
Publikováno v:
Ecological Research. 35:733-741
E4BP4 in macrophages induces an anti-inflammatory phenotype that ameliorates the severity of colitis
Autor:
Yasuko Kajimura, Akihiko Taguchi, Yuko Nagao, Kaoru Yamamoto, Konosuke Masuda, Kensuke Shibata, Yoichi Asaoka, Makoto Furutani-Seiki, Yukio Tanizawa, Yasuharu Ohta
Publikováno v:
Communications Biology, Vol 7, Iss 1, Pp 1-13 (2024)
Abstract Macrophages are versatile cells of the innate immune system that work by altering their pro- or anti-inflammatory features. Their dysregulation leads to inflammatory disorders such as inflammatory bowel disease. We show that macrophage-speci
Externí odkaz:
https://doaj.org/article/879803a9b0d84608b6eb34f41f5360eb
Autor:
Makoto Furuta
Publikováno v:
Kampo Medicine. 70:65-71
Autor:
Takaya Iida, Arisa Igarashi, Kae Fukunaga, Taiga Aoki, Tomomi Hidai, Kumiko Yanagi, Masahiko Yamamori, Kazuhito Satou, Hayato Go, Tomoki Kosho, Ryuto Maki, Takashi Suzuki, Yohei Nitta, Atsushi Sugie, Yoichi Asaoka, Makoto Furutani-Seiki, Tetsuaki Kimura, Yoichi Matsubara, Tadashi Kaname
Publikováno v:
Frontiers in Genetics, Vol 15 (2024)
Introduction: RRAS2, a member of the R-Ras subfamily of Ras-like low-molecular-weight GTPases, is considered to regulate cell proliferation and differentiation via the RAS/MAPK signaling pathway. Seven RRAS2 pathogenic variants have been reported in
Externí odkaz:
https://doaj.org/article/be8d0351718044458620c887c218580e
Autor:
Kensuke Shibata, Chihiro Motozono, Masamichi Nagae, Takashi Shimizu, Eri Ishikawa, Daisuke Motooka, Daisuke Okuzaki, Yoshihiro Izumi, Masatomo Takahashi, Nao Fujimori, James B. Wing, Takahide Hayano, Yoshiyuki Asai, Takeshi Bamba, Yoshihiro Ogawa, Makoto Furutani-Seiki, Mutsunori Shirai, Sho Yamasaki
Publikováno v:
Nature Communications, Vol 13, Iss 1, Pp 1-15 (2022)
MR1 functions as an antigen presenting protein on cells in addition to MAIT cells. Here the authors use an early T cell-specific Bcl11b-deficient mouse that develops autoimmunity through a population of nonconventional MR1-restricted T cells and char
Externí odkaz:
https://doaj.org/article/1ce0fcb8575d4b299f6edbd60a8b61b6
Autor:
Toshihiko Matsumoto, Shuji Terai, Toshiyuki Oishi, Shinya Kuwashiro, Koichi Fujisawa, Naoki Yamamoto, Yusuke Fujita, Yoshihiko Hamamoto, Makoto Furutani-Seiki, Hiroshi Nishina, Isao Sakaida
Publikováno v:
Disease Models & Mechanisms, Vol 16, Iss 7 (2023)
Externí odkaz:
https://doaj.org/article/879bdce8d28748eb9bd847620008f9bf
Publikováno v:
Biochemistry and Biophysics Reports, Vol 33, Iss , Pp 101374- (2023)
Externí odkaz:
https://doaj.org/article/b3969bd291704f3194464f7b9bea7d4d
Autor:
Edward H. Oldfield, Weifen Zeng, Youn-Soo Lee, Jie Li, Makoto Furuta, Zhengping Zhuang, Alexander O. Vortmeyer, Hiroaki Okamoto, Timothy W. Vogel, Robert J. Weil
Publikováno v:
Clinical Cancer Research. 11:3624-3632
Introduction: Research into the pathogenesis, molecular signaling, and treatment of glioblastoma multiforme (GBM) has traditionally been conducted using cell lines derived from malignant gliomas. We compared protein expression patterns between solid
Autor:
Makoto Furuta, Hiroaki Okamoto, Tetsuya Shiraishi, Kazuo Tabuchi, Mieko Shiwa, Toshihiro Mineta
Publikováno v:
Developmental Brain Research. 152:189-197
Neural stem cells (NSCs) are multipotential progenitor cells that have self-renewal activity. Since the fates of the NSCs in situ depend on their niche containing growth factors and cytokines, we performed surface enhanced laser desorption/ionization
Autor:
Robert J. Weil, Jingqi Lei, Steve Huang, Makoto Furuta, Deb A. Bhowmick, Tai Nan Huang, Youn Soo Lee, Alexander O. Vortmeyer, Zhengping Zhuang, Irina A. Lubensky, Edward H. Oldfield
Publikováno v:
Oncogene. 23:6806-6814
Glioblastoma multiforme (GBM) has been subdivided into two types based on clinical and genetic findings: primary tumors, which arise de novo, and secondary tumors, which progress from lower grade gliomas to GBMs. To analyse this dichotomy at the prot