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Abstract Background The highly pathogenic Nipah virus (NiV) causes fatal respiratory and brain infections in animals and humans. The major hallmark of the infection is a systemic endothelial infection, predominantly in the CNS. Infection of brain end

Externí odkaz: https://doaj.org/article/227cfe2dc0594603a27fc31b3e64dbe3

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Akademický článek

Ephrin-B2 expression critically influences Nipah virus infection independent of its cytoplasmic tail

Autor: Augustin Hellmut G, Pfaff Dennis, Erbar Stephanie, Diederich Sandra, Thiel Lena, Maisner Andrea

Publikováno v: Virology Journal, Vol 5, Iss 1, p 163 (2008)

Abstract Background Cell entry and cell-to-cell spread of the highly pathogenic Nipah virus (NiV) requires binding of the NiV G protein to cellular ephrin receptors and subsequent NiV F-mediated fusion. Since expression levels of the main NiV entry r

Externí odkaz: https://doaj.org/article/4c9171f66a67448bb26bfb764951ae2a

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Akademický článek

Selective receptor expression restricts Nipah virus infection of endothelial cells

Autor: Diederich Sandra, Erbar Stephanie, Maisner Andrea

Publikováno v: Virology Journal, Vol 5, Iss 1, p 142 (2008)

Abstract Nipah virus (NiV) is a highly pathogenic paramyxovirus that causes severe diseases in animals and humans. Endothelial cell (EC) infection is an established hallmark of NiV infection in vivo. Despite systemic virus spread via the vascular sys

Externí odkaz: https://doaj.org/article/607bae64fb7f4106bf2673f299f309a9

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