Llgl1 prevents metaplastic survival driven by epidermal growth factor dependent migration

Autor: Greenwood, Erin, Maisel, Sabrina, Ebertz, David, Russ, Atlantis, Pandey, Ritu, Schroeder, Joyce

We have previously demonstrated that Llgl1 loss results in a gain of mesenchymal phenotypes and a loss of apicobasal and planar polarity. We now demonstrate that these changes represent a fundamental shift in cellular phenotype. Llgl1 regulates the e

Externí odkaz: http://hdl.handle.net/10150/622116
http://arizona.openrepository.com/arizona/handle/10150/622116

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MOESM1 of Stapled EGFR peptide reduces inflammatory breast cancer and inhibits additional HER-driven models of cancer

Autor: Maisel, Sabrina, Broka, Derrick, Atwell, Benjamin, Bunch, Thomas, Kupp, Robert, Singh, Shiv, Shwetal Mehta, Schroeder, Joyce

Additional file 1. Additional data for novel pan-HER therapeutic. Figure S1. Cell line HER protein expression. Figure S2. IV-SAH5-EJ1 post 24Â h. Figure S3. Glioma-luciferase in vivo. Table S1. Peptide COA. Table S2. Injection site reactogenicity. T

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::4130fe221e1c4959f8668e371b4133e9

Llgl1 prevents metaplastic survival driven by epidermal growth factor dependent migration.

Autor: Greenwood E; Department of Molecular and Cellular Biology, University of Arizona, Tucson, Arizona., Maisel S; Arizona Cancer Center, University of Arizona, Tucson, Arizona.; Cancer Biology Program, University of Arizona, Tucson, Arizona., Ebertz D; Department of Molecular and Cellular Biology, University of Arizona, Tucson, Arizona., Russ A; Department of Molecular and Cellular Biology, University of Arizona, Tucson, Arizona.; Genetics Program, University of Arizona, Tucson, Arizona., Pandey R; Arizona Cancer Center, University of Arizona, Tucson, Arizona.; Department of Cell and Molecular Medicine, University of Arizona, Tucson, Arizona., Schroeder J; Department of Molecular and Cellular Biology, University of Arizona, Tucson, Arizona.; Arizona Cancer Center, University of Arizona, Tucson, Arizona.; BIO5 Institute, University of Arizona, Tucson, Arizona.; Genetics Program, University of Arizona, Tucson, Arizona.; Cancer Biology Program, University of Arizona, Tucson, Arizona.

Publikováno v: Oncotarget [Oncotarget] 2016 Sep 20; Vol. 7 (38), pp. 60776-60792.