Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Maiko Shoji"'
Autor:
Maiko Shoji, Osamu Takeichi, Masao Maeno, Akira Morozumi, Tomoko Katono, Natsuko Tanabe, Naoto Suzuki, Narihiro Mitsui
Publikováno v:
Acta Biochimica et Biophysica Sinica. 39:163-172
Previous studies have indicated that lipopolysaccharide (LPS) from Gram-negative bacteria in plaque induces the release of prostaglandin E2 (PGE2), which promotes alveolar bone resorption in periodontitis, and that tobacco smoking might be an importa
Lipopolysaccharide stimulates the production of prostaglandin E2 and the receptor Ep4 in osteoblasts
Autor:
Naoto Suzuki, Natsuko Tanabe, Masao Maeno, Osamu Takeichi, Narihiro Mitsui, Hideki Tanaka, Atsuto Sugaya, Maiko Shoji
Publikováno v:
Life Sciences. 78:2012-2018
Previous studies have indicated that one of the causes of alveolar bone destruction with periodontitis is lipopolysaccharide (LPS) from the cell wall of gram-negative bacteria in plaque, and that prostaglandin E(2) (PGE(2)) is one of the bone resorpt
Autor:
Masafumi Motohashi, Maiko Shoji, Natsuko Tanabe, Hideki Tanaka, Tomoko Katono, Setsuko Sato, Masao Maeno, Naoto Suzuki
Publikováno v:
Life Sciences. 78:1733-1740
Several studies have indicated that one of the causes of alveolar bone destruction with periodontitis is lipopolysaccharide (LPS) from the cell wall of Gram-negative bacteria in plaque and that tobacco smoking may be an important risk factor for the
Autor:
Maiko, Shoji, Natsuko, Tanabe, Narihiro, Mitsui, Naoto, Suzuki, Osamu, Takeichi, Tomoko, Katono, Akira, Morozumi, Masao, Maeno
Publikováno v:
Acta biochimica et biophysica Sinica. 39(3)
Previous studies have indicated that lipopolysaccharide (LPS) from Gram-negative bacteria in plaque induces the release of prostaglandin E(2) (PGE(2)), which promotes alveolar bone resorption in periodontitis, and that tobacco smoking might be an imp
Autor:
Atsuto Sugaya, Hirotaka Torigoe, Naoto Suzuki, Natsuko Tanabe, Hideki Tanaka, Maiko Shoji, Masao Maeno, Masafumi Motohashi
Publikováno v:
Life sciences. 77(18)
Several in vitro and in vivo studies have indicated that tobacco smoking may be an important risk factor for the development and severity of inflammatory periodontal disease. In the present study, we examined the effect of nicotine on cell proliferat