Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Mai M. Abdelmageed"'
Autor:
Akshayakeerthi Arthanarisami, Yohei Komaru, Charikleia Katsouridi, Julian Schumacher, Deborah K. Verges, Liang Ning, Mai M. Abdelmageed, Andreas Herrlich, Eirini Kefaloyianni
Publikováno v:
Cells, Vol 12, Iss 18, p 2214 (2023)
Elevated levels of circulating tumor necrosis factor receptors 1 and 2 (cTNFR1/2) predict chronic kidney disease (CKD) progression; however, the mechanisms of their release remain unknown. Whether acute kidney injury (AKI) drives cTNFR1/2 elevations
Externí odkaz:
https://doaj.org/article/a87232c156664889be27bb2659fedced
Autor:
Mohamed M Elmazar, Ebtehal El-Demerdash, Samar S Azab, Reem N Elnaga, Eman M Mantawy, Mai M Abdelmageed
Publikováno v:
Archives of Pharmaceutical Sciences Ain Shams University, Vol 4, Iss 2, Pp 215-223 (2020)
Cisplatin is a major classical anticancer agents. However, cisplatin induced nephrotoxicity remains a major dose limiting side effect. Several studies has highlighted the role of catalytic iron content in several diseases including cispatin nephrotox
Externí odkaz:
https://doaj.org/article/7ef9395be900458ba89fb0ed349d5cef
Autor:
Mai M Abdelmageed, Eirini Kefaloyianni, Akshayakeerthi Arthanarisami, Yohei Komaru, Jeffrey J Atkinson, Andreas Herrlich
Publikováno v:
Nephrology Dialysis Transplantation. 38:1139-1150
Background Inflammation is a key driver of the transition of acute kidney injury to progressive fibrosis and chronic kidney disease (AKI-to-CKD transition). Blocking a-disintegrin-and-metalloprotease-17 (ADAM17)-dependent ectodomain shedding, in part
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ed70440cf0053b389e6e218a80c19bbc
https://doi.org/10.1093/ndt/gfac290
https://doi.org/10.1093/ndt/gfac290
Autor:
Mai M. Abdelmageed, Eman M. Mantawy, Mohamed M. Elmazar, Reem N. El-Naga, Ebtehal El-Demerdash, Samar S. Azab
Publikováno v:
Archives of Pharmaceutical Sciences Ain Shams University, Vol 4, Iss 2, Pp 215-223 (2020)
Cisplatin is a major classical anticancer agents. However, cisplatin induced nephrotoxicity remains a major dose limiting side effect. Several studies has highlighted the role of catalytic iron content in several diseases including cispatin nephrotox
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::17ec2aaa8376e866fb98ae1047c0f65f
https://doi.org/10.21608/aps.2020.42722.1040
https://doi.org/10.21608/aps.2020.42722.1040
Autor:
Jeffrey J. Atkinson, Fatima Zohra Khamissi, Andreas Herrlich, Mai M Abdelmageed, Eirini Kefaloyianni, Akshayakeerthi Arthanarisami
Proximal-tubule-cell EGFR activation mediates tubule cell proliferation and repair early after kidney injury, while sustained EGFR activation causes kidney fibrosis. Inflammation is a key driver of AKI-to-CKD transition and fibrosis, but mechanisms o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4525d4fb5daf17e37598188269e129d0
https://doi.org/10.1101/2021.04.29.442002
https://doi.org/10.1101/2021.04.29.442002
Autor:
Benjamin D. Humphreys, Andreas Herrlich, Mai M. Abdelmageed, Farid F. Kadyrov, Shiyo Ikeda, Eirini Kefaloyianni, Haojia Wu, Akio Kobayashi, Matthew A. Lalli, Monica Chang-Panesso
Publikováno v:
The Journal of clinical investigation. 129(12)
The proximal tubule has a remarkable capacity for repair after acute injury, but the cellular lineage and molecular mechanisms underlying this repair response are incompletely understood. Here, we developed a Kim1-GFPCreERt2 knockin mouse line (Kim1-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ffef1b544c9557a24cf045c7bd1a0644
https://pubmed.ncbi.nlm.nih.gov/32331592
https://pubmed.ncbi.nlm.nih.gov/32331592
Publikováno v:
Scientific Reports
Sorafenib is the only chemotherapeutic agent currently approved for unresectable hepatocellular carcinoma (HCC). However, poor response rates have been widely reported. Indole-3-carbinol (I3C) is a potential chemopreventive phytochemical. The present
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c677492dc6c3b105599d3608c375d0e