Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Mahya, Eslami"'
Autor:
Md Ashik Ullah, Beatriz Garcillán, Eden Whitlock, William A. Figgett, Simona Infantino, Mahya Eslami, SiLing Yang, M. Arifur Rahman, Yong H. Sheng, Nicholas Weber, Pascal Schneider, Constantine S. Tam, Fabienne Mackay
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
BackgroundChronic Lymphocytic Leukemia (CLL) is characterized by the expansion of CD19+ CD5+ B cells but its origin remains debated. Mutated CLL may originate from post-germinal center B cells and unmutated CLL from CD5+ mature B cell precursors. Irr
Externí odkaz:
https://doaj.org/article/947282a58a244f7ab452eed5a10ac908
Autor:
Mahya Eslami, Daniela Willen, Orestis Papasouliotis, Sonia Schuepbach-Mallpell, Laure Willen, Olivier Donzé, Özkan Yalkinoglu, Pascal Schneider
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
BAFF (B cell activation factor of the TNF family/B lymphocyte stimulator, BLyS) and APRIL (a proliferation-inducing ligand) are targeted by atacicept, a decoy receptor consisting of the extracellular domain of TACI (transmembrane activator and calciu
Externí odkaz:
https://doaj.org/article/9e52c1302a504221b6764fbf467e5860
Autor:
Mahya Eslami, Edgar Meinl, Hermann Eibel, Laure Willen, Olivier Donzé, Ottmar Distl, Holm Schneider, Daniel E. Speiser, Dimitrios Tsiantoulas, Özkan Yalkinoglu, Eileen Samy, Pascal Schneider
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 8 (2020)
B cell activation factor of the TNF family (BAFF/BLyS), an essential B cell survival factor of which circulating levels are elevated in several autoimmune disorders, is targeted in the clinic for the treatment of systemic lupus erythematosus (SLE). T
Externí odkaz:
https://doaj.org/article/76027ff55cba448a9c4dc593f4326ad2
Autor:
Michele Vigolo, Melissa G. Chambers, Laure Willen, Dehlia Chevalley, Klaus Maskos, Alfred Lammens, Aubry Tardivel, Dolon Das, Christine Kowalczyk-Quintas, Sonia Schuepbach-Mallepell, Cristian R. Smulski, Mahya Eslami, Antonius Rolink, Edith Hummler, Eileen Samy, Yves Fomekong Nanfack, Fabienne Mackay, Maofu Liao, Henry Hess, Xuliang Jiang, Pascal Schneider
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-15 (2018)
BAFF is an important cytokine for B cell survival, and is a therapeutic target for autoimmune disorders. Here the authors show that a 'flap' region of BAFF converts BAFFR binding events into survival signals and, with structural data, that this ‘fl
Externí odkaz:
https://doaj.org/article/843bbc0b96a640bc806daedcfd909b7b
Autor:
Pascal Schneider, Mahya Eslami
Publikováno v:
Current Opinion in Immunology. 71:75-80
B cell activating factor (BAFF or BLyS), an important cytokine for B cell survival and humoral immune responses, is targeted in the clinic for the treatment of systemic lupus erythematosus. This review focuses on the structure, function and inhibitio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1556869fce032993888e5966bafdd175
https://doi.org/10.1016/j.coi.2021.06.009
https://doi.org/10.1016/j.coi.2021.06.009
Autor:
Michele Vigolo, Christine Kowalczyk-Quintas, Özkan Yalkinoglu, Daniela Willen, Mahya Eslami, Benjamin Peter, Eileen Samy, Michaela Golob, Pascal Schneider, Hervé Broly, Laure Willen, Sonia Schuepbach-Mallepell
Publikováno v:
British journal of pharmacology, vol. 176, no. 20, pp. 4019-4033
British Journal of Pharmacology
British Journal of Pharmacology
Background and Purpose The TNF family ligands, B cell activating factor of the TNF family (BAFF, also known as B lymphocyte stimulator, BLyS) and a proliferation‐inducing ligand (APRIL), share the transmembrane activator and calcium‐modulator and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::654b0584a5fc1a11e6e8c6ea08f6e7e7
https://doi.org/10.1111/bph.14811
https://doi.org/10.1111/bph.14811
Autor:
Sonia, Schuepbach-Mallepell, Christine, Kowalczyk-Quintas, Angela, Dick, Mahya, Eslami, Michele, Vigolo, Denis J, Headon, Michael, Cheeseman, Holm, Schneider, Pascal, Schneider
Publikováno v:
Methods in molecular biology, vol. 2248, pp. 167-183
Genetic deficiency of ectodysplasin A (EDA) causes X-linked hypohidrotic ectodermal dysplasia, a congenital condition characterized by the absence or abnormal formation of sweat glands, teeth, and several skin appendages. Stimulation of the EDA recep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::27ed27b12e17063186650f845d02b0a0
https://serval.unil.ch/resource/serval:BIB_7E7970A5AB18.P002/REF.pdf
https://serval.unil.ch/resource/serval:BIB_7E7970A5AB18.P002/REF.pdf
Autor:
Holm Schneider, Michael Cheeseman, Sonia Schuepbach-Mallepell, Angela Dick, Denis J. Headon, Michele Vigolo, Christine Kowalczyk-Quintas, Mahya Eslami, Pascal Schneider
Publikováno v:
Methods in Molecular Biology ISBN: 9781071611296
Genetic deficiency of ectodysplasin A (EDA) causes X-linked hypohidrotic ectodermal dysplasia, a congenital condition characterized by the absence or abnormal formation of sweat glands, teeth, and several skin appendages. Stimulation of the EDA recep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b3194090556e7961a1b7f62ca5f7671e
https://doi.org/10.1007/978-1-0716-1130-2_12
https://doi.org/10.1007/978-1-0716-1130-2_12
Autor:
Ottmar Distl, Holm Schneider, Laure Willen, Özkan Yalkinoglu, Daniel E. Speiser, Dimitrios Tsiantoulas, Eileen Samy, Olivier Donzé, Pascal Schneider, Hermann Eibel, Mahya Eslami, Edgar Meinl
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 8 (2020)
Frontiers in cell and developmental biology, vol. 8, pp. 577662
Frontiers in Cell and Developmental Biology
Frontiers in cell and developmental biology, vol. 8, pp. 577662
Frontiers in Cell and Developmental Biology
B cell activation factor of the TNF family (BAFF/BLyS), an essential B cell survival factor of which circulating levels are elevated in several autoimmune disorders, is targeted in the clinic for the treatment of systemic lupus erythematosus (SLE). T
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::14ed958af14f015bcdc7aedf70f60859
https://www.frontiersin.org/articles/10.3389/fcell.2020.577662/full
https://www.frontiersin.org/articles/10.3389/fcell.2020.577662/full
Autor:
Dimitrios Tsiantoulas, Jordi Lambert, Florian J. Mayer, Winfried März, Laure Willen, Lennart Enders, Juliane Weißer, Laura Göderle, Marc Clement, Florentina Porsch, Jan Borén, Georg Obermayer, M. Kiss, Stefan Kubicek, Jane E. Murphy, Ziad Mallat, Gerard Pasterkamp, Per Fogelstrand, Florian Frommlet, Hubert Scharnagl, Tabassome Simon, Tim Hendrikx, Henry Hess, Diede Smeets, Maria Ozsvar-Kozma, Nicolas Danchin, Matthias Hoke, André C. Mueller, Olivier Donzé, Helle F. Jørgensen, Mahya Eslami, Pascal Schneider, Christoph J. Binder, Taras Afonyushkin
Publikováno v:
Nature, vol. 597, no. 7874, pp. 92-96
Atherosclerotic cardiovascular disease causes heart attacks and strokes, which are the leading causes of mortality worldwide1. The formation of atherosclerotic plaques is initiated when low-density lipoproteins bind to heparan-sulfate proteoglycans (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::21f61c974cba0cb57596228c9724deb4
https://pubmed.ncbi.nlm.nih.gov/34475542
https://pubmed.ncbi.nlm.nih.gov/34475542