Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Magdalena Grundmann"'
Autor:
Lars Brodowski, Jennifer Burlakov, Ashley C Myerski, Constantin S von Kaisenberg, Magdalena Grundmann, Carl A Hubel, Frauke von Versen-Höynck
Publikováno v:
PLoS ONE, Vol 9, Iss 6, p e98527 (2014)
Placenta-derived circulating factors contribute to the maternal endothelial dysfunction underlying preeclampsia. Endothelial colony forming cells (ECFC), a sub-population of endothelial progenitor cells (EPCs), are thought to be involved in vasculoge
Externí odkaz:
https://doaj.org/article/b4a543908e15424da543045d3c77dcbb
Publikováno v:
Pregnancy hypertension. 2(3)
Introduction Preeclampsia is multifactorial in origin but the primary trigger is thought to be related to impaired placentation which is followed by systemic maternal responses. Vitamin D3 deficiency is a worldwide problem and is associated with a su
Autor:
Ashley Myerski, Jennifer Burlakov, Magdalena Grundmann, Carl A. Hubel, Frauke von Versen-Höynck, Constantin von Kaisenberg, Lars Brodowski
Publikováno v:
PLoS ONE, Vol 9, Iss 6, p e98527 (2014)
PLoS ONE
PLoS ONE
Context: Placenta-derived circulating factors contribute to the maternal endothelial dysfunction underlying preeclampsia. Endothelial colony forming cells (ECFC), a sub-population of endothelial progenitor cells (EPCs), are thought to be involved in
Autor:
Magdalena Grundmann, N Darashchonak, S. Haß, S. Placzko, R. Niendorf, M. Haidar, F von Versen-Höynck, Carl A. Hubel
Publikováno v:
Geburtshilfe und Frauenheilkunde. 73
Autor:
N Darashchonak, R. Niendorf, Magdalena Grundmann, F. von Versen-Höynck, Carl A. Hubel, S. Placzko, M. Haidar
The main pathogenic feature of preeclampsia is maternal endothelial dysfunction that results from impaired angiogenesis and reduced endothelial repair capacity. In addition, preeclampsia risk is associated with vitamin D deficiency. We hypothesized t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd6c6abdf6a15b5938ffbe926c9a68bb
https://europepmc.org/articles/PMC3492823/
https://europepmc.org/articles/PMC3492823/
Publikováno v:
Zeitschrift für Geburtshilfe und Neonatologie. 215
Autor:
Douglas F. Easton, Nicola J. Camp, Magdalena Grundmann, Jolanta Lissowska, Javier Benitez, Martha S. Linet, Angela Cox, Thilo Dörk, Natalia Dubrowinskaja, Graham G. Giles, Wei-Yu Lin, Melissa C. Southey, Malcolm W.R. Reed, Kristina Allen-Brady, Lisa A. Cannon-Albright, Preetha Rajaraman, José Ignacio Arias-Perez, Paul D.P. Pharoah, John L. Hopper, Jonine D. Figueroa, Carmel Apicella, Tjoung Won Park-Simon, Marjanka K. Schmidt, Roger L. Milne, Primitiva Menendez-Rodriguez, Alison M. Dunning, Sabapathy P. Balasubramanian, Alice J. Sigurdson, Mark E. Sherman, Montserrat Garcia-Closas, Michele M. Doody
Background \ud The XRCC2 gene is a key mediator in the homologous recombination repair of DNA double strand breaks. It is hypothesised that inherited variants in the XRCC2 gene might also affect susceptibility to, and survival from, breast cancer.\ud
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ca9d3120f8ef45694ea8c2c166744bf
https://europepmc.org/articles/PMC3932658/
https://europepmc.org/articles/PMC3932658/
Autor:
Hermann Haller, Marion Haubitz, Bettina Hollwitz, Uta Erdbruegger, Magdalena Grundmann, Alexander Woywodt, Katrin Oehler, Torsten Kirsch
Publikováno v:
American Journal of Obstetrics and Gynecology. 198:317.e1-317.e5
Preeclampsia is a disorder of endothelial cells, and novel markers of the disease are eagerly awaited. We tested the hypothesis that circulating endothelial cells (CECs) are elevated in preeclampsia and that cell numbers correlate with disease activi
Publikováno v:
Reproductive Biology and Endocrinology, Vol 9, Iss 1, p 146 (2011)
Reproductive Biology and Endocrinology : RB&E
Reproductive Biology and Endocrinology : RB&E
In the past few years a growing interest in vitamin D can be observed in the lay and biomedical literature due to findings demonstrating a low vitamin D status in the population. In addition to its importance for the regulation of calcium and phospho