Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Magda M. Melkonyan"'
Autor:
Elisabeth Singer, Lilit Hunanyan, Magda M. Melkonyan, Jonasz J. Weber, Lusine Danielyan, Huu Phuc Nguyen
Publikováno v:
Pharmaceuticals, Vol 14, Iss 3, p 257 (2021)
Huntington’s disease (HD) is a monogenetic neurodegenerative disorder characterized by the accumulation of polyglutamine-expanded huntingtin (mHTT). There is currently no cure, and therefore disease-slowing remedies are sought to alleviate symptoms
Externí odkaz:
https://doaj.org/article/84cac017ab2c42369e0a0807e2db2a90
Autor:
Ute A. Schwinghammer, Magda M. Melkonyan, Lilit Hunanyan, Roman Tremmel, Ralf Weiskirchen, Erawan Borkham-Kamphorst, Elke Schaeffeler, Torgom Seferyan, Wolfgang Mikulits, Konstantin Yenkoyan, Matthias Schwab, Lusine Danielyan
Publikováno v:
Cells, Vol 9, Iss 2, p 456 (2020)
The noradrenergic system is proposed to play a prominent role in the pathogenesis of liver fibrosis. While α1- and β-adrenergic receptors (ARs) are suggested to be involved in a multitude of profibrogenic actions, little is known about α2-AR-media
Externí odkaz:
https://doaj.org/article/8e42606377594dc5aff0979f78946255
Autor:
Huu Phuc Nguyen, Magda M Melkonyan, Elisabeth Singer, Lilit Hunanyan, Lusine Danielyan, Jonasz J. Weber
Publikováno v:
Pharmaceuticals
Volume 14
Issue 3
Pharmaceuticals, Vol 14, Iss 257, p 257 (2021)
Volume 14
Issue 3
Pharmaceuticals, Vol 14, Iss 257, p 257 (2021)
Huntington’s disease (HD) is a monogenetic neurodegenerative disorder characterized by the accumulation of polyglutamine-expanded huntingtin (mHTT). There is currently no cure, and therefore disease-slowing remedies are sought to alleviate symptoms
Autor:
Magda M. Melkonyan, Lilit Hunanyan, Ali Lourhmati, Nikolas Layer, Sandra Beer-Hammer, Konstantin Yenkoyan, Matthias Schwab, Lusine Danielyan
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 19, Iss 1, p 9 (2017)
International Journal of Molecular Sciences, Vol 19, Iss 1, p 9 (2017)
Locus coeruleus-noradrenergic system dysfunction is known to contribute to the progression of Alzheimer’s disease (AD). Besides a variety of reports showing the involvement of norepinephrine and its receptor systems in cognition, amyloid β (Aβ) m