Defects in mitophagy promote redox-driven metabolic syndrome in the absence of TP53INP1

Autor: Seillier, Marion, Pouyet, Laurent, N Guessan, Prudence, Nollet, Marie, Capo, Florence, Guillaumond, Fabienne, Peyta, Laure, Dumas, Jean-Francois, Varrault, Annie, Bertrand, Gyslaine, Bonnafous, Stephanie, Tran, Albert, Meur, Gargi, Marchetti, Piero, Magalie Ravier, Dalle, Stephane, Gual, Philippe, Muller, Dany, Rutter, Guy A., Servais, Stephane, Iovanna, Juan L., Carrier, Alice

Publikováno v: EMBO Molecular Medicine
EMBO Molecular Medicine, Wiley Open Access, 2015, pp.802-818. ⟨10.15252/emmm.201404318⟩
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EMBO Molecular Medicine, Wiley Open Access, 2015, 7, pp.802-818. ⟨10.15252/emmm.201404318⟩
EMBO Molecular Medicine, 2015, pp.802-818. ⟨10.15252/emmm.201404318⟩

International audience; The metabolic syndrome covers metabolic abnormalities including obesity and type 2 diabetes (T2D). T2D is characterized by insulin resistance resulting from both environmental and genetic factors. A genome-wide association stu

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::62f23e8d3370580f2ad330e32c0e5f0e
https://hal-amu.archives-ouvertes.fr/hal-01208177/document