Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Madison T, Gray"'
Autor:
Madison T. Gray, Julie L. Lefebvre
Publikováno v:
Neuron. 111(1)
Visual impairments in albinism result from decreased ipsilateral retinal projections. In this issue of Neuron, Slavi, Balasubramanian, and colleagues
Autor:
Madison T. Gray, John Woulfe
Publikováno v:
Journal of Cerebral Blood Flow & Metabolism. 35:747-750
In vivo studies have shown that blood–brain barrier (BBB) dysfunction is involved in the course of Parkinson's disease (PD). However, these have lacked either anatomic definition or the ability to recognize minute changes in BBB integrity. Here, us
Publikováno v:
Woulfe, J M, Gray, M T, Gray, D A, Munoz, D G & Middeldorp, J M 2014, ' Hypothesis: A role for EBV-induced molecular mimicry in Parkinson's disease ', Parkinsonism and Related Disorders, vol. 20, no. 7, pp. 685-694 . https://doi.org/10.1016/j.parkreldis.2014.02.031
Parkinsonism and Related Disorders, 20(7), 685-694. Elsevier BV
Parkinsonism and Related Disorders, 20(7), 685-694. Elsevier BV
Current concepts regarding the pathogenesis of Parkinson's disease support a model whereby environmental factors conspire with a permissive genetic background to initiate the disease. The identity of the responsible environmental trigger has remained
Publikováno v:
Movement Disorders. 29:991-998
Parkinson's disease is characterized by the pathological aggregation of Alpha-synuclein. The dual-hit hypothesis proposed by Braak implicates the enteric nervous system as an initial site of α-synuclein aggregation with subsequent spread to the cent
Autor:
Pedro Guarionex Lopez, Madison T. Gray, Bruce Patterson, Michael Nevin Rutherford, Denis Maurice Bonin
Publikováno v:
Journal of Clinical Oncology. 31:e410-e412
Autor:
Matthew Y. Tang, P.P.K.M. Gamage, Madison T. Gray, Josée Coulombe, Mei Zhang, John Woulfe, Douglas A. Gray, M. Jill Saffrey
Publikováno v:
Frontiers in Aging Neuroscience
UCHL1 (ubiquitin carboxyterminal hydrolase 1) is a deubiquitinating enzyme that is particularly abundant in neurons. From studies of a spontaneous mutation arising in a mouse line it is clear that loss of function of UCHL1 generates profound degenera
Publikováno v:
Movement disorders : official journal of the Movement Disorder Society. 29(9)
Publikováno v:
Annals of Neurology. 78:834-835
Autor:
Claudio Ruffmann, David G. Munoz, Laura Parkkinen, John Woulfe, Madison T. Gray, Naomi P. Visanji, Lili-Naz Hazrai, Anthony E. Lang
Publikováno v:
Neurology. 85:834-835
Editors' Note: Commenting on the lack of specificity of colonic mucosal α-synuclein as a potential biomarker in Parkinson disease, Ruffmann and Parkkinen share their “Discovery study” findings, which in part agree with the work of Visanji et al.
Publikováno v:
Movement disorders : official journal of the Movement Disorder Society. 29(8)
Parkinson's disease is characterized by the pathological aggregation of Alpha-synuclein. The dual-hit hypothesis proposed by Braak implicates the enteric nervous system as an initial site of α-synuclein aggregation with subsequent spread to the cent